Activated microglia inhibit axonal growth through RGMa (Q28744661)

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scientific article (publication date: 2011)

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English	Activated microglia inhibit axonal growth through RGMa	scientific article (publication date: 2011)

Statements

instance of

scholarly article

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title

Activated microglia inhibit axonal growth through RGMa (English)

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main subject

microglia

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author

Toshihide Yamashita

object named as

Toshihide Yamashita

series ordinal

4

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author name string

Mari Kitayama

series ordinal

1

0 references

Masaki Ueno

series ordinal

2

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Toru Itakura

series ordinal

3

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language of work or name

English

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publication date

2011

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published in

PLOS One

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volume

6

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issue

9

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page(s)

e25234

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copyright license

Creative Commons Attribution 4.0 International

start time

21 September 2011

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stated in

April 2022 Public Data File from Crossref

copyright status

copyrighted

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describes a project that uses

ImageJ

1 reference

stated in

Europe PubMed Central

retrieved

11 June 2022

reference URL

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3177894/fullTextXML

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inferred from PubMed Central ID database lookup

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RGMa inhibition promotes axonal growth and recovery after spinal cord injury.

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Alternative activation of macrophages

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RGM is a repulsive guidance molecule for retinal axons

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Identification of two distinct macrophage subsets with divergent effects causing either neurotoxicity or regeneration in the injured mouse spinal cord

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EphA4 deficient mice maintain astroglial-fibrotic scar formation after spinal cord injury

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Strategies for regenerating injured axons after spinal cord injury - insights from brain development

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Another barrier to regeneration in the CNS: activated macrophages induce extensive retraction of dystrophic axons through direct physical interactions

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Minocycline treatment reduces delayed oligodendrocyte death, attenuates axonal dieback, and improves functional outcome after spinal cord injury.

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Minocycline, a tetracycline derivative, is neuroprotective against excitotoxicity by inhibiting activation and proliferation of microglia.

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RGMa modulates T cell responses and is involved in autoimmune encephalomyelitis

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Neuroprotection by minocycline facilitates significant recovery from spinal cord injury in mice

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Minocycline neuroprotects, reduces microgliosis, and inhibits caspase protease expression early after spinal cord injury

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Minocycline alleviates death of oligodendrocytes by inhibiting pro-nerve growth factor production in microglia after spinal cord injury.

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Minocycline inhibits apoptotic cell death via attenuation of TNF-alpha expression following iNOS/NO induction by lipopolysaccharide in neuron/glia co-cultures

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Lack of neuroprotective effects of simvastatin and minocycline in a model of cervical spinal cord injury.

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Activated macrophage/microglial cells can promote the regeneration of sensory axons into the injured spinal cord

1 reference

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retrieved

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based on heuristic

inferred from PubMed ID database lookup

Much Ado about Nogo

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/21957482

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

DOI

10.1371/JOURNAL.PONE.0025234

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2624731

ADS bibcode

2011PLoSO...625234K

0 references

OpenCitations bibliographic resource ID

2624731

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2624731

PMC publication ID

3177894

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2624731

PubMed publication ID

21957482

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2624731

ResearchGate publication ID

51679886

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