Pages that link to "Q36319909"

The following pages link to Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction. (Q36319909):

Displaying 39 items.

• The circular relationship between matrix metalloproteinase-9 and inflammation following myocardial infarction (Q26798328) (← links)
• Translating Koch's postulates to identify matrix metalloproteinase roles in postmyocardial infarction remodeling: cardiac metalloproteinase actions (CarMA) postulates (Q26865099) (← links)
• Stem cell mechanisms during left ventricular remodeling post-myocardial infarction: Repair and regeneration (Q27026285) (← links)
• Myofibroblasts and the extracellular matrix network in post-myocardial infarction cardiac remodeling (Q33663388) (← links)
• Integrative computational and experimental approaches to establish a post-myocardial infarction knowledge map. (Q35126269) (← links)
• Resolvin D1 activates the inflammation resolving response at splenic and ventricular site following myocardial infarction leading to improved ventricular function. (Q35744344) (← links)
• Secreted protein acidic and rich in cysteine facilitates age-related cardiac inflammation and macrophage M1 polarization. (Q35749231) (← links)
• Deriving a cardiac ageing signature to reveal MMP-9-dependent inflammatory signalling in senescence. (Q35837349) (← links)
• Differential Expression of Matrix Metalloproteinases 2, 9 and Cytokines by Neutrophils and Monocytes in the Clinical Forms of Chagas Disease. (Q36258684) (← links)
• Systemic inflammation on postnatal days 21 and 28 and indicators of brain dysfunction 2years later among children born before the 28th week of gestation (Q36522018) (← links)
• Tissue Inhibitor of Metalloproteinase-1: Actions beyond Matrix Metalloproteinase Inhibition. (Q36526033) (← links)
• Matrix metalloproteinases as input and output signals for post-myocardial infarction remodeling (Q36609629) (← links)
• Using the laws of thermodynamics to understand how matrix metalloproteinases coordinate the myocardial response to injury (Q37058562) (← links)
• MMP-9 signaling in the left ventricle following myocardial infarction (Q37139613) (← links)
• Matrix metalloproteinase-9: Many shades of function in cardiovascular disease. (Q37380847) (← links)
• Neutrophil roles in left ventricular remodeling following myocardial infarction. (Q38111517) (← links)
• Trans-system mechanisms against ischemic myocardial injury (Q38318626) (← links)
• Cellular immunity and cardiac remodeling after myocardial infarction: role of neutrophils, monocytes, and macrophages (Q38365651) (← links)
• Monocyte and macrophage subsets along the continuum to heart failure: Misguided heroes or targetable villains? (Q38620886) (← links)
• Matrix Metalloproteinases in Myocardial Infarction and Heart Failure. (Q38729906) (← links)
• Biomarkers to Predict Reverse Remodeling and Myocardial Recovery in Heart Failure (Q38977493) (← links)
• The Role of Inflammation in Cardiovascular Outcome. (Q39134471) (← links)
• Using plasma matrix metalloproteinase-9 and monocyte chemoattractant protein-1 to predict future cardiovascular events in subjects with carotid atherosclerosis (Q39277249) (← links)
• Vinexin-β exacerbates cardiac dysfunction post-myocardial infarction via mediating apoptotic and inflammatory responses (Q41469937) (← links)
• Soluble guanylyl cyclase-activated cyclic GMP-dependent protein kinase inhibits arterial smooth muscle cell migration independent of VASP-serine 239 phosphorylation (Q41869044) (← links)
• IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation. (Q42776371) (← links)
• Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution (Q43136508) (← links)
• Aldose reductase modulates acute activation of mesenchymal markers via the β-catenin pathway during cardiac ischemia-reperfusion. (Q47346466) (← links)
• Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction (Q47610191) (← links)
• Cardiac macrophage biology in the steady-state heart, the aging heart, and following myocardial infarction (Q47718174) (← links)
• Macrophages in the infarct: fiery friends or friendly fire? (Q47773105) (← links)
• Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis. (Q47909549) (← links)
• Myocardial Infarction Remodeling that Progresses to Heart Failure: A Signaling Misunderstanding. (Q52619556) (← links)
• The Mouse Heart Attack Research Tool 1.0 database (Q56887927) (← links)
• Assigning matrix metalloproteinase roles in ischaemic cardiac remodelling (Q61420974) (← links)
• Increasing Cardiomyocyte Atrogin-1 Reduces Aging-Associated Fibrosis and Regulates Remodeling in Vivo (Q88664220) (← links)
• Delivery of a matrix metalloproteinase-responsive hydrogel releasing TIMP-3 after myocardial infarction: effects on left ventricular remodeling (Q89444886) (← links)
• Sodium tanshinone IIA sulfonate prevents the adverse left ventricular remodelling: Focus on polymorphonuclear neutrophil-derived granule components (Q91843486) (← links)
• Exogenous IL-4 shuts off pro-inflammation in neutrophils while stimulating anti-inflammation in macrophages to induce neutrophil phagocytosis following myocardial infarction (Q96648083) (← links)