Pages that link to "Q36908536"
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The following pages link to Functional expression of Kir4.1 channels in spinal cord astrocytes (Q36908536):
Displaying 50 items.
- Inwardly rectifying potassium channels: their structure, function, and physiological roles (Q24296441) (← links)
- Gap junctions couple astrocytes and oligodendrocytes (Q24645936) (← links)
- New Insights on Astrocyte Ion Channels: Critical for Homeostasis and Neuron-Glia Signaling (Q26782952) (← links)
- Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy. (Q27340721) (← links)
- Downregulation of Kir4.1 inward rectifying potassium channel subunits by RNAi impairs potassium transfer and glutamate uptake by cultured cortical astrocytes (Q28569155) (← links)
- Analysis of astroglial K+ channel expression in the developing hippocampus reveals a predominant role of the Kir4.1 subunit (Q28591676) (← links)
- Development and validation of fluorescence-based and automated patch clamp-based functional assays for the inward rectifier potassium channel Kir4.1. (Q30423818) (← links)
- Microglial cells in astroglial cultures: a cautionary note (Q30480840) (← links)
- Kir4.1 channels regulate swelling of astroglial processes in experimental spinal cord edema (Q33303469) (← links)
- Spinal cord injury causes a wide-spread, persistent loss of Kir4.1 and glutamate transporter 1: benefit of 17 beta-oestradiol treatment (Q33773510) (← links)
- VGluT1+ neuronal glutamatergic signaling regulates postnatal developmental maturation of cortical protoplasmic astroglia (Q34037564) (← links)
- Changes in the astrocytic aquaporin-4 and inwardly rectifying potassium channel expression in the brain of the amyotrophic lateral sclerosis SOD1(G93A) rat model (Q34417341) (← links)
- Functional changes in glutamate transporters and astrocyte biophysical properties in a rodent model of focal cortical dysplasia (Q34722256) (← links)
- Molecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels (Q34800154) (← links)
- Regulation of neurovascular coupling in autoimmunity to water and ion channels (Q35001946) (← links)
- Reactive astrogliosis causes the development of spontaneous seizures (Q35117565) (← links)
- Postnatal development of the molecular complex underlying astrocyte polarization (Q35783432) (← links)
- Voltage-dependent K+ currents contribute to heterogeneity of olfactory ensheathing cells (Q35862566) (← links)
- Elevated GFAP induces astrocyte dysfunction in caudal brain regions: A potential mechanism for hindbrain involved symptoms in type II Alexander disease (Q36014767) (← links)
- Differential distribution of Kir4.1 in spinal cord astrocytes suggests regional differences in K+ homeostasis (Q36089472) (← links)
- Human glioma cells induce hyperexcitability in cortical networks (Q36162265) (← links)
- Spatial and temporal correlation in progressive degeneration of neurons and astrocytes in contusion-induced spinal cord injury (Q36162573) (← links)
- Correlating Gene-specific DNA Methylation Changes with Expression and Transcriptional Activity of Astrocytic KCNJ10 (Kir4.1) (Q36408222) (← links)
- Novel KCNJ10 Gene Variations Compromise Function of Inwardly Rectifying Potassium Channel 4.1. (Q36755104) (← links)
- Functional implications for Kir4.1 channels in glial biology: from K+ buffering to cell differentiation (Q36968600) (← links)
- A three ion channel genes-based signature predicts prognosis of primary glioblastoma patients and reveals a chemotherapy sensitive subtype (Q37688866) (← links)
- DNA methylation functions as a critical regulator of Kir4.1 expression during CNS development (Q37709950) (← links)
- Functions of mature mammalian astrocytes: a current view (Q37710857) (← links)
- How do astrocytes shape synaptic transmission? Insights from electrophysiology (Q38150327) (← links)
- Astrocyte Dysfunctions and HIV-1 Neurotoxicity (Q38192643) (← links)
- The role of glial-specific Kir4.1 in normal and pathological states of the CNS. (Q38767547) (← links)
- Glia: A Neglected Player in Non-invasive Direct Current Brain Stimulation (Q38828798) (← links)
- Glutamate Neurotransmission in Rodent Models of Traumatic Brain Injury. (Q38852061) (← links)
- Astrogliosis is a possible player in preventing delayed neuronal death (Q39304000) (← links)
- Kir4.1 K+channels are regulated by external cations (Q39430985) (← links)
- Purinergic P2Y1 Receptors Control Rapid Expression of Plasma Membrane Processes in Hippocampal Astrocytes. (Q39672793) (← links)
- Role of Kir4.1 channels in growth control of glia (Q40079176) (← links)
- Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis (Q40240892) (← links)
- Absence of Delayed Neuronal Death in ATP-Injected Brain: Possible Roles of Astrogliosis (Q41882894) (← links)
- Neuron-glia cross talk revealed in reverberating networks by simultaneous extracellular recording of spikes and astrocytes' glutamate transporter and K+ currents. (Q42360046) (← links)
- Implication of Kir4.1 channel in excess potassium clearance: an in vivo study on anesthetized glial-conditional Kir4.1 knock-out mice (Q43796986) (← links)
- Stratification of astrocytes in healthy and diseased brain (Q46321594) (← links)
- Osmotic swelling characteristics of glial cells in the murine hippocampus, cerebellum, and retina in situ (Q46790384) (← links)
- Oxygen and Glucose Deprivation Induces Bergmann Glia Membrane Depolarization and Ca2+ Rises Mainly Mediated by K+ and ATP Increases in the Extracellular Space (Q47156520) (← links)
- Inwardly Rectifying K+ Currents in Cultured Oligodendrocytes from Rat Optic Nerve are Insensitive to pH. (Q48360658) (← links)
- Relationship between glial potassium regulation and axon excitability: a role for glial Kir4.1 channels (Q48683655) (← links)
- MeCP2 Deficiency Leads to Loss of Glial Kir4.1. (Q49724123) (← links)
- Physiology of Astroglia (Q49917383) (← links)
- Astrocytes, Microglia, and Parkinson's Disease. (Q55000928) (← links)
- Kir4.1-Dependent Astrocyte-Fast Motor Neuron Interactions Are Required for Peak Strength. (Q55083482) (← links)