Inhibition of JNK by cellular stress- and tumor necrosis factor alpha-induced AKT2 through activation of the NF kappa B pathway in human epithelial Cells (Q28202566)

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scientific article (publication date: 16 August 2002)

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English	Inhibition of JNK by cellular stress- and tumor necrosis factor alpha-induced AKT2 through activation of the NF kappa B pathway in human epithelial Cells	scientific article (publication date: 16 August 2002)

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retracted paper

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Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

Inhibition of JNK by cellular stress- and tumor necrosis factor alpha-induced AKT2 through activation of the NF kappa B pathway in human epithelial Cells (English)

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Zeng-qiang Yuan

1

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Richard I Feldman

2

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Mei Sun

3

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Nancy E Olashaw

4

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Domenico Coppola

5

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Gene E Sussman

6

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Sue A Shelley

7

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Santo V Nicosia

8

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Jin Q Cheng

9

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language of work or name

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publication date

16 August 2002

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Journal of Biological Chemistry

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277

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33

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29973-82

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Ultraviolet light and osmotic stress: activation of the JNK cascade through multiple growth factor and cytokine receptors

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Mitogen-activated protein kinase cascades as regulators of stress responses

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A retroviral oncogene, akt, encoding a serine-threonine kinase containing an SH2-like region

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Molecular cloning and identification of a serine/threonine protein kinase of the second-messenger subfamily

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AKT2, a putative oncogene encoding a member of a subfamily of protein-serine/threonine kinases, is amplified in human ovarian carcinomas

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Identification of a human Akt3 (protein kinase B gamma) which contains the regulatory serine phosphorylation site.

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AKT/PKB and other D3 phosphoinositide-regulated kinases: kinase activation by phosphoinositide-dependent phosphorylation

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Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

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Regulation of cell death protease caspase-9 by phosphorylation

1 reference

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Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

1 reference

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NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase

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NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

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AFX-like Forkhead transcription factors mediate cell-cycle regulation by Ras and PKB through p27kip1

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BCR/ABL regulates expression of the cyclin-dependent kinase inhibitor p27Kip1 through the phosphatidylinositol 3-Kinase/AKT pathway

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Activation of RAC-protein kinase by heat shock and hyperosmolarity stress through a pathway independent of phosphatidylinositol 3-kinase

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The activation of protein kinase B by H2O2 or heat shock is mediated by phosphoinositide 3-kinase and not by mitogen-activated protein kinase-activated protein kinase-2

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Distinct phosphatidylinositol 3-kinase lipid products accumulate upon oxidative and osmotic stress and lead to different cellular responses

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Insulin resistance and a diabetes mellitus-like syndrome in mice lacking the protein kinase Akt2 (PKB beta)

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Growth retardation and increased apoptosis in mice with homozygous disruption of the Akt1 gene

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Akt1/PKBalpha is required for normal growth but dispensable for maintenance of glucose homeostasis in mice

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The phosphoinositide 3-OH kinase/AKT2 pathway as a critical target for farnesyltransferase inhibitor-induced apoptosis

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NF-kappaB as a primary regulator of the stress response

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Induction of gadd45beta by NF-kappaB downregulates pro-apoptotic JNK signalling.

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Inhibition of JNK activation through NF-kappaB target genes

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Transforming activity and mitosis-related expression of the AKT2 oncogene: evidence suggesting a link between cell cycle regulation and oncogenesis

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Akt2 mRNA is highly expressed in embryonic brown fat and the AKT2 kinase is activated by insulin

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Protein kinase B beta/Akt2 plays a specific role in muscle differentiation

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Phosphatidylinositol 3-kinase mediates epidermal growth factor-induced activation of the c-Jun N-terminal kinase signaling pathway.

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The activation of p38 and apoptosis by the inhibition of Erk is antagonized by the phosphoinositide 3-kinase/Akt pathway

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Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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Hsp72 functions as a natural inhibitory protein of c-Jun N-terminal kinase

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Deciphering the mammalian stress response - a stressful task

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Activation of protein kinase B induced by H(2)O(2) and heat shock through distinct mechanisms dependent and independent of phosphatidylinositol 3-kinase

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Frequent activation of AKT2 and induction of apoptosis by inhibition of phosphoinositide-3-OH kinase/Akt pathway in human ovarian cancer

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Stress-activated kinases regulate protein stability

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p38 mitogen-activated protein kinase mediates tumor necrosis factor-alpha-induced apoptosis in rat fetal brown adipocytes

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10.1074/JBC.M203636200

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