A splice variant of stress response gene ATF3 counteracts NF-kappaB-dependent anti-apoptosis through inhibiting recruitment of CREB-binding protein/p300 coactivator (Q24295232)

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English	A splice variant of stress response gene ATF3 counteracts NF-kappaB-dependent anti-apoptosis through inhibiting recruitment of CREB-binding protein/p300 coactivator	scientific article

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scholarly article

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A splice variant of stress response gene ATF3 counteracts NF-kappaB-dependent anti-apoptosis through inhibiting recruitment of CREB-binding protein/p300 coactivator (English)

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apoptotic process

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author name string

Bayin Hua

1

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Mimi Tamamori-Adachi

2

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Yang Luo

3

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Kiyoshi Tamura

4

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Masaki Morioka

5

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Mizue Fukuda

6

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Yujiro Tanaka

7

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Shigetaka Kitajima

8

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language of work or name

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publication date

20 January 2006

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Journal of Biological Chemistry

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281

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3

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1620-9

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Transgenic mice with cardiac-specific expression of activating transcription factor 3, a stress-inducible gene, have conduction abnormalities and contractile dysfunction

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Stress response gene ATF3 is a target of c-myc in serum-induced cell proliferation

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gadd153/Chop10, a Potential Target Gene of the Transcriptional Repressor ATF3

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Amino acid deprivation and endoplasmic reticulum stress induce expression of multiple activating transcription factor-3 mRNA species that, when overexpressed in HepG2 cells, modulate transcription by the human asparagine synthetase promoter

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An alternatively spliced isoform of transcriptional repressor ATF3 and its induction by stress stimuli

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NF-kappaB at the crossroads of life and death

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IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases

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Regulation of NF-kappaB by cyclin-dependent kinases associated with the p300 coactivator

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Transcriptional activation by NF-kappaB requires multiple coactivators

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Steroid receptor coactivator-1 interacts with the p50 subunit and coactivates nuclear factor kappaB-mediated transactivations

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Expression of the activating transcription factor 3 prevents c-Jun N-terminal kinase-induced neuronal death by promoting heat shock protein 27 expression and Akt activation.

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Identifiers

10.1074/JBC.M508471200

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