The insulin receptor is essential for virus-induced tumorigenesis of Kaposi's sarcoma (Q24304458)

GOA release 2020-03-11

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P A Carroll

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author name string

P P Rose

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J M Carroll

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V R DeFilippis

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M Lagunoff

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A V Moses

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C T Roberts

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K Früh

The beta-core fragment of human chorionic gonadotrophin inhibits growth of Kaposi's sarcoma-derived cells and a new immortalized Kaposi's sarcoma cell line

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language of work or name

English

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publication date

29 March 2007

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1995-2005

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Isolation and Characterization of an Immortal Neoplastic Cell Line (KS Y-1) From AIDS-Associated Kaposi's Sarcoma

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Kaposi's sarcoma-associated herpesvirus-induced upregulation of the c-kit proto-oncogene, as identified by gene expression profiling, is essential for the transformation of endothelial cells

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Design of a selective insulin receptor tyrosine kinase inhibitor and its effect on glucose uptake and metabolism in intact cells

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In IGF-I receptor-deficient leiomyosarcoma cells autocrine IGF-II induces cell invasion and protection from apoptosis via the insulin receptor isoform A.

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ERK1/2 and MEK1/2 induced by Kaposi's sarcoma-associated herpesvirus (human herpesvirus 8) early during infection of target cells are essential for expression of viral genes and for establishment of infection

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Kaposi sarcoma herpesvirus-induced cellular reprogramming contributes to the lymphatic endothelial gene expression in Kaposi sarcoma.

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Kaposi's sarcoma-associated herpesvirus infects endothelial and spindle cells

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Absence of human herpesvirus 8 DNA sequences in neoplastic Kaposi's sarcoma cell lines

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Inefficient establishment of KSHV latency suggests an additional role for continued lytic replication in Kaposi sarcoma pathogenesis.

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Insulin receptor activation by IGF-II in breast cancers: evidence for a new autocrine/paracrine mechanism

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Kaposi's sarcoma-associated herpesvirus gene expression in endothelial (spindle) tumor cells

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