Pages that link to "Q34668446"

The following pages link to Stable cell lines expressing high levels of the herpes simplex virus type 1 LAT are refractory to caspase 3 activation and DNA laddering following cold shock induced apoptosis (Q34668446):

Displaying 31 items.

• Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting Apoptosis (Q26864175) (← links)
• The molecular basis of herpes simplex virus latency (Q27001272) (← links)
• Herpes simplex virus type 1 (HSV-1)-induced apoptosis in human dendritic cells as a result of downregulation of cellular FLICE-inhibitory protein and reduced expression of HSV-1 antiapoptotic latency-associated transcript sequences (Q33558390) (← links)
• Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycle (Q33654414) (← links)
• The suppression of apoptosis by α-herpesvirus (Q33815866) (← links)
• Small non-coding RNAs encoded within the herpes simplex virus type 1 latency associated transcript (LAT) cooperate with the retinoic acid inducible gene I (RIG-I) to induce beta-interferon promoter activity and promote cell survival (Q33820678) (← links)
• Regulation of herpes simplex virus type 1 thymidine kinase gene expression by thyroid hormone receptor in cultured neuronal cells (Q33883878) (← links)
• Identification of a novel herpes simplex virus type 1 transcript and protein (AL3) expressed during latency. (Q33922876) (← links)
• Cellular FLIP can substitute for the herpes simplex virus type 1 latency-associated transcript gene to support a wild-type virus reactivation phenotype in mice (Q34307385) (← links)
• Update on herpes virus infections of the nervous system (Q34379006) (← links)
• HSV-1 genome subnuclear positioning and associations with host-cell PML-NBs and centromeres regulate LAT locus transcription during latency in neurons. (Q34388469) (← links)
• The Herpes Simplex Virus 1 Latency-Associated Transcript Promotes Functional Exhaustion of Virus-Specific CD8 + T Cells in Latently Infected Trigeminal Ganglia: a Novel Immune Evasion Mechanism (Q35193019) (← links)
• Localization of sequences in a protein (ORF2) encoded by the latency-related gene of bovine herpesvirus 1 that inhibits apoptosis and interferes with Notch1-mediated trans-activation of the bICP0 promoter (Q35531486) (← links)
• Two microRNAs encoded within the bovine herpesvirus 1 latency-related gene promote cell survival by interacting with RIG-I and stimulating NF-κB-dependent transcription and beta interferon signaling pathways (Q35689581) (← links)
• Immunological control of herpes simplex virus infections (Q37135532) (← links)
• Two small RNAs encoded within the first 1.5 kilobases of the herpes simplex virus type 1 latency-associated transcript can inhibit productive infection and cooperate to inhibit apoptosis (Q37333965) (← links)
• Impairment in reactivation of a latency associated transcript (LAT)-deficient HSV-2 is not solely dependent on the latent viral load or the number of CD8(+) T cells infiltrating the ganglia (Q37435593) (← links)
• The herpes simplex virus type 1 (HSV-1) latency-associated transcript (LAT) protects cells against cold-shock-induced apoptosis by maintaining phosphorylation of protein kinase B (AKT) (Q37699101) (← links)
• Interactions between herpesvirus entry mediator (TNFRSF14) and latency-associated transcript during herpes simplex virus 1 latency. (Q38310640) (← links)
• The Herpes Simplex Virus Type 1 Latency-Associated Transcript Can Protect Neuron-Derived C1300 and Neuro2A Cells from Granzyme B-Induced Apoptosis and CD8 T-Cell Killing (Q39617169) (← links)
• Identification of herpes simplex virus type 1 proteins encoded within the first 1.5 kb of the latency-associated transcript (Q39736685) (← links)
• Herpes simplex virus type 1 latency-associated transcript inhibits apoptosis and promotes neurite sprouting in neuroblastoma cells following serum starvation by maintaining protein kinase B (AKT) levels (Q39768142) (← links)
• Open reading frame 2, encoded by the latency-related gene of bovine herpesvirus 1, has antiapoptotic activity in transiently transfected neuroblastoma cells (Q39948831) (← links)
• Coregulatory interactions among CD8α dendritic cells, the latency-associated transcript, and programmed death 1 contribute to higher levels of herpes simplex virus 1 latency (Q40605924) (← links)
• Overexpression of thyroid hormone receptor β1 altered thyroid hormone-mediated regulation of herpes simplex virus-1 replication in differentiated cells (Q40806196) (← links)
• The role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1. (Q42143302) (← links)
• A speculated ribozyme site in the herpes simplex virus type 1 latency-associated transcript gene is not essential for a wild-type reactivation phenotype (Q42699446) (← links)
• Programmed cell death: the battlefield between the host and alpha-herpesviruses and a potential avenue for cancer treatment (Q57022352) (← links)
• Disturbed Yin-Yang balance: stress increases the susceptibility to primary and recurrent infections of herpes simplex virus type 1 (Q90094364) (← links)
• The Latency-Associated Transcript Inhibits Apoptosis via Downregulation of Components of the Type I Interferon Pathway during Latent Herpes Simplex Virus 1 Ocular Infection (Q91983853) (← links)
• Herpes Simplex Virus: The Hostile Guest That Takes Over Your Home (Q95840901) (← links)