Mitochondrial free [Ca2+] increases during ATP/ADP antiport and ADP phosphorylation: exploration of mechanisms (Q34062193)

30 July 2017

title

Mitochondrial free [Ca2+] increases during ATP/ADP antiport and ADP phosphorylation: exploration of mechanisms (English)

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30 July 2017

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Johan Haumann

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30 July 2017

Ranjan K Dash

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30 July 2017

David F Stowe

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30 July 2017

Age D Boelens

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30 July 2017

Daniel A Beard

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30 July 2017

Amadou K S Camara

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30 July 2017

language of work or name

English

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publication date

1 August 2010

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30 July 2017

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inferred from page(s)

published in

Biophysical Journal

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30 July 2017

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30 July 2017

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30 July 2017

page(s)

997-1006

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Mitochondrial matrix K+ flux independent of large-conductance Ca2+-activated K+ channel opening

30 July 2017

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5 July 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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28 October 2018

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Controlled reperfusion after hypothermic heart preservation inhibits mitochondrial permeability transition-pore opening and enhances functional recovery

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Ca(2+) activation of heart mitochondrial oxidative phosphorylation: role of the F(0)/F(1)-ATPase

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Effect of Ca2+ on cardiac mitochondrial energy production is modulated by Na+ and H+ dynamics

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The Na(+)-independent Ca2+ efflux mechanism of liver mitochondria is not a passive Ca2+/2H+ exchanger

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Mitochondrial dehydrogenase activity affects adaptation of cardiac oxygen consumption to demand

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Intracellular Ca2+ increases the mitochondrial NADH concentration during elevated work in intact cardiac muscle

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Isocitrate as calcium ion activity buffer in coagulation assays

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10.1016/J.BPJ.2010.04.069

Identifiers

DOI

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30 July 2017

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30 July 2017

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