Pages that link to "Q37349744"

The following pages link to Alterations in ionotropic glutamate receptor subunits during binge cocaine self-administration and withdrawal in rats. (Q37349744):

Displaying 49 items.

• Glutamatergic substrates of drug addiction and alcoholism (Q24648924) (← links)
• Inhibition of cyclooxygenase-2 by NS398 attenuates noise-induced hearing loss in mice. (Q30388736) (← links)
• Animal models of substance abuse and addiction: implications for science, animal welfare, and society (Q30433958) (← links)
• Stability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine. (Q30490470) (← links)
• Integrative proteomic analysis of the nucleus accumbens in rhesus monkeys following cocaine self-administration (Q30504753) (← links)
• Cytosolic proteomic alterations in the nucleus accumbens of cocaine overdose victims (Q33262178) (← links)
• Cocaine-induced neuroadaptations in glutamate transmission: potential therapeutic targets for craving and addiction (Q33626978) (← links)
• Upregulation of Arc mRNA expression in the prefrontal cortex following cue-induced reinstatement of extinguished cocaine-seeking behavior. (Q33703575) (← links)
• Assessment of genome and proteome profiles in cocaine abuse (Q33722792) (← links)
• AMPA receptor plasticity in the nucleus accumbens after repeated exposure to cocaine (Q34233602) (← links)
• Bidirectional effects of inhibiting or potentiating NMDA receptors on extinction after cocaine self-administration in rats (Q34517093) (← links)
• Continuous exposure to the competitive N-methyl-D: -aspartate receptor antagonist, LY235959, facilitates escalation of cocaine consumption in Sprague-Dawley rats (Q34602356) (← links)
• Differential glutamate AMPA-receptor plasticity in subpopulations of VTA neurons in the presence or absence of residual cocaine: Implications for the development of addiction (Q34860748) (← links)
• Suppression of activity-regulated cytoskeleton-associated gene expression in the dorsal striatum attenuates extinction of cocaine-seeking (Q35059900) (← links)
• Changes in levels of D1, D2, or NMDA receptors during withdrawal from brief or extended daily access to IV cocaine (Q35642558) (← links)
• Cocainomics: new insights into the molecular basis of cocaine addiction. (Q35663924) (← links)
• Ethanol self-administration modulation of NMDA receptor subunit and related synaptic protein mRNA expression in prefrontal cortical fields in cynomolgus monkeys (Q35738651) (← links)
• Fos and glutamate AMPA receptor subunit coexpression associated with cue-elicited cocaine-seeking behavior in abstinent rats. (Q35809536) (← links)
• Extinction training regulates neuroadaptive responses to withdrawal from chronic cocaine self-administration (Q35909931) (← links)
• Extinction-induced neuroplasticity attenuates stress-induced cocaine seeking: a state-dependent learning hypothesis (Q36069525) (← links)
• Dopamine and glutamate release in the dorsolateral caudate putamen following withdrawal from cocaine self-administration in rats (Q36388857) (← links)
• GDNF and addiction (Q36414656) (← links)
• Region-specific involvement of AMPA/Kainate receptors in Fos protein expression induced by cocaine-conditioned cues (Q36703310) (← links)
• Cocaine but not natural reward self-administration nor passive cocaine infusion produces persistent LTP in the VTA. (Q36994841) (← links)
• Phosphorylation-dependent trafficking of GluR2-containing AMPA receptors in the nucleus accumbens plays a critical role in the reinstatement of cocaine seeking (Q37012544) (← links)
• Deficits in ventromedial prefrontal cortex group 1 metabotropic glutamate receptor function mediate resistance to extinction during protracted withdrawal from an extensive history of cocaine self-administration (Q37013642) (← links)
• Contribution of ventral tegmental GABA receptors to cocaine self-administration in rats (Q37086778) (← links)
• Neuroplasticity in the mesolimbic dopamine system and cocaine addiction (Q37111631) (← links)
• Short and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex (Q37200993) (← links)
• Persistent proteomic alterations in the medial prefrontal cortex with abstinence from cocaine self-administration (Q37343024) (← links)
• Differential regulation of ionotropic glutamate receptor subunits following cocaine self-administration (Q37349710) (← links)
• Continuous exposure to dizocilpine facilitates escalation of cocaine consumption in male Sprague-Dawley rats (Q37397242) (← links)
• Disruption of glutamate receptor-interacting protein in nucleus accumbens enhances vulnerability to cocaine relapse. (Q37491638) (← links)
• Cocaine-induced metaplasticity in the nucleus accumbens: Silent synapse and beyond (Q37828239) (← links)
• Methylphenidate administration to adolescent rats determines plastic changes on reward-related behavior and striatal gene expression (Q38318237) (← links)
• Glutamatergic synaptic plasticity in the mesocorticolimbic system in addiction (Q38344826) (← links)
• Glutamatergic plasticity in medial prefrontal cortex and ventral tegmental area following extended-access cocaine self-administration (Q38974060) (← links)
• Immunolocalization of NMDA receptor subunit NR3B in selected structures in the rat forebrain, cerebellum, and lumbar spinal cord (Q39990014) (← links)
• Extended daily access to cocaine results in distinct alterations in Homer 1b/c and NMDA receptor subunit expression within the medial prefrontal cortex (Q42069029) (← links)
• Contributions of prolonged contingent and non-contingent cocaine exposure to escalation of cocaine intake and glutamatergic gene expression. (Q46191741) (← links)
• Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking (Q46579491) (← links)
• Behavioral sensitization to cocaine is associated with increased AMPA receptor surface expression in the nucleus accumbens. (Q46738269) (← links)
• Involvement of the AMPA receptor GluR-C subunit in alcohol-seeking behavior and relapse. (Q46911576) (← links)
• AMPA-receptor GluR1 subunits are involved in the control over behavior by cocaine-paired cues (Q46958768) (← links)
• Increased corticolimbic connectivity in cocaine dependence versus pathological gambling is associated with drug severity and emotion-related impulsivity (Q47724906) (← links)
• Accumbal neurons that are activated during cocaine self-administration are spared from inhibitory effects of repeated cocaine self-administration (Q48405101) (← links)
• Recent updates on drug abuse analyzed by neuroproteomics studies: Cocaine, Methamphetamine and MDMA (Q58114574) (← links)
• Neurotoxic Agent-Induced Injury in Neurodegenerative Disease Model: Focus on Involvement of Glutamate Receptors (Q58694067) (← links)
• Prefrontal-accumbens opioid plasticity: Implications for relapse and dependence (Q93349778) (← links)