Pages that link to "Q38913800"
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The following pages link to YB-1 overexpression promotes a TGF-β1-induced epithelial-mesenchymal transition via Akt activation (Q38913800):
Displaying 13 items.
- YBX1/YB-1 induces partial EMT and tumourigenicity through secretion of angiogenic factors into the extracellular microenvironment. (Q35955424) (← links)
- Interplay between YB-1 and IL-6 promotes the metastatic phenotype in breast cancer cells (Q36546008) (← links)
- Nuclear expression of Y-box binding protein-1 is associated with poor prognosis in patients with pancreatic cancer and its knockdown inhibits tumor growth and metastasis in mice tumor models (Q38293424) (← links)
- Y-box-binding protein-1 (YB-1) promotes cell proliferation, adhesion and drug resistance in diffuse large B-cell lymphoma (Q38758924) (← links)
- CD4 + T cells promote renal cell carcinoma proliferation via modulating YBX1. (Q49385987) (← links)
- Hyper-O-GlcNAcylation of YB-1 affects Ser102 phosphorylation and promotes cell proliferation in hepatocellular carcinoma. (Q51383074) (← links)
- The Role of YB1 in Renal Cell Carcinoma Cell Adhesion (Q57135008) (← links)
- The long noncoding RNA LINC00312 induces lung adenocarcinoma migration and vasculogenic mimicry through directly binding YBX1 (Q59812694) (← links)
- Y-box binding protein-1 promotes tumorigenesis and progression via the epidermal growth factor receptor/AKT pathway in spinal chordoma (Q60946494) (← links)
- Elevated expression of G3BP1 associates with YB1 and p-AKT and predicts poor prognosis in nonsmall cell lung cancer patients after surgical resection (Q90326233) (← links)
- Downregulation of miR-216a-5p by long noncoding RNA PVT1 suppresses colorectal cancer progression via modulation of YBX1 expression (Q92799038) (← links)
- Y-box binding protein 1 acts as a negative regulator of stearoyl CoA desaturase 1 in clear cell renal cell carcinoma (Q99579047) (← links)
- A novel β2-AR/YB-1/β-catenin axis mediates chronic stress-associated metastasis in hepatocellular carcinoma (Q99710794) (← links)