Pages that link to "Q35896759"
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The following pages link to Partial inhibition of the ubiquitin-proteasome system ameliorates cardiac dysfunction following ischemia-reperfusion in the presence of high glucose (Q35896759):
Displaying 9 items.
- Activation of Chymotrypsin-Like Activity of the Proteasome during Ischemia Induces Myocardial Dysfunction and Death (Q36104970) (← links)
- Inhibition of dynamin-related protein 1 protects against myocardial ischemia-reperfusion injury in diabetic mice. (Q37631011) (← links)
- Increased 4-hydroxy-2-nonenal-induced proteasome dysfunction is correlated with cardiac damage in streptozotocin-injected rats with isoproterenol infusion (Q38765976) (← links)
- 3-Hydroxy-3-methylglutaric and 3-methylglutaric acids impair redox status and energy production and transfer in rat heart: relevance for the pathophysiology of cardiac dysfunction in 3-hydroxy-3-methylglutaryl-coenzyme A lyase deficiency. (Q39588663) (← links)
- Myocyte membrane and microdomain modifications in diabetes: determinants of ischemic tolerance and cardioprotection. (Q46034115) (← links)
- Interrelationship between diabetes mellitus and heart failure: the role of peroxisome proliferator-activated receptors in left ventricle performance. (Q49908591) (← links)
- Glycine Protects H9C2 Cardiomyocytes from High Glucose- and Hypoxia/Reoxygenation-Induced Injury via Inhibiting PKCβ2 Activation and Improving Mitochondrial Quality. (Q55018528) (← links)
- Resveratrol attenuates high glucose-induced cardiomyocytes injury via interfering ROS-MAPK-NF-κB signaling pathway (Q92644774) (← links)
- Ubiquitin‑proteasomes are the dominant mediators of the regulatory effect of microRNA‑1 on cardiac remodeling after myocardial infarction (Q93111194) (← links)