Pages that link to "Q37466115"

The following pages link to The mitochondrial permeability transition pore: a molecular target for amyotrophic lateral sclerosis therapy (Q37466115):

Displaying 30 items.

• Olesoxime, a cholesterol-like neuroprotectant for the potential treatment of amyotrophic lateral sclerosis (Q24596378) (← links)
• Dynamin-related protein 1 and mitochondrial fragmentation in neurodegenerative diseases (Q28300234) (← links)
• Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice (Q33832905) (← links)
• Mitochondrial pathobiology in ALS. (Q34038775) (← links)
• Oxidative stress, mitochondrial damage and neurodegenerative diseases (Q34094532) (← links)
• Laminarin induces apoptosis of human colon cancer LOVO cells through a mitochondrial pathway. (Q34385983) (← links)
• Inducible nitric oxide synthase is present in motor neuron mitochondria and Schwann cells and contributes to disease mechanisms in ALS mice (Q34441113) (← links)
• Mitochondrial and Cell Death Mechanisms in Neurodegenerative Diseases (Q34502221) (← links)
• GNX-4728, a novel small molecule drug inhibitor of mitochondrial permeability transition, is therapeutic in a mouse model of amyotrophic lateral sclerosis (Q34741619) (← links)
• Repetitive nerve stimulation transiently opens the mitochondrial permeability transition pore in motor nerve terminals of symptomatic mutant SOD1 mice (Q34819311) (← links)
• Apoptosis of human gastric cancer SGC-7901 cells induced by podophyllotoxin (Q35190172) (← links)
• Translocator protein (TSPO) role in aging and Alzheimer's disease. (Q35618035) (← links)
• Human coronavirus-induced neuronal programmed cell death is cyclophilin d dependent and potentially caspase dispensable (Q35666094) (← links)
• Mitochondria as a Therapeutic Target for Aging and Neurodegenerative Diseases (Q35807409) (← links)
• Transient mitochondrial permeability transition mediates excitotoxicity in glutamate-sensitive NSC34D motor neuron-like cells (Q36101596) (← links)
• Oxidative stress modulates mitochondrial failure and cyclophilin D function in X-linked adrenoleukodystrophy (Q36474703) (← links)
• Biology of mitochondria in neurodegenerative diseases (Q36488715) (← links)
• Basic mechanisms of neurodegeneration: a critical update (Q37673274) (← links)
• Low molecular weight phospholipases A2 in mammalian brain and neural cells: roles in functions and dysfunctions (Q37711265) (← links)
• Neuronal cell death in neonatal hypoxia-ischemia (Q37724332) (← links)
• Mitochondrial channels: ion fluxes and more (Q38201407) (← links)
• The ER mitochondria calcium cycle and ER stress response as therapeutic targets in amyotrophic lateral sclerosis. (Q38218666) (← links)
• Mitochondrial oxidative stress and dysfunction in arsenic neurotoxicity: A review. (Q38618563) (← links)
• A mitochondrial-targeted cyclosporin A with high binding affinity for cyclophilin D yields improved cytoprotection of cardiomyocytes. (Q40478827) (← links)
• Mitochondrial Perturbation in Alzheimer's Disease and Diabetes (Q48284335) (← links)
• Retinal genes are differentially expressed in areas of primary versus secondary degeneration following partial optic nerve injury (Q50051397) (← links)
• P53/Drp1-dependent mitochondrial fission mediates aldosterone-induced podocyte injury and mitochondrial dysfunction (Q50918245) (← links)
• Swim Training Modulates Skeletal Muscle Energy Metabolism, Oxidative Stress, and Mitochondrial Cholesterol Content in Amyotrophic Lateral Sclerosis Mice. (Q55197953) (← links)
• Altered Slc25 family gene expression as markers of mitochondrial dysfunction in brain regions under experimental mixed anxiety/depression-like disorder (Q59789193) (← links)
• Swim Training Modulates Mouse Skeletal Muscle Energy Metabolism and Ameliorates Reduction in Grip Strength in a Mouse Model of Amyotrophic Lateral Sclerosis (Q61799568) (← links)