Substance P is expressed in hippocampal principal neurons during status epilepticus and plays a critical role in the maintenance of status epilepticus (Q36342611)

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English	Substance P is expressed in hippocampal principal neurons during status epilepticus and plays a critical role in the maintenance of status epilepticus	scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Substance P is expressed in hippocampal principal neurons during status epilepticus and plays a critical role in the maintenance of status epilepticus (English)

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

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status epilepticus

1 reference

based on heuristic

inferred from title

author name string

H Liu

1

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

A M Mazarati

2

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

H Katsumori

3

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

R Sankar

4

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

C G Wasterlain

5

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Europe PubMed Central

PMC publication ID

13 August 2017

language of work or name

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publication date

1 April 1999

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Europe PubMed Central

PMC publication ID

13 August 2017

Proceedings of the National Academy of Sciences of the United States of America

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Europe PubMed Central

PMC publication ID

13 August 2017

96

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

9

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

5286-5291

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

Primary afferent tachykinins are required to experience moderate to intense pain

1 reference

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Excitatory amino acids as a final common pathway for neurologic disorders.

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Neurotransmitter functions of mammalian tachykinins

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Modulation of spinal excitability: co-operation between neurokinin and excitatory amino acid neurotransmitters

1 reference

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Slow excitatory transmission in rat dorsal horn: possible mediation by peptides

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A comparison of the anatomical distribution of substance P and substance P receptors in the rat central nervous system

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1 reference

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Self-sustaining status epilepticus after brief electrical stimulation of the perforant path

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Substance P innervation of the rat hippocampal formation

1 reference

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Modulation of excitatory amino acid responses by tachykinins and selective tachykinin receptor agonists in the rat spinal cord

1 reference

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Effects of neuropeptides on rat cortical neurons: laminar distribution and interaction with the effect of acetylcholine

1 reference

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Substance P excitation of cerebral cortical Betz cells

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1 reference

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Substance P raises neuronal membrane excitability by reducing inward rectification

1 reference

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1 reference

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Comparison of substance K-like and substance P-like fibers and cells in the rat hippocampus

1 reference

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Exocytosis from large dense cored vesicles outside the active synaptic zones of terminals within the trigeminal subnucleus caudalis: a possible mechanism for neuropeptide release

1 reference

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1 reference

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Dopamine differentially regulates dynorphin, substance P, and enkephalin expression in striatal neurons: in situ hybridization histochemical analysis

1 reference

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Kainic acid-mediated increase of preprotachykinin-A messenger RNA expression in the rat hippocampus and a region-selective attenuation by dexamethasone

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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A tachykinin NK1 receptor antagonist, CP-122,721-1, attenuates kainic acid-induced seizure activity

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Tachykinins may modify spontaneous epileptiform activity in the rat entorhinal cortex in vitro by activating GABAergic inhibition

1 reference

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1 reference

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1 reference

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Lamellar organization of hippocampal excitatory pathways

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=21856

22 September 2018

The substance P innervation of the rat hippocampal region

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=21856

22 September 2018

Chronic naltrexone treatment increases expression of preproenkephalin and preprotachykinin mRNA in discrete brain regions

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=21856

22 September 2018

Interactions between substance P, calcitonin gene-related peptide, taurine and excitatory amino acids in the spinal cord

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Enzyme-linked immunosorbent assay of substance P and its metabolite SP 1-7. A comparison with RIA

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulation

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Electroencephalographic and behavioral assessment of intracerebroventricular somatostatin and a substance P analogue

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Substance P elevates intracellular calcium in both neurons and glial cells from the dorsal horn of the spinal cord

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Tachykinins and calcitonin gene-related peptide enhance release of endogenous glutamate and aspartate from the rat spinal dorsal horn slice

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Tachykinins Potentiate N‐Methyl‐D‐Aspartate Responses in Acutely Isolated Neurons from the Dorsal Horn

1 reference

https://pubmed.ncbi.nlm.nih.gov/10220458

12 December 2020

based on heuristic

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Identifiers

10.1073/PNAS.96.9.5286

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

1999PNAS...96.5286L

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PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

13 August 2017

ResearchGate publication ID

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