Id proteins at the cross-road of development and cancer (Q28200732)

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scientific article (publication date: 20 December 2001)

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English	Id proteins at the cross-road of development and cancer	scientific article (publication date: 20 December 2001)

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instance of

scholarly article

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Europe PubMed Central

title

Id proteins at the cross-road of development and cancer (English)

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author name string

A Lasorella

series ordinal

1

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T Uo

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2

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A Iavarone

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3

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language of work or name

English

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publication date

20 December 2001

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published in

Oncogene

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volume

20

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issue

58

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page(s)

8326-33

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cites work

Immortalization of primary human keratinocytes by the helix-loop-helix protein, Id-1

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Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts

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Regulation of the helix-loop-helix proteins, E2A and Id3, by the Ras-ERK MAPK cascade

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Id proteins control growth induction in mammalian cells

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Identification of Id4 as a regulator of BRCA1 expression by using a ribozyme-library-based inverse genomics approach

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Regulation of Id2 gene expression by the insulin-like growth factor I receptor requires signaling by phosphatidylinositol 3-kinase.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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The protein Id: a negative regulator of helix-loop-helix DNA binding proteins

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Defective repression of c-myc in breast cancer cells: A loss at the core of the transforming growth factor beta growth arrest program

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Expression of the dominant-negative regulator Id4 is induced during adipocyte differentiation

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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An Id-related helix-loop-helix protein encoded by a growth factor-inducible gene

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Regulation of Id3 Cell Cycle Function by Cdk-2-Dependent Phosphorylation

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Suppression of mammary epithelial cell differentiation by the helix-loop-helix protein Id-1.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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A novel pathway for mammary epithelial cell invasion induced by the helix-loop-helix protein Id-1.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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The regulation of E2F by pRB-family proteins

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Proliferation, cell cycle and apoptosis in cancer

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Id2 promotes apoptosis by a novel mechanism independent of dimerization to basic helix-loop-helix factors

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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The Myc/Max/Mad network and the transcriptional control of cell behavior

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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The Hallmarks of Cancer

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Cdk2-dependent phosphorylation of Id2 modulates activity of E2A-related transcription factors

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Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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The helix-loop-helix protein Id-2 enhances cell proliferation and binds to the retinoblastoma protein

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Each member of the Id gene family exhibits a unique expression pattern in mouse gastrulation and neurogenesis

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Advances in the diagnosis and treatment of neuroblastoma

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The helix-loop-helix transcription factor, Id-1, is overexpressed in medullary thyroid cancer

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Id3 inhibits B lymphocyte progenitor growth and survival in response to TGF-beta

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Massive apoptosis of thymocytes in T-cell-deficient Id1 transgenic mice

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Negative regulation of the growth-promoting transcription factor E2F-1 by a stably bound cyclin A-dependent protein kinase

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Id2 specifically alters regulation of the cell cycle by tumor suppressor proteins

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Id2 is a retinoblastoma protein target and mediates signalling by Myc oncoproteins

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Dual roles of the retinoblastoma protein in cell cycle regulation and neuron differentiation

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Impaired recruitment of bone-marrow-derived endothelial and hematopoietic precursor cells blocks tumor angiogenesis and growth

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Molecular biology of neuroblastoma

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Neural crest specification regulated by the helix-loop-helix repressor Id2.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Id-1 and Id-2 are overexpressed in pancreatic cancer and in dysplastic lesions in chronic pancreatitis

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Lactation defect in mice lacking the helix-loop-helix inhibitor Id2

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Overexpression of the Helix-Loop-Helix protein Id2 blocks T cell development at multiple stages.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Phosphorylation of helix-loop-helix proteins ID1, ID2 and ID3

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Activation of helix-loop-helix proteins Id1, Id2 and Id3 during neural differentiation

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Neuronal expression of regulatory helix-loop-helix factor Id2 gene in mouse

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Coupling of cell growth control and apoptosis functions of Id proteins

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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Id helix-loop-helix proteins antagonize pax transcription factor activity by inhibiting DNA binding

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Cancer cell cycles

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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A critical temporal requirement for the retinoblastoma protein family during neuronal determination

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Id1 expression is associated with histological grade and invasive behavior in endometrial carcinoma

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Evidence that helix-loop-helix proteins collaborate with retinoblastoma tumor suppressor protein to regulate cortical neurogenesis

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Embryonic development and pediatric oncogenesis.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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A role for the helix-loop-helix protein Id2 in the control of oligodendrocyte development

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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7 January 2021

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The retinoblastoma protein and cell cycle control

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Forced expression of Id-1 in the adult mouse small intestinal epithelium is associated with development of adenomas

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Id helix-loop-helix proteins inhibit nucleoprotein complex formation by the TCF ETS-domain transcription factors

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Development of peripheral lymphoid organs and natural killer cells depends on the helix-loop-helix inhibitor Id2.

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https://api.crossref.org/works/10.1038%2FSJ.ONC.1205093

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Identifiers

DOI

10.1038/SJ.ONC.1205093

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stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4905809

Dimensions Publication ID

1047741799

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OpenCitations bibliographic resource ID

4905809

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4905809

PubMed publication ID

11840325

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

4905809

ResearchGate publication ID

11521287

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