Pages that link to "Q36554262"
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The following pages link to Alterations in p53 and p16INK4 expression and telomere length during spontaneous immortalization of Li-Fraumeni syndrome fibroblasts (Q36554262):
Displaying 50 items.
- Nek4 regulates entry into replicative senescence and the response to DNA damage in human fibroblasts (Q24294537) (← links)
- Suppression of alternative lengthening of telomeres by Sp100-mediated sequestration of the MRE11/RAD50/NBS1 complex. (Q24298041) (← links)
- Superoxide dismutase 1 knock-down induces senescence in human fibroblasts (Q24310059) (← links)
- The SMC5/6 complex maintains telomere length in ALT cancer cells through SUMOylation of telomere-binding proteins (Q24313641) (← links)
- Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a (Q24324559) (← links)
- Regulation of p16CDKN2 expression and its implications for cell immortalization and senescence (Q24650807) (← links)
- Cellular Senescence:Ex Vivop53-Dependent Asymmetric Cell Kinetics (Q24803348) (← links)
- Molecular interactions between telomerase and the tumor suppressor protein p53 in vitro (Q28118564) (← links)
- Inactivation of p53 and life span extension of human diploid fibroblasts by mot-2 (Q28145704) (← links)
- The Y-box-binding protein, YB1, is a potential negative regulator of the p53 tumor suppressor (Q28564441) (← links)
- Y-box factor YB1 controls p53 apoptotic function (Q28578217) (← links)
- Tumor suppression at the mouse INK4a locus mediated by the alternative reading frame product p19ARF (Q29619663) (← links)
- The proline-rich region of mouse p53 influences transactivation and apoptosis but is largely dispensable for these functions. (Q30333229) (← links)
- A reassessment of the telomere hypothesis of senescence (Q33536846) (← links)
- Telomere maintenance mechanisms and cellular immortalization. (Q33540084) (← links)
- Cell cycle control, checkpoint mechanisms, and genotoxic stress (Q33608768) (← links)
- Alternative pathways for the extension of cellular life span: inactivation of p53/pRb and expression of telomerase (Q33807819) (← links)
- HP1-mediated formation of alternative lengthening of telomeres-associated PML bodies requires HIRA but not ASF1a (Q33828273) (← links)
- A new functional classification of tumor-suppressing genes and its therapeutic implications (Q33843457) (← links)
- The role of senescence and immortalization in carcinogenesis (Q33846221) (← links)
- Telomere Biology-Insights into an Intriguing Phenomenon. (Q33855946) (← links)
- Physical and functional interaction between p53 and the Werner's syndrome protein (Q33875710) (← links)
- The alternative product from the human CDKN2A locus, p14(ARF), participates in a regulatory feedback loop with p53 and MDM2. (Q33889383) (← links)
- Differential roles for cyclin-dependent kinase inhibitors p21 and p16 in the mechanisms of senescence and differentiation in human fibroblasts (Q33957508) (← links)
- Analysis of genomic integrity and p53-dependent G1 checkpoint in telomerase-induced extended-life-span human fibroblasts (Q33957590) (← links)
- Induction of p21(WAF1/CIP1) and inhibition of Cdk2 mediated by the tumor suppressor p16(INK4a) (Q33958412) (← links)
- Human keratinocytes that express hTERT and also bypass a p16(INK4a)-enforced mechanism that limits life span become immortal yet retain normal growth and differentiation characteristics (Q33962085) (← links)
- Uncoupling between phenotypic senescence and cell cycle arrest in aging p21-deficient fibroblasts (Q33965486) (← links)
- DNA strand break-sensing molecule poly(ADP-Ribose) polymerase cooperates with p53 in telomere function, chromosome stability, and tumor suppression (Q33968476) (← links)
- A mutation in the STN1 gene triggers an alternative lengthening of telomere-like runaway recombinational telomere elongation and rapid deletion in yeast (Q34042826) (← links)
- Telomeres and their control (Q34090772) (← links)
- Alternative lengthening of telomeres: models, mechanisms and implications (Q34107386) (← links)
- Alternative lengthening of telomeres in mammalian cells (Q34114656) (← links)
- Homologous recombination as a mechanism of carcinogenesis (Q34183753) (← links)
- Loss of retinoblastoma but not p16 function allows bypass of replicative senescence in human fibroblasts (Q34250440) (← links)
- Homologous recombination in human telomerase‐positive and ALT cells occurs with the same frequency (Q34250567) (← links)
- Telomere maintenance mechanisms in cancer: clinical implications (Q34426966) (← links)
- Li-Fraumeni syndrome – a molecular and clinical review (Q34432117) (← links)
- Long telomeres produced by telomerase-resistant recombination are established from a single source and are subject to extreme sequence scrambling (Q34469050) (← links)
- Human Rad51 protein displays enhanced homologous pairing of DNA sequences resembling those at genetically unstable loci (Q34658178) (← links)
- Disruption of telomere maintenance by depletion of the MRE11/RAD50/NBS1 complex in cells that use alternative lengthening of telomeres (Q34662153) (← links)
- Recombination Can Cause Telomere Elongations as Well as Truncations Deep within Telomeres in Wild-Type Kluyveromyces lactis Cells (Q34739230) (← links)
- Replicative senescence in organ transplantation-mechanisms and significance (Q34786096) (← links)
- CPEB regulation of human cellular senescence, energy metabolism, and p53 mRNA translation (Q34920958) (← links)
- Yeast telomeres exert a position effect on recombination between internal tracts of yeast telomeric DNA (Q35210267) (← links)
- From cells to ageing: a review of models and mechanisms of cellular senescence and their impact on human ageing. (Q35895097) (← links)
- Cyclin D1 overexpression and p53 inactivation immortalize primary oral keratinocytes by a telomerase-independent mechanism (Q36167748) (← links)
- Relationship between the expression of human telomerase reverse transcriptase gene and cell cycle regulators in gastric cancer and its significance (Q36209684) (← links)
- Maintenance of very long telomeres by recombination in the Kluyveromyces lactis stn1-M1 mutant involves extreme telomeric turnover, telomeric circles, and concerted telomeric amplification (Q36210968) (← links)
- p53 in recombination and repair (Q36423948) (← links)