Knockout of Vdac1 activates hypoxia-inducible factor through reactive oxygen species generation and induces tumor growth by promoting metabolic reprogramming and inflammation (Q36002110)

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English	Knockout of Vdac1 activates hypoxia-inducible factor through reactive oxygen species generation and induces tumor growth by promoting metabolic reprogramming and inflammation	scientific article

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scholarly article

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Europe PubMed Central

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11 August 2017

Knockout of Vdac1 activates hypoxia-inducible factor through reactive oxygen species generation and induces tumor growth by promoting metabolic reprogramming and inflammation (English)

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Europe PubMed Central

PMC publication ID

11 August 2017

reactive oxygen species

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Isabelle Bourget

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Isabelle Bourget

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11 August 2017

Jacques Pouysségur

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Jacques Pouysségur

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11 August 2017

Joffrey Pelletier

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Joffrey Pelletier

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Alexandra Popa

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Chloé C Féral

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Chloé Féral

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Michel Tauc

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M Christiane Brahimi-Horn

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11 August 2017

Sandy Giuliano

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11 August 2017

Estelle Saland

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Sandra Lacas-Gervais

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Tatiana Sheiko

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Frédéric Bost

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Etienne Boulter

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Mircea Ivan

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Barbara Garmy-Susini

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Bernard Mari

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Jean-Emmanuel Sarry

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William J Craigen

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Nathalie M Mazure

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26 August 2015

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11 August 2017

Cancer & Metabolism

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11 August 2017

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11 August 2017

1

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11 August 2017

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Limma

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Europe PubMed Central

22 June 2022

https://europepmc.org/article/PMC/4551760

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CZ Software Mentions: A large dataset of software mentions in the biomedical literature

Local mitochondrial-endolysosomal microfusion cleaves voltage-dependent anion channel 1 to promote survival in hypoxia

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2 October 2017

An uncoupling channel within the c-subunit ring of the F1FO ATP synthase is the mitochondrial permeability transition pore

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2 October 2017

Silencing VDAC1 Expression by siRNA Inhibits Cancer Cell Proliferation and Tumor Growth In Vivo

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2 October 2017

Hypoxic VDAC1: a potential mitochondrial marker for cancer therapy

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4551760

2 October 2017

Sites of reactive oxygen species generation by mitochondria oxidizing different substrates

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4551760

2 October 2017

Cellular and molecular mechanisms in the hypoxic tissue: role of HIF-1 and ROS.

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2 October 2017

Functions of BCL-X L at the Interface between Cell Death and Metabolism

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2 October 2017

VDAC1: from structure to cancer therapy

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2 October 2017

Mitochondria: master regulators of danger signalling

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2 October 2017

The cBio cancer genomics portal: an open platform for exploring multidimensional cancer genomics data

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2 October 2017

Expression of a truncated active form of VDAC1 in lung cancer associates with hypoxic cell survival and correlates with progression to chemotherapy resistance

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2 October 2017

Thrombomodulin and its role in inflammation.

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2 October 2017

Systematic investigation of genetic vulnerabilities across cancer cell lines reveals lineage-specific dependencies in ovarian cancer

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2 October 2017

A tag at the carboxy terminus prevents membrane integration of VDAC1 in mammalian mitochondria

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2 October 2017

The redox state of glutathione regulates the hypoxic induction of HIF-1.

1 reference

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Reactive oxygen species and HIF-1 signalling in cancer

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4551760

2 October 2017

Red blood cells induce hypoxic lung inflammation

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4551760

2 October 2017

Hypoxia and cancer

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4551760

2 October 2017

Reactive oxygen species and cellular oxygen sensing

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2 October 2017

HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells

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2 October 2017

Voltage-dependent anion channels are dispensable for mitochondrial-dependent cell death

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Properties of the permeability transition in VDAC1(-/-) mitochondria

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Voltage-dependent anion channel (VDAC) as mitochondrial governator--thinking outside the box

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2 October 2017

Arrest-defective-1 protein, an acetyltransferase, does not alter stability of hypoxia-inducible factor (HIF)-1alpha and is not induced by hypoxia or HIF

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On respiratory impairment in cancer cells

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Bcl-xL promotes the open configuration of the voltage-dependent anion channel and metabolite passage through the outer mitochondrial membrane

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Methods to monitor ROS production by fluorescence microscopy and fluorometry.

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23 September 2018

Red blood cells increase secretion of matrix metalloproteinases from human lung fibroblasts in vitro

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p42/p44 mitogen-activated protein kinases phosphorylate hypoxia-inducible factor 1alpha (HIF-1alpha) and enhance the transcriptional activity of HIF-1.

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https://pubmed.ncbi.nlm.nih.gov/26322231

12 December 2020

based on heuristic

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10.1186/S40170-015-0133-5

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Europe PubMed Central

PMC publication ID

11 August 2017

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Europe PubMed Central

PMC publication ID

11 August 2017

PubMed publication ID

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11 August 2017

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