Pages that link to "Q39342489"
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The following pages link to A new type of microglia gene targeting shows TAK1 to be pivotal in CNS autoimmune inflammation (Q39342489):
Displaying 50 items.
- Oligodendrocyte, Astrocyte, and Microglia Crosstalk in Myelin Development, Damage, and Repair (Q26739719) (← links)
- Signaling in Effector Lymphocytes: Insights toward Safer Immunotherapy (Q26744654) (← links)
- Linking Activation of Microglia and Peripheral Monocytic Cells to the Pathophysiology of Psychiatric Disorders (Q26745417) (← links)
- Double Roles of Macrophages in Human Neuroimmune Diseases and Their Animal Models (Q26749131) (← links)
- Infection as an Environmental Trigger of Multiple Sclerosis Disease Exacerbation (Q26779416) (← links)
- Immunosuppressive Mechanisms of Malignant Gliomas: Parallels at Non-CNS Sites (Q26798402) (← links)
- Glia-neuron interactions in the mammalian retina (Q26827435) (← links)
- From the Cajal alumni Achúcarro and Río-Hortega to the rediscovery of never-resting microglia (Q26851257) (← links)
- Persistence of Toxoplasma gondii in the central nervous system: a fine-tuned balance between the parasite, the brain and the immune system (Q27026477) (← links)
- Origin, fate and dynamics of macrophages at central nervous system interfaces (Q27315656) (← links)
- Microglia protect against brain injury and their selective elimination dysregulates neuronal network activity after stroke. (Q27317371) (← links)
- Astrocyte galectin-9 potentiates microglial TNF secretion (Q28567332) (← links)
- LRP1 expression in microglia is protective during CNS autoimmunity (Q30276379) (← links)
- Macrophages: An Inflammatory Link Between Angiogenesis and Lymphangiogenesis (Q30278005) (← links)
- Unexpected cellular players in Rett syndrome pathology (Q30299929) (← links)
- Methyl-CpG Binding Protein 2 Regulates Microglia and Macrophage Gene Expression in Response to Inflammatory Stimuli (Q30300139) (← links)
- Microglia contribute to circuit defects in Mecp2 null mice independent of microglia-specific loss of Mecp2 expression (Q30357465) (← links)
- The role of microglia in human disease: therapeutic tool or target? (Q34038205) (← links)
- Interacting chemokine signals regulate dendritic cells in acute brain injury (Q34083673) (← links)
- Stem cells in neuroinjury and neurodegenerative disorders: challenges and future neurotherapeutic prospects. (Q34095999) (← links)
- The p38alpha mitogen-activated protein kinase limits the CNS proinflammatory cytokine response to systemic lipopolysaccharide, potentially through an IL-10 dependent mechanism (Q34324047) (← links)
- Progressive replacement of embryo-derived cardiac macrophages with age (Q34375144) (← links)
- Microglia: unique and common features with other tissue macrophages (Q34411277) (← links)
- Host microbiota constantly control maturation and function of microglia in the CNS. (Q34478756) (← links)
- Ontogeny and polarization of macrophages in inflammation: blood monocytes versus tissue macrophages (Q35000376) (← links)
- Metabolic reprograming of mononuclear phagocytes in progressive multiple sclerosis. (Q35167056) (← links)
- Infiltration of circulating myeloid cells through CD95L contributes to neurodegeneration in mice (Q35289164) (← links)
- Involvement of neuronal TGF-β activated kinase 1 in the development of tolerance to morphine-induced antinociception in rat spinal cord (Q35631488) (← links)
- USP18 lack in microglia causes destructive interferonopathy of the mouse brain (Q35762623) (← links)
- Fibronectin Matrix Assembly after Spinal Cord Injury (Q35866061) (← links)
- TGFβ-activated Kinase 1 (TAK1) Inhibition by 5Z-7-Oxozeaenol Attenuates Early Brain Injury after Experimental Subarachnoid Hemorrhage (Q35925884) (← links)
- Interferon-beta signaling in retinal mononuclear phagocytes attenuates pathological neovascularization. (Q36006004) (← links)
- Communicating systems in the body: how microbiota and microglia cooperate. (Q36071876) (← links)
- Brain endothelial TAK1 and NEMO safeguard the neurovascular unit (Q36080694) (← links)
- TAK1 inhibition prevents the development of autoimmune diabetes in NOD mice (Q36149886) (← links)
- Control of IL-17 receptor signaling and tissue inflammation by the p38α-MKP-1 signaling axis in a mouse model of multiple sclerosis (Q36267838) (← links)
- Retinoid X receptor activation reverses age-related deficiencies in myelin debris phagocytosis and remyelination (Q36344854) (← links)
- Wild-type microglia do not reverse pathology in mouse models of Rett syndrome (Q36386922) (← links)
- Fate mapping reveals that microglia and recruited monocyte-derived macrophages are definitively distinguishable by phenotype in the retina (Q36560105) (← links)
- T cell-derived interleukin-10 is an important regulator of the Th17 response during lethal alphavirus encephalomyelitis (Q36945768) (← links)
- TAK1 determines susceptibility to endoplasmic reticulum stress and leptin resistance in the hypothalamus. (Q36970891) (← links)
- Decrease of VEGF-A in myeloid cells attenuates glioma progression and prolongs survival in an experimental glioma model (Q36977700) (← links)
- On-demand erythrocyte disposal and iron recycling requires transient macrophages in the liver (Q37117562) (← links)
- Dorsal horn neurons release extracellular ATP in a VNUT-dependent manner that underlies neuropathic pain. (Q37188320) (← links)
- Loss of SOCS3 in myeloid cells prolongs survival in a syngeneic model of glioma. (Q37190394) (← links)
- A novel P2X4 receptor-selective antagonist produces anti-allodynic effect in a mouse model of herpetic pain (Q37220013) (← links)
- Microglial CX3CR1 promotes adult neurogenesis by inhibiting Sirt 1/p65 signaling independent of CX3CL1 (Q37263310) (← links)
- TGFβ regulates persistent neuroinflammation by controlling Th1 polarization and ROS production via monocyte-derived dendritic cells (Q37316413) (← links)
- LYN- and AIRE-mediated tolerance checkpoint defects synergize to trigger organ-specific autoimmunity (Q37381383) (← links)
- Caspase-8 inhibition represses initial human monocyte activation in septic shock model (Q37437178) (← links)