FUS transgenic rats develop the phenotypes of amyotrophic lateral sclerosis and frontotemporal lobar degeneration (Q27342067)

0 references

Jianbin Tong

0 references

Han Chen

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Yong-Jian Liu

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Dian Wang

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Xiaotao Wei

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Xu-Gang Xia

Creative Commons Attribution 4.0 International

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language of work or name

English

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publication date

March 2011

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e1002011

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start time

3 March 2011

1 reference

April 2022 Public Data File from Crossref

copyrighted

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cites work

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21 March 2017

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Oncoprotein TLS interacts with serine-arginine proteins involved in RNA splicing

21 March 2017

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21 March 2017

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Ubiquitinated TDP-43 in frontotemporal lobar degeneration and amyotrophic lateral sclerosis

21 March 2017

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Mutations in FUS, an RNA processing protein, cause familial amyotrophic lateral sclerosis type 6

21 March 2017

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Mutations in the FUS/TLS gene on chromosome 16 cause familial amyotrophic lateral sclerosis

21 March 2017

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TLS facilitates transport of mRNA encoding an actin-stabilizing protein to dendritic spines

21 March 2017

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21 March 2017

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Sporadic amyotrophic lateral sclerosis of long duration mimicking spinal progressive muscular atrophy exists: additional autopsy case with a clinical course of 19 years

7 April 2017

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TLS (FUS) binds RNA in vivo and engages in nucleo-cytoplasmic shuttling

21 March 2017

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Delocalization of the multifunctional RNA splicing factor TLS/FUS in hippocampal neurones: exclusion from the nucleus and accumulation in dendritic granules and spine heads

21 March 2017

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Onset and progression in inherited ALS determined by motor neurons and microglia

21 March 2017

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Amyotrophic lateral sclerosis-associated proteins TDP-43 and FUS/TLS function in a common biochemical complex to co-regulate HDAC6 mRNA.

7 April 2017

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Frameshift and novel mutations in FUS in familial amyotrophic lateral sclerosis and ALS/dementia

29 September 2017

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Elevated expression of TDP-43 in the forebrain of mice is sufficient to cause neurological and pathological phenotypes mimicking FTLD-U.

29 September 2017

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FUS-immunoreactive inclusions are a common feature in sporadic and non-SOD1 familial amyotrophic lateral sclerosis

29 September 2017

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FUS pathology defines the majority of tau- and TDP-43-negative frontotemporal lobar degeneration

29 September 2017

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29 September 2017

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TDP-43 transgenic mice develop spastic paralysis and neuronal inclusions characteristic of ALS and frontotemporal lobar degeneration

29 September 2017

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Frequency of ubiquitin and FUS-positive, TDP-43-negative frontotemporal lobar degeneration

29 September 2017

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Clinical entity of frontotemporal dementia with motor neuron disease

29 September 2017

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29 September 2017

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Frontotemporal dementia and motor neurone disease: overlapping clinic-pathological disorders

29 September 2017

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TARDBP mutations in motoneuron disease with frontotemporal lobar degeneration

29 September 2017

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Developing tTA transgenic rats for inducible and reversible gene expression

29 September 2017

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Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS mice.

29 September 2017

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Spatial and nonspatial escape strategies in the Barnes maze

29 September 2017

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Frontotemporal dementia: clinicopathological correlations

29 September 2017

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Hippocampal atrophy on MRI in frontotemporal lobar degeneration and Alzheimer's disease

29 September 2017

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Male sterility and enhanced radiation sensitivity in TLS(-/-) mice

29 September 2017

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A topogenic role for the oncogenic N-terminus of TLS: nucleolar localization when transcription is inhibited

29 September 2017

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Epigenetic regulation of neuron-dependent induction of astroglial synaptic protein GLT1.

29 September 2017

1 reference

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Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common feature

2 June 2018

1 reference

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The RNA binding protein TLS is translocated to dendritic spines by mGluR5 activation and regulates spine morphology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

The chimeric FUS/TLS-CHOP fusion protein specifically induces liposarcomas in transgenic mice.

21 January 2018

1 reference

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Myosin-Va facilitates the accumulation of mRNA/protein complex in dendritic spines.

21 January 2018

1 reference

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Fus deficiency in mice results in defective B-lymphocyte development and activation, high levels of chromosomal instability and perinatal death.

21 January 2018

1 reference

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Genetic contribution of FUS to frontotemporal lobar degeneration

21 January 2018

1 reference

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TDP-43 mutation in familial amyotrophic lateral sclerosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

Sustained expression of TDP-43 and FUS in motor neurons in rodent's lifetime.

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

Atypical frontotemporal lobar degeneration with ubiquitin-positive, TDP-43-negative neuronal inclusions

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

Sporadic amyotrophic lateral sclerosis of long duration is associated with relatively mild TDP-43 pathology.

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

Simvastatin impairs spatial memory in rats at a specific dose level

21 January 2018

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PGEN.1002011

C-terminal FUS/TLS mutations in familial and sporadic ALS in Germany

21 January 2018

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3048370

Sporadic amyotrophic lateral sclerosis with multiple system degeneration: a report of an autopsy case without respirator administration

27 November 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/21408206

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1371/JOURNAL.PGEN.1002011

1 reference

1 reference

1 reference

PubMed publication ID

21408206

1 reference