Pages that link to "Q36514267"
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The following pages link to Muscle damage in mdx (dystrophic) mice: role of calcium and reactive oxygen species. (Q36514267):
Displaying 50 items.
- Oxidative Stress-Mediated Skeletal Muscle Degeneration: Molecules, Mechanisms, and Therapies (Q26769962) (← links)
- Rescue of dystrophic skeletal muscle by PGC-1α involves a fast to slow fiber type shift in the mdx mouse (Q27310125) (← links)
- Characterization of dystrophin deficient rats: a new model for Duchenne muscular dystrophy (Q27335054) (← links)
- The role of oxidative stress in skeletal muscle injury and regeneration: focus on antioxidant enzymes (Q28076877) (← links)
- Muscle degeneration and leukocyte infiltration caused by mutation of zebrafish Fad24 (Q30493839) (← links)
- Microtubules underlie dysfunction in duchenne muscular dystrophy. (Q30557507) (← links)
- Microarchitecture is severely compromised but motor protein function is preserved in dystrophic mdx skeletal muscle (Q33647871) (← links)
- Eccentric exercise in aging and diseased skeletal muscle: good or bad? (Q33708184) (← links)
- Therapeutic potential of proteasome inhibition in Duchenne and Becker muscular dystrophies (Q33745227) (← links)
- Enhanced Ca²⁺ influx from STIM1-Orai1 induces muscle pathology in mouse models of muscular dystrophy. (Q33784460) (← links)
- Green tea extract decreases muscle pathology and NF-kappaB immunostaining in regenerating muscle fibers of mdx mice (Q33901041) (← links)
- The different impact of a high fat diet on dystrophic mdx and control C57Bl/10 mice (Q34077151) (← links)
- Increased Store-Operated Ca2+ Entry in Skeletal Muscle with Reduced Calsequestrin-1 Expression (Q34098989) (← links)
- P38α MAPK underlies muscular dystrophy and myofiber death through a Bax-dependent mechanism (Q34285775) (← links)
- Bowman-Birk inhibitor attenuates dystrophic pathology in mdx mice. (Q34305281) (← links)
- Debio-025 is more effective than prednisone in reducing muscular pathology in mdx mice (Q34307101) (← links)
- Long-term blocking of calcium channels in mdx mice results in differential effects on heart and skeletal muscle (Q34469185) (← links)
- Orai1 mediates exacerbated Ca(2+) entry in dystrophic skeletal muscle (Q34490590) (← links)
- Mitigation of muscular dystrophy in mice by SERCA overexpression in skeletal muscle (Q34627089) (← links)
- Bmi1 enhances skeletal muscle regeneration through MT1-mediated oxidative stress protection in a mouse model of dystrophinopathy (Q34708380) (← links)
- Disruption of calcium homeostasis in cardiomyocytes underlies cardiac structural and functional changes in severe sepsis. (Q34918581) (← links)
- Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy (Q35054988) (← links)
- Dystropathology increases energy expenditure and protein turnover in the mdx mouse model of duchenne muscular dystrophy (Q35106988) (← links)
- Transgenic overexpression of γ-cytoplasmic actin protects against eccentric contraction-induced force loss in mdx mice (Q35545904) (← links)
- SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse models (Q35572589) (← links)
- Characterization of a novel chicken muscle disorder through differential gene expression and pathway analysis using RNA-sequencing (Q35637260) (← links)
- Chronic administration of a leupeptin-derived calpain inhibitor fails to ameliorate severe muscle pathology in a canine model of duchenne muscular dystrophy (Q35660362) (← links)
- Low-Level Laser Therapy (LLLT) in Dystrophin-Deficient Muscle Cells: Effects on Regeneration Capacity, Inflammation Response and Oxidative Stress (Q35666182) (← links)
- The Effects of Experimental Sleep Apnea on Cardiac and Respiratory Functions in 6 and 18 Month Old Dystrophic (mdx) Mice (Q35903669) (← links)
- Quantitative evaluation of the beneficial effects in the mdx mouse of epigallocatechin gallate, an antioxidant polyphenol from green tea. (Q35962987) (← links)
- Increased resting intracellular calcium modulates NF-κB-dependent inducible nitric-oxide synthase gene expression in dystrophic mdx skeletal myotubes (Q36033118) (← links)
- Abnormalities in brain structure and biochemistry associated with mdx mice measured by in vivo MRI and high resolution localized (1)H MRS. (Q36075156) (← links)
- Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy. (Q36422465) (← links)
- Induction of a regenerative microenvironment in skeletal muscle is sufficient to induce embryonal rhabdomyosarcoma in p53-deficient mice (Q36503375) (← links)
- Cardiac phenotype of Duchenne Muscular Dystrophy: insights from cellular studies (Q36736081) (← links)
- Identification of disease specific pathways using in vivo SILAC proteomics in dystrophin deficient mdx mouse. (Q36832437) (← links)
- Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy (Q36841572) (← links)
- Endurance capacity in maturing mdx mice is markedly enhanced by combined voluntary wheel running and green tea extract (Q36877501) (← links)
- Systemic elevation of interleukin-15 in vivo promotes apoptosis in skeletal muscles of young adult and aged rats (Q36942445) (← links)
- Myostatin genetic inactivation inhibits myogenesis by muscle-derived stem cells in vitro but not when implanted in the mdx mouse muscle. (Q36998190) (← links)
- Restricting calcium currents is required for correct fiber type specification in skeletal muscle. (Q37009767) (← links)
- Insights into bone health in Duchenne muscular dystrophy (Q37056368) (← links)
- Implications of cross-talk between tumour necrosis factor and insulin-like growth factor-1 signalling in skeletal muscle. (Q37063632) (← links)
- Genetic and pharmacologic inhibition of mitochondrial-dependent necrosis attenuates muscular dystrophy (Q37127322) (← links)
- Malformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling (Q37338902) (← links)
- Role of reactive oxygen species in the defective regeneration seen in aging muscle (Q37384695) (← links)
- Calcium influx is sufficient to induce muscular dystrophy through a TRPC-dependent mechanism (Q37419160) (← links)
- Mechanisms of excitation-contraction uncoupling relevant to activity-induced muscle fatigue (Q37486012) (← links)
- Finding the missing link: disulfide-containing proteins via a high-throughput proteomics approach. (Q37582155) (← links)
- Immune-mediated mechanisms potentially regulate the disease time-course of duchenne muscular dystrophy and provide targets for therapeutic intervention (Q37583167) (← links)