Pages that link to "Q39927199"

The following pages link to FUS/TLS deficiency causes behavioral and pathological abnormalities distinct from amyotrophic lateral sclerosis (Q39927199):

Displaying 32 items.

• From animal models to human disease: a genetic approach for personalized medicine in ALS (Q26741250) (← links)
• Pathogenesis of FUS-associated ALS and FTD: insights from rodent models (Q28067147) (← links)
• Characterization of genetic loss-of-function of Fus in zebrafish. (Q30836650) (← links)
• Motor neuron intrinsic and extrinsic mechanisms contribute to the pathogenesis of FUS-associated amyotrophic lateral sclerosis (Q30848999) (← links)
• A comprehensive analysis of rare genetic variation in amyotrophic lateral sclerosis in the UK (Q33729908) (← links)
• Toxic gain of function from mutant FUS protein is crucial to trigger cell autonomous motor neuron loss (Q36906420) (← links)
• Differential roles of NF-Y transcription factor in ER chaperone expression and neuronal maintenance in the CNS (Q37298844) (← links)
• FUS/TLS acts as an aggregation-dependent modifier of polyglutamine disease model mice (Q37339987) (← links)
• Roles for RNA-binding proteins in development and disease (Q38772987) (← links)
• Physiological functions and pathobiology of TDP-43 and FUS/TLS proteins (Q38789192) (← links)
• Role of FET proteins in neurodegenerative disorders (Q38896526) (← links)
• FET proteins regulate lifespan and neuronal integrity (Q40681429) (← links)
• Dendritic Homeostasis Disruption in a Novel Frontotemporal Dementia Mouse Model Expressing Cytoplasmic Fused in Sarcoma (Q42651961) (← links)
• De novo design of RNA-binding proteins with a prion-like domain related to ALS/FTD proteinopathies (Q47163862) (← links)
• RNA binding proteins and the pathological cascade in ALS/FTD neurodegeneration (Q47423589) (← links)
• FUS Mutant Human Motoneurons Display Altered Transcriptome and microRNA Pathways with Implications for ALS Pathogenesis. (Q47680582) (← links)
• Causative Genes in Amyotrophic Lateral Sclerosis and Protein Degradation Pathways: a Link to Neurodegeneration. (Q48265316) (← links)
• Mislocated FUS is sufficient for gain-of-toxic-function amyotrophic lateral sclerosis phenotypes in mice (Q48645659) (← links)
• The Role of Post-Translational Modifications on Prion-Like Aggregation and Liquid-Phase Separation of FUS. (Q54967377) (← links)
• Importance of Functional Loss of FUS in FTLD/ALS. (Q55343691) (← links)
• genetically interacts with the ALS-associated orthologue and mediates its toxicity (Q58700147) (← links)
• Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis (Q58751835) (← links)
• From Mouse Models to Human Disease: An Approach for Amyotrophic Lateral Sclerosis (Q58765919) (← links)
• RNA Dysregulation in Amyotrophic Lateral Sclerosis (Q61811636) (← links)
• Overriding FUS autoregulation in mice triggers gain-of-toxic dysfunctions in RNA metabolism and autophagy-lysosome axis (Q64228212) (← links)
• Pathogenesis of Frontotemporal Lobar Degeneration: Insights From Loss of Function Theory and Early Involvement of the Caudate Nucleus. (Q64983354) (← links)
• The Overlapping Genetics of Amyotrophic Lateral Sclerosis and Frontotemporal Dementia (Q89963639) (← links)
• Conditional depletion of Fus in oligodendrocytes leads to motor hyperactivity and increased myelin deposition associated with Akt and cholesterol activation (Q90428731) (← links)
• ALS Genetics, Mechanisms, and Therapeutics: Where Are We Now? (Q92187628) (← links)
• FUS pathology in ALS is linked to alterations in multiple ALS-associated proteins and rescued by drugs stimulating autophagy (Q92773828) (← links)
• New Mechanisms of DNA Repair Defects in Fused in Sarcoma-Associated Neurodegeneration: Stage Set for DNA Repair-Based Therapeutics? (Q92886481) (← links)
• ALS Genetics: Gains, Losses, and Implications for Future Therapies (Q99547009) (← links)