KCa3.1 channels mediate the increase of cell migration and proliferation by advanced glycation endproducts in cultured rat vascular smooth muscle cells (Q28573918)

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English	KCa3.1 channels mediate the increase of cell migration and proliferation by advanced glycation endproducts in cultured rat vascular smooth muscle cells	scientific journal article

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scholarly article

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PubMed publication ID

31 January 2017

KCa3.1 channels mediate the increase of cell migration and proliferation by advanced glycation endproducts in cultured rat vascular smooth muscle cells (English)

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PubMed publication ID

31 January 2017

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author name string

Li-Mei Zhao

1

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31 January 2017

Xing-Li Su

2

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31 January 2017

Yan Wang

3

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31 January 2017

Gui-Rong Li

4

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31 January 2017

Xiu-Ling Deng

5

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31 January 2017

publication date

1 February 2013

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31 January 2017

Laboratory Investigation

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31 January 2017

93

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31 January 2017

2

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31 January 2017

159–167

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31 January 2017

Blockade of the intermediate-conductance calcium-activated potassium channel as a new therapeutic strategy for restenosis

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https://api.crossref.org/works/10.1038/LABINVEST.2012.163

21 April 2017

The intermediate-conductance calcium-activated potassium channel KCa3.1 contributes to atherogenesis in mice and humans

1 reference

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21 April 2017

The integrin alpha2beta1 agonist, aggretin, promotes proliferation and migration of VSMC through NF-kB translocation and PDGF production

1 reference

https://api.crossref.org/works/10.1038/LABINVEST.2012.163

21 April 2017

Up-regulation of the IKCa1 potassium channel during T-cell activation. Molecular mechanism and functional consequences

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https://api.crossref.org/works/10.1038/LABINVEST.2012.163

21 April 2017

Advanced glycation end-products: a review

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

RAGE: a novel target for drug intervention in diabetic vascular disease

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Advanced protein glycosylation in diabetes and aging

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Morphologic findings of coronary atherosclerotic plaques in diabetics: a postmortem study

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

RAGE blockade stabilizes established atherosclerosis in diabetic apolipoprotein E-null mice.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

RAGE modulates vascular inflammation and atherosclerosis in a murine model of type 2 diabetes

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Central role of RAGE-dependent neointimal expansion in arterial restenosis

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Pathobiological role of advanced glycation endproducts via mitogen-activated protein kinase dependent pathway in the diabetic vasculopathy.

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Key role of Src kinase in S100B-induced activation of the receptor for advanced glycation end products in vascular smooth muscle cells

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Molecular cloning and characterization of the intermediate-conductance Ca(2+)-activated K(+) channel in vascular smooth muscle: relationship between K(Ca) channel diversity and smooth muscle cell function

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Mitogenic modulation of Ca2+ -activated K+ channels in proliferating A7r5 vascular smooth muscle cells

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Experimental diabetes mellitus down-regulates large-conductance Ca2+-activated K+ channels in cerebral artery smooth muscle and alters functional conductance

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Insulin-mediated upregulation of KCa3.1 channels promotes cell migration and proliferation in rat vascular smooth muscle

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Advanced glycation end product in familial amyloidotic polyneuropathy (FAP)

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Regulation of cell proliferation by intermediate-conductance Ca2+-activated potassium and volume-sensitive chloride channels in mouse mesenchymal stem cells.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Cell cycle-dependent expression of potassium channels and cell proliferation in rat mesenchymal stem cells from bone marrow.

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Formin mDia1 mediates vascular remodeling via integration of oxidative and signal transduction pathways.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Membrane depolarization mediates phosphorylation and nuclear translocation of CREB in vascular smooth muscle cells.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Store-operated Ca2+ entry activates the CREB transcription factor in vascular smooth muscle

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

betaig-h3 triggers signaling pathways mediating adhesion and migration of vascular smooth muscle cells through alphavbeta5 integrin

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Histidine phosphorylation of the potassium channel KCa3.1 by nucleoside diphosphate kinase B is required for activation of KCa3.1 and CD4 T cells

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Nucleoside diphosphate kinase B knock-out mice have impaired activation of the K+ channel KCa3.1, resulting in defective T cell activation.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Globotriaosylceramide leads to K(Ca)3.1 channel dysfunction: a new insight into endothelial dysfunction in Fabry disease.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

21 January 2018

Rat aortic smooth muscle cells expressing charybdotoxin-sensitive potassium channels exhibit enhanced proliferative responses

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

7 January 2021

based on heuristic

inferred from DOI database lookup

Upregulation of intermediate-conductance Ca2+-activated K+ channel (IKCa1) mediates phenotypic modulation of coronary smooth muscle

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https://api.crossref.org/works/10.1038%2FLABINVEST.2012.163

7 January 2021

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10.1038/LABINVEST.2012.163

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PubMed publication ID

31 January 2017

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PubMed publication ID

31 January 2017

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