P21-activated kinase 4 regulates the cyclin-dependent kinase inhibitor p57(kip2) in human breast cancer (Q28294949)

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Di Wang

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Hongyan Zhang

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Chunyu Wang

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Wei Dai

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Zhenguo Cheng

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Guanqiao Wang

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Feng Li

Argonaute-2 expression is regulated by epidermal growth factor receptor and mitogen-activated protein kinase signaling and correlates with a transformed phenotype in breast cancer cells

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language of work or name

English

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publication date

October 2013

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published in

The Anatomical Record

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volume

296

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issue

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page(s)

1561-7

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cites work

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Identification of PAK4 as a putative target gene for amplification within 19q13.12-q13.2 in oral squamous-cell carcinoma.

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CDK inhibitors: cell cycle regulators and beyond

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p57Kip2 is a downstream effector of BCR-ABL kinase inhibitors in chronic myelogenous leukemia cells.

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Pak4 induces premature senescence via a pathway requiring p16INK4/p19ARF and mitogen-activated protein kinase signaling

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Copy number alterations in pancreatic cancer identify recurrent PAK4 amplification

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MiR-221 controls CDKN1C/p57 and CDKN1B/p27 expression in human hepatocellular carcinoma

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MEK-ERK pathway regulates EZH2 overexpression in association with aggressive breast cancer subtypes

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Downregulation of p57 accelerates the growth and invasion of hepatocellular carcinoma

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The p57 CDKi integrates stress signals into cell-cycle progression to promote cell survival upon stress

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p57(KIP2) control of actin cytoskeleton dynamics is responsible for its mitochondrial pro-apoptotic effect.

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Myc-induced microRNAs integrate Myc-mediated cell proliferation and cell fate

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p21-activated kinases in cancer

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CDKN1C/p57kip2 is a candidate tumor suppressor gene in human breast cancer

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PAK as a therapeutic target in gastric cancer

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Nucleo-cytoplasmic shuttling of PAK4 modulates β-catenin intracellular translocation and signaling

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The pak4 protein kinase plays a key role in cell survival and tumorigenesis in athymic mice

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Ubiquitylation and proteasomal degradation of the p21(Cip1), p27(Kip1) and p57(Kip2) CDK inhibitors

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PAK4 is required for regulation of the cell‐cycle regulatory protein p21, and for control of cell‐cycle progression

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p57KIP2: "Kip"ing the cell under control.

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Activated PAK4 regulates cell adhesion and anchorage-independent growth

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p21-activated kinase 4 regulates ovarian cancer cell proliferation, migration, and invasion and contributes to poor prognosis in patients

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Akt and ERK control the proliferative response of mammary epithelial cells to the growth factors IGF-1 and EGF through the cell cycle inhibitor p57Kip2.

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CDKN1C (p57) is a direct target of EZH2 and suppressed by multiple epigenetic mechanisms in breast cancer cells

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