Pages that link to "Q39662062"
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The following pages link to The C9orf72 protein interacts with Rab1a and the ULK1 complex to regulate initiation of autophagy (Q39662062):
Displaying 50 items.
- C9orf72-SMCR8 complex subunit (Q21102398) (← links)
- RAB1A, member RAS oncogene family (Q21113198) (← links)
- RB1 inducible coiled-coil 1 (Q21115135) (← links)
- Unc-51 like autophagy activating kinase 1 (Q21116760) (← links)
- C9orf72's Interaction with Rab GTPases-Modulation of Membrane Traffic and Autophagy (Q28069365) (← links)
- Multiplex image-based autophagy RNAi screening identifies SMCR8 as ULK1 kinase activity and gene expression regulator (Q28854564) (← links)
- A C9ORF72/SMCR8-containing complex regulates ULK1 and plays a dual role in autophagy (Q29465530) (← links)
- Loss of C9orf72 Enhances Autophagic Activity via Deregulated mTOR and TFEB Signaling (Q29465544) (← links)
- C9ORF72 hexanucleotide repeat exerts toxicity in a stable, inducible motor neuronal cell model, which is rescued by partial depletion of Pten (Q33614811) (← links)
- Protein Homeostasis in Amyotrophic Lateral Sclerosis: Therapeutic Opportunities? (Q33621868) (← links)
- Protein Quality Control and the Amyotrophic Lateral Sclerosis/Frontotemporal Dementia Continuum (Q33653996) (← links)
- Modelling amyotrophic lateral sclerosis: progress and possibilities (Q33746289) (← links)
- SRSF1-dependent nuclear export inhibition of C9ORF72 repeat transcripts prevents neurodegeneration and associated motor deficits (Q33891056) (← links)
- Look who's talking-the crosstalk between oxidative stress and autophagy supports exosomal-dependent release of HCV particles (Q36227134) (← links)
- The essential and downstream common proteins of amyotrophic lateral sclerosis: A protein-protein interaction network analysis. (Q36304197) (← links)
- Loss of C9orf72 function leads to autoimmunity (Q37666980) (← links)
- Modeling the C9ORF72 repeat expansion mutation using human induced pluripotent stem cells (Q38741408) (← links)
- Pathogenic determinants and mechanisms of ALS/FTD linked to hexanucleotide repeat expansions in the C9orf72 gene (Q38952327) (← links)
- Decoding ALS: from genes to mechanism. (Q39004348) (← links)
- Rabs, Membrane Dynamics, and Parkinson's Disease (Q39031304) (← links)
- Could Sirtuin Activities Modify ALS Onset and Progression? (Q39036105) (← links)
- Sec16 in conventional and unconventional exocytosis: Working at the interface of membrane traffic and secretory autophagy? (Q39119334) (← links)
- Neurobiology of axonal transport defects in motor neuron diseases: Opportunities for translational research? (Q39149919) (← links)
- C9orf72: At the intersection of lysosome cell biology and neurodegenerative disease (Q39165154) (← links)
- Mechanisms of Autophagy Initiation (Q39182119) (← links)
- Autophagy and Its Impact on Neurodegenerative Diseases: New Roles for TDP-43 and C9orf72. (Q39370811) (← links)
- Multifaceted role of SMCR8 as autophagy regulator. (Q39428501) (← links)
- RNA Misprocessing in C9orf72-Linked Neurodegeneration (Q39458329) (← links)
- C9ORF72 is a GDP/GTP exchange factor for Rab8 and Rab39 and regulates autophagy (Q39520699) (← links)
- The Enigmatic Role of C9ORF72 in Autophagy (Q41222674) (← links)
- Autophagy Dysregulation in ALS: When Protein Aggregates Get Out of Hand (Q41525479) (← links)
- Proteostasis disturbance in amyotrophic lateral sclerosis (Q41991266) (← links)
- Autophagy-Independent Lysosomal Targeting Regulated by ULK1/2-FIP200 and ATG9. (Q42514504) (← links)
- Endocytosis regulates TDP-43 toxicity and turnover (Q47108096) (← links)
- Is amyotrophic lateral sclerosis/frontotemporal dementia an autophagy disease? (Q47108927) (← links)
- The emerging role of Rab GTPases in the pathogenesis of Parkinson's disease (Q47217972) (← links)
- Unaffected mosaic C9orf72 case: RNA foci, dipeptide proteins, but upregulated C9orf72 expression (Q47250883) (← links)
- Consequences of Rab GTPase dysfunction in genetic or acquired human diseases (Q47294747) (← links)
- The mammalian ULK1 complex and autophagy initiation. (Q47301180) (← links)
- A proteomic network approach across the ALS-FTD disease spectrum resolves clinical phenotypes and genetic vulnerability in human brain. (Q47344945) (← links)
- C9orf72 poly GA RAN-translated protein plays a key role in amyotrophic lateral sclerosis via aggregation and toxicity (Q47707045) (← links)
- Amyotrophic lateral sclerosis-associated mutant SOD1 inhibits anterograde axonal transport of mitochondria by reducing Miro1 levels (Q47707422) (← links)
- Current Advances and Limitations in Modeling ALS/FTD in a Dish Using Induced Pluripotent Stem Cells (Q47868914) (← links)
- Dysregulated molecular pathways in amyotrophic lateral sclerosis-frontotemporal dementia spectrum disorder. (Q47946104) (← links)
- Haploinsufficiency leads to neurodegeneration in C9ORF72 ALS/FTD human induced motor neurons. (Q48106083) (← links)
- TRAPPC13 modulates autophagy and the response to Golgi stress. (Q48110705) (← links)
- Distinct roles for motor neuron autophagy early and late in the SOD1G93A mouse model of ALS. (Q48114502) (← links)
- A Molecular Perspective of Mammalian Autophagosome Biogenesis (Q48163587) (← links)
- Causative Genes in Amyotrophic Lateral Sclerosis and Protein Degradation Pathways: a Link to Neurodegeneration. (Q48265316) (← links)
- Unraveling the Role of RNA Mediated Toxicity of C9orf72 Repeats in C9-FTD/ALS. (Q49314473) (← links)