Escape from a dominant HLA-B*15-restricted CD8+ T cell response against hepatitis C virus requires compensatory mutations outside the epitope. (Q35665865)

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scientific article published on 9 November 2011

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English	Escape from a dominant HLA-B*15-restricted CD8+ T cell response against hepatitis C virus requires compensatory mutations outside the epitope.	scientific article published on 9 November 2011

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16 January 2020

Escape from a dominant HLA-B*15-restricted CD8+ T cell response against hepatitis C virus requires compensatory mutations outside the epitope (English)

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16 January 2020

Hepatitis C virus

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16 January 2020

Daniel Hoffmann

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16 January 2020

Thomas Pietschmann

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Thomas Pietschmann

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16 January 2020

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Jörg Timm

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16 January 2020

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Marianne Ruhl

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16 January 2020

Patrick Chhatwal

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16 January 2020

Heiko Strathmann

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16 January 2020

Thomas Kuntzen

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16 January 2020

Dorothea Bankwitz

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16 January 2020

Kathrin Skibbe

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16 January 2020

Falko M Heinemann

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16 January 2020

Peter A Horn

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16 January 2020

Todd M Allen

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16 January 2020

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16 January 2020

Journal of Virology

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16 January 2020

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16 January 2020

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16 January 2020

991-1000

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16 January 2020

Compensatory mutations restore the replication defects caused by cytotoxic T lymphocyte escape mutations in hepatitis C virus polymerase

1 reference

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Transmission and long-term stability of compensated CD8 escape mutations

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Naturally occurring dominant resistance mutations to hepatitis C virus protease and polymerase inhibitors in treatment-naïve patients

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Escape from HLA-B*08-Restricted CD8 T Cells by Hepatitis C Virus Is Associated with Fitness Costs

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Analysis of CD127 and KLRG1 expression on hepatitis C virus-specific CD8+ T cells reveals the existence of different memory T-cell subsets in the peripheral blood and liver

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Evidence of Viral Adaptation to HLA Class I-Restricted Immune Pressure in Chronic Hepatitis C Virus Infection

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Hepatitis C virus mutation affects proteasomal epitope processing

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https://pubmed.ncbi.nlm.nih.gov/22072759

12 December 2020

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16 January 2020

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16 January 2020

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16 January 2020

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