Curcumin differentially regulates endoplasmic reticulum stress through transcriptional corepressor SMILE (small heterodimer partner-interacting leucine zipper protein)-mediated inhibition of CREBH (cAMP responsive element-binding protein H) (Q35605076)

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scientific article published on 12 October 2011

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English	Curcumin differentially regulates endoplasmic reticulum stress through transcriptional corepressor SMILE (small heterodimer partner-interacting leucine zipper protein)-mediated inhibition of CREBH (cAMP responsive element-binding protein H)	scientific article published on 12 October 2011

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14 February 2020

Curcumin differentially regulates endoplasmic reticulum stress through transcriptional corepressor SMILE (small heterodimer partner-interacting leucine zipper protein)-mediated inhibition of CREBH (cAMP responsive element-binding protein H) (English)

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14 February 2020

endoplasmic reticulum

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Jagannath Misra

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14 February 2020

Dipanjan Chanda

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14 February 2020

Don-kyu Kim

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14 February 2020

Tiangang Li

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Sung-Hoon Back

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Hueng-Sik Choi

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12 October 2011

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14 February 2020

Journal of Biological Chemistry

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286

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14 February 2020

41972-41984

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14 February 2020

Curcumin improves insulin resistance in skeletal muscle of rats

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Endoplasmic reticulum stress-induced activation of activating transcription factor 6 decreases cAMP-stimulated hepatic gluconeogenesis via inhibition of CREB.

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Hepatocyte growth factor family negatively regulates hepatic gluconeogenesis via induction of orphan nuclear receptor small heterodimer partner in primary hepatocytes

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Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

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Cooperative interaction of Zhangfei and ATF4 in transactivation of the cyclic AMP response element

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The neuronal host cell factor-binding protein Zhangfei inhibits herpes simplex virus replication

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The liver-enriched transcription factor CREB-H is a growth suppressor protein underexpressed in hepatocellular carcinoma

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The tumor suppressor LKB1 kinase directly activates AMP-activated kinase and regulates apoptosis in response to energy stress

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The luminal domain of ATF6 senses endoplasmic reticulum (ER) stress and causes translocation of ATF6 from the ER to the Golgi

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ER stress induces cleavage of membrane-bound ATF6 by the same proteases that process SREBPs

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Identifiers

10.1074/JBC.M111.274514

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14 February 2020

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14 February 2020

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