Pages that link to "Q36702652"

The following pages link to Neurotransmitter release evoked by nerve impulses without Ca2+ entry through Ca2+ channels in frog motor nerve endings (Q36702652):

Displaying 16 items.

• Inositol derivatives modulate spontaneous transmitter release at the frog neuromuscular junction (Q28203745) (← links)
• Quantal ATP release from motor nerve endings and its role in neurally mediated depression. (Q33768397) (← links)
• Autoreceptors, membrane potential and the regulation of transmitter release (Q33826757) (← links)
• Can the Ca2+ hypothesis and the Ca2+-voltage hypothesis for neurotransmitter release be reconciled? (Q34431126) (← links)
• Calmodulin increases transmitter release by mobilizing quanta at the frog motor nerve terminal (Q35044702) (← links)
• On the feedback between theory and experiment in elucidating the molecular mechanisms underlying neurotransmitter release (Q36533029) (← links)
• Control of neurotransmitter release: From Ca2+ to voltage dependent G-protein coupled receptors (Q37784724) (← links)
• Depolarization-induced calcium-independent synaptic vesicle exo- and endocytosis at frog motor nerve terminals (Q41870211) (← links)
• Modulation of spontaneous transmitter release from the frog neuromuscular junction by interacting intracellular Ca(2+) stores: critical role for nicotinic acid-adenine dinucleotide phosphate (NAADP). (Q43002997) (← links)
• Evidence for two distinct processes in the final stages of neurotransmitter release as detected by binomial analysis in calcium and strontium solutions (Q43920095) (← links)
• Bromoacetylcholine and acetylcholinesterase introduced via liposomes into motor nerve endings block increases in quantal size (Q47329111) (← links)
• Parallel computation enables precise description of Ca2+ distribution in nerve terminals (Q51649172) (← links)
• Release of neurotransmitter induced by Ca2+-uncaging: reexamination of the ca-voltage hypothesis for release. (Q51965232) (← links)
• Tetramerisation of α-latrotoxin by divalent cations is responsible for toxin-induced non-vesicular release and contributes to the Ca2+-dependent vesicular exocytosis from synaptosomes (Q61685349) (← links)
• Inositol trisphosphate and cyclic adenosine diphosphate-ribose increase quantal transmitter release at frog motor nerve terminals: possible involvement of smooth endoplasmic reticulum (Q73465228) (← links)
• A simulation study on the Ca2+-independent but voltage-dependent exocytosis and endocytosis in dorsal root ganglion neurons (Q81833889) (← links)