Developmental biology: vasculogenesis is a wreck without RECK. (Q34574350)

3 August 2017

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title

Developmental biology: vasculogenesis is a wreck without RECK. (English)

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3 August 2017

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developmental biology

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Zena Werb

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author name string

Bryan Welm

series ordinal

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3 August 2017

Joni Mott

series ordinal

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3 August 2017

Zena Werb

series ordinal

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3 August 2017

language of work or name

English

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publication date

1 March 2002

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3 August 2017

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3 August 2017

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3 August 2017

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3 August 2017

page(s)

R209-11

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How matrix metalloproteinases regulate cell behavior

3 August 2017

cites work

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The membrane-anchored MMP inhibitor RECK is a key regulator of extracellular matrix integrity and angiogenesis

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Regulation of matrix metalloproteinase-9 and inhibition of tumor invasion by the membrane-anchored glycoprotein RECK

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7 January 2021

Tissue factor pathway inhibitor-2 is a novel inhibitor of matrix metalloproteinases with implications for atherosclerosis

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Post-translational proteolytic processing of procollagen C-terminal proteinase enhancer releases a metalloproteinase inhibitor.

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7 January 2021

Mechanism of inhibition of the human matrix metalloproteinase stromelysin-1 by TIMP-1

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The NTR module: domains of netrins, secreted frizzled related proteins, and type I procollagen C-proteinase enhancer protein are homologous with tissue inhibitors of metalloproteases

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7 January 2021

Function of an axonal chemoattractant modulated by metalloprotease activity

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Spontaneous air space enlargement in the lungs of mice lacking tissue inhibitor of metalloproteinases-3 (TIMP-3).

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Murine TIMP-2 gene-targeted mutation

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Effects of gene deletion of the tissue inhibitor of the matrix metalloproteinase-type 1 (TIMP-1) on left ventricular geometry and function in mice

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Targeted inactivation of the mouse alpha 2-macroglobulin gene

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Mechanisms of angiogenesis and arteriogenesis

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MMP-9 supplied by bone marrow-derived cells contributes to skin carcinogenesis

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The role of matrix metalloproteinases in tumor angiogenesis and tumor metastasis

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RECK gene expression in hepatocellular carcinoma: correlation with invasion-related clinicopathological factors and its clinical significance. Reverse-inducing--cysteine-rich protein with Kazal motifs

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Multiple sequence alignment with hierarchical clustering

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10.1016/S0960-9822(02)00752-2

Identifiers

DOI

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3 August 2017

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