Pages that link to "Q36836273"

The following pages link to Accumulation of mutants in "aging" bacterial colonies is due to growth under selection, not stress-induced mutagenesis (Q36836273):

Displaying 44 items.

• Experimental evolution in biofilm populations. (Q26768691) (← links)
• Culture history and population heterogeneity as determinants of bacterial adaptation: the adaptomics of a single environmental transition (Q26825267) (← links)
• Molecular and cellular bases of adaptation to a changing environment in microorganisms (Q28079255) (← links)
• Costs of antibiotic resistance - separating trait effects and selective effects (Q28082095) (← links)
• Spontaneous Emergence of Multiple Drug Resistance in Tuberculosis before and during Therapy (Q28477566) (← links)
• Interplay between pleiotropy and secondary selection determines rise and fall of mutators in stress response (Q33543179) (← links)
• Frequent beneficial mutations during single-colony serial transfer of Streptococcus pneumoniae. (Q34005325) (← links)
• Effect of Growth Under Selection on Appearance of Chromosomal Mutations inSalmonella enterica (Q34195488) (← links)
• The evolution of antibiotic susceptibility and resistance during the formation of Escherichia coli biofilms in the absence of antibiotics (Q34565434) (← links)
• Selection-driven accumulation of suppressor mutants in bacillus subtilis: the apparent high mutation frequency of the cryptic gudB gene and the rapid clonal expansion of gudB(+) suppressors are due to growth under selection (Q34778850) (← links)
• Acetate availability and utilization supports the growth of mutant sub-populations on aging bacterial colonies (Q35295130) (← links)
• Phenotypic Diversity Caused by Differential RpoS Activity among Environmental Escherichia coli Isolates (Q35530168) (← links)
• Alarmingly High Segregation Frequencies of Quinolone Resistance Alleles within Human and Animal Microbiomes Are Not Explained by Direct Clinical Antibiotic Exposure (Q35644230) (← links)
• Reversibility of replicative senescence in Saccharomyces cerevisiae: Effect of homologous recombination and cell cycle checkpoints (Q35661345) (← links)
• Rifampin Resistance rpoB Alleles or Multicopy Thioredoxin/Thioredoxin Reductase Suppresses the Lethality of Disruption of the Global Stress Regulator spx in Staphylococcus aureus (Q36081351) (← links)
• Fitness Cost of Rifampin Resistance in Neisseria meningitidis: In Vitro Study of Mechanisms Associated with rpoB H553Y Mutation. (Q36290567) (← links)
• A shifting mutational landscape in 6 nutritional states: Stress-induced mutagenesis as a series of distinct stress input-mutation output relationships. (Q36395746) (← links)
• Competitive fitness during feast and famine: how SOS DNA polymerases influence physiology and evolution in Escherichia coli (Q36878608) (← links)
• A global view of antibiotic resistance (Q37340607) (← links)
• The impact of drug resistance on Mycobacterium tuberculosis physiology: what can we learn from rifampicin? (Q37683485) (← links)
• Antibiotic resistance and its cost: is it possible to reverse resistance? (Q37704177) (← links)
• Cellular mechanisms that control mistranslation (Q37809740) (← links)
• Hypermutation and stress adaptation in bacteria (Q37922538) (← links)
• Isolation and characterization of transcription fidelity mutants (Q37988262) (← links)
• Mutation--The Engine of Evolution: Studying Mutation and Its Role in the Evolution of Bacteria (Q38578737) (← links)
• Rifampicin Resistance: Fitness Costs and the Significance of Compensatory Evolution (Q38793891) (← links)
• Potential for adaptation overrides cost of resistance. (Q38966881) (← links)
• Spontaneous mutation rate is a plastic trait associated with population density across domains of life (Q40072185) (← links)
• Stress-Induced Mutagenesis. (Q40459862) (← links)
• Natural selection underlies apparent stress-induced mutagenesis in a bacteriophage infection model. (Q40559189) (← links)
• Mutational Consequences of Ciprofloxacin in Escherichia coli (Q41194789) (← links)
• Testing the role of genetic background in parallel evolution using the comparative experimental evolution of antibiotic resistance. (Q41846993) (← links)
• Elevated mutagenesis does not explain the increased frequency of antibiotic resistant mutants in starved aging colonies (Q41855236) (← links)
• Rapid Genetic Adaptation during the First Four Months of Survival under Resource Exhaustion (Q42227289) (← links)
• Effect of translesion DNA polymerases, endonucleases and RpoS on mutation rates in Salmonella typhimurium (Q42719517) (← links)
• Genetic characterization of compensatory evolution in strains carrying rpoB Ser531Leu, the rifampicin resistance mutation most frequently found in clinical isolates. (Q50014863) (← links)
• Fitness-compensatory mutations in rifampicin-resistant RNA polymerase (Q50026431) (← links)
• Rapid evolution of acetic acid-detoxifying Escherichia coli under phosphate starvation conditions requires activation of the cryptic PhnE permease and induction of translesion synthesis DNA polymerases (Q51136979) (← links)
• The onion model, a simple neutral model for the evolution of diversity in bacterial biofilms (Q54352667) (← links)
• Death and population dynamics affect mutation rate estimates and evolvability under stress in bacteria. (Q55162233) (← links)
• Rapid cell division of Staphylococcus aureus during colonization of the human nose (Q64068826) (← links)
• Fitness of Spontaneous Rifampicin-Resistant Isolates in a Biofilm Environment (Q64248518) (← links)
• Evaluating evolutionary models of stress-induced mutagenesis in bacteria (Q86095467) (← links)
• Aging of a Bacterial Colony Enforces the Evolvement of Nondifferentiating Mutants (Q93084901) (← links)