Pages that link to "Q34808927"

The following pages link to Role of small GTPases and alphavbeta5 integrin in Pseudomonas aeruginosa-induced increase in lung endothelial permeability (Q34808927):

Displaying 20 items.

• In the absence of effector proteins, the Pseudomonas aeruginosa type three secretion system needle tip complex contributes to lung injury and systemic inflammatory responses (Q27300857) (← links)
• Cytoprotective-Selective Activated Protein C Attenuates Pseudomonas aeruginosa–Induced Lung Injury in Mice (Q35219875) (← links)
• Pharmacological activation of Rap1 antagonizes the endothelial barrier disruption induced by exotoxins ExoS and ExoT of Pseudomonas aeruginosa. (Q35439607) (← links)
• The Role of ExoS in Dissemination of Pseudomonas aeruginosa during Pneumonia (Q35668315) (← links)
• SH2 domain-containing protein tyrosine phosphatase 2 and focal adhesion kinase protein interactions regulate pulmonary endothelium barrier function (Q35816393) (← links)
• Neuronal Wiskott-Aldrich syndrome protein regulates TGF-β1-mediated lung vascular permeability (Q36996355) (← links)
• Interleukin-8 regulates endothelial permeability by down-regulation of tight junction but not dependent on integrins induced focal adhesions (Q37249263) (← links)
• The type III secretion system of Pseudomonas aeruginosa: infection by injection (Q37397923) (← links)
• Heat shock protein 90 inhibitors prevent LPS-induced endothelial barrier dysfunction by disrupting RhoA signaling (Q37596088) (← links)
• Identification of miRNA biomarkers of pneumonia using RNA-sequencing and bioinformatics analysis (Q37734509) (← links)
• Mechanisms of phagocytosis and host clearance of Pseudomonas aeruginosa. (Q38182026) (← links)
• Bacteria and endothelial cells: a toxic relationship (Q39053084) (← links)
• Cellular responses of A549 alveolar epithelial cells to serially collected Pseudomonas aeruginosa from cystic fibrosis patients at different stages of pulmonary infection (Q39696154) (← links)
• T3SS effector ExoY reduces inflammasome-related responses by suppressing bacterial motility and delaying activation of NF-κB and caspase-1. (Q40090334) (← links)
• Actin activates Pseudomonas aeruginosa ExoY nucleotidyl cyclase toxin and ExoY-like effector domains from MARTX toxins. (Q40423732) (← links)
• PAI-1 is an essential component of the pulmonary host response during Pseudomonas aeruginosa pneumonia in mice (Q41261629) (← links)
• Postexposure aerosolized heparin reduces lung injury in chlorine-exposed mice. (Q42860753) (← links)
• Sequential inactivation of Rho GTPases and Lim kinase by Pseudomonas aeruginosa toxins ExoS and ExoT leads to endothelial monolayer breakdown (Q47708196) (← links)
• Pseudomonas aeruginosa Takes a Multi-Target Approach to Achieve Junction Breach. (Q49406055) (← links)
• The extreme C-terminus of the effector ExoY is crucial for binding to its eukaryotic activator, F-actin (Q58095936) (← links)