The CB(2) cannabinoid receptor controls myeloid progenitor trafficking: involvement in the pathogenesis of an animal model of multiple sclerosis. (Q34589360)

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English	The CB(2) cannabinoid receptor controls myeloid progenitor trafficking: involvement in the pathogenesis of an animal model of multiple sclerosis.	scientific article

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4 January 2020

The CB(2) cannabinoid receptor controls myeloid progenitor trafficking: involvement in the pathogenesis of an animal model of multiple sclerosis (English)

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4 January 2020

multiple sclerosis

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cannabinoid receptor

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Javier Palazuelos

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4 January 2020

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4 January 2020

Raphael Mechoulam

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Raphael Mechoulam

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4 January 2020

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Nathalie Davoust

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4 January 2020

Boris Julien

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4 January 2020

Eric Hatterer

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4 January 2020

Tania Aguado

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Cristina Benito

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4 January 2020

Augusto Silva

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4 January 2020

Manuel Guzmán

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4 January 2020

Serge Nataf

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4 January 2020

Ismael Galve-Roperh

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11 March 2008

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4 January 2020

Journal of Biological Chemistry

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4 January 2020

283

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4 January 2020

13320-13329

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4 January 2020

The molecular logic of endocannabinoid signalling

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M707960200

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Emerging properties of cannabinoid medicines in management of multiple sclerosis

1 reference

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Decreased endocannabinoid levels in the brain and beneficial effects of agents activating cannabinoid and/or vanilloid receptors in a rat model of multiple sclerosis

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Immunoregulation of a viral model of multiple sclerosis using the synthetic cannabinoid R+WIN55,212

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Activation of the endocannabinoid system as therapeutic approach in a murine model of multiple sclerosis

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The endocannabinoid anandamide protects neurons during CNS inflammation by induction of MKP-1 in microglial cells

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Cannabinoids and neuroinflammation

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Control of spasticity in a multiple sclerosis model is mediated by CB1, not CB2, cannabinoid receptors

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Cannabinoid CB2 receptor: a new target for controlling neural cell survival?

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Modulation of the cannabinoid CB2 receptor in microglial cells in response to inflammatory stimuli.

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The neurobiology of multiple sclerosis: genes, inflammation, and neurodegeneration

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Randomized controlled trial of cannabis-based medicine in spasticity caused by multiple sclerosis

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Bone marrow stem cells have the ability to populate the entire central nervous system into fully differentiated parenchymal microglia

1 reference

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Bone marrow CD34+/B220+ progenitors target the inflamed brain and display in vitro differentiation potential toward microglia

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Cannabinoid receptors as therapeutic targets.

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Immunomodulation by cannabinoids is absent in mice deficient for the cannabinoid CB(2) receptor

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The endocannabinoid system promotes astroglial differentiation by acting on neural progenitor cells.

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Flt3+ macrophage precursors commit sequentially to osteoclasts, dendritic cells and microglia

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Cannabinoid CB1 and CB2 receptors and fatty acid amide hydrolase are specific markers of plaque cell subtypes in human multiple sclerosis.

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HU-308: a specific agonist for CB(2), a peripheral cannabinoid receptor

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Microglia: active sensor and versatile effector cells in the normal and pathologic brain

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Human CD34+ cells differentiate into microglia and express recombinant therapeutic protein

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Microglia in the adult brain arise from Ly-6ChiCCR2+ monocytes only under defined host conditions

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Expression of CCR2 in Both Resident and Bone Marrow-Derived Microglia Plays a Critical Role in Neuropathic Pain

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Non-psychoactive CB2 cannabinoid agonists stimulate neural progenitor proliferation

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The peripheral cannabinoid receptor Cb2, frequently expressed on AML blasts, either induces a neutrophilic differentiation block or confers abnormal migration properties in a ligand-dependent manner

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Cultured rat microglial cells synthesize the endocannabinoid 2-arachidonylglycerol, which increases proliferation via a CB2 receptor-dependent mechanism

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COX-2, CB2 and P2X7-immunoreactivities are increased in activated microglial cells/macrophages of multiple sclerosis and amyotrophic lateral sclerosis spinal cord

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Experimental autoimmune encephalomyelitis disrupts endocannabinoid-mediated neuroprotection

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Cannabinoids in multiple sclerosis (CAMS) study: safety and efficacy data for 12 months follow up.

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