Pages that link to "Q34529852"
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The following pages link to Mutations associated with amyotrophic lateral sclerosis convert superoxide dismutase from an antiapoptotic gene to a proapoptotic gene: studies in yeast and neural cells (Q34529852):
Displaying 50 items.
- Analysis of aldehyde oxidase and xanthine dehydrogenase/oxidase as possible candidate genes for autosomal recessive familial amyotrophic lateral sclerosis (Q24312170) (← links)
- HIP1, a human homologue of S. cerevisiae Sla2p, interacts with membrane-associated huntingtin in the brain (Q24318878) (← links)
- Misfolded CuZnSOD and amyotrophic lateral sclerosis (Q24683677) (← links)
- The responses of Ht22 cells to oxidative stress induced by buthionine sulfoximine (BSO) (Q24793293) (← links)
- Structure and dynamics of copper-free SOD: The protein before binding copper (Q27639669) (← links)
- The structure of holo and metal-deficient wild-type human Cu, Zn superoxide dismutase and its relevance to familial amyotrophic lateral sclerosis (Q27641126) (← links)
- The perplexing role of copper-zinc superoxide dismutase in amyotrophic lateral sclerosis (Lou Gehrig's disease) (Q28184546) (← links)
- Increased apoptosis and early embryonic lethality in mice nullizygous for the Huntington's disease gene homologue (Q28506834) (← links)
- The copper transport protein Atox1 promotes neuronal survival (Q28569901) (← links)
- Corticotropin-releasing factor (CRF) and related peptides confer neuroprotection via type 1 CRF receptors (Q28575168) (← links)
- Bax and Bcl-2 interaction in a transgenic mouse model of familial amyotrophic lateral sclerosis (Q28585525) (← links)
- Mutations in copper-zinc superoxide dismutase that cause amyotrophic lateral sclerosis alter the zinc binding site and the redox behavior of the protein (Q30425734) (← links)
- Cystamine inhibits transglutaminase and caspase-3 cleavage in glutamate-exposed astroglial cells (Q31160300) (← links)
- Transforming growth factor-beta inhibits apoptosis induced by beta-amyloid peptide fragment 25-35 in cultured neuronal cells (Q31976274) (← links)
- IAP family proteins delay motoneuron cell death in vivo (Q33180652) (← links)
- Mitochondrial proteomic analysis of a cell line model of familial amyotrophic lateral sclerosis (Q33208192) (← links)
- Apoptosis in neurodegenerative diseases: the role of mitochondria (Q33540922) (← links)
- Caspase-1 is activated in neural cells and tissue with amyotrophic lateral sclerosis-associated mutations in copper-zinc superoxide dismutase. (Q33612216) (← links)
- Motor neurone metabolism. (Q33762425) (← links)
- Modeling Lou Gehrig's disease in the fruit fly. (Q33907487) (← links)
- Apoptosis and neurologic disease (Q34051306) (← links)
- Apoptosis in the nervous system (Q34068965) (← links)
- Possible extrapyramidal system degradation in Parkinson's disease (Q34117395) (← links)
- Ten years on: mediation of cell death by the common neurotrophin receptor p75(NTR). (Q34203015) (← links)
- Sporadic ALS associated with the D90A Cu,Zn superoxide dismutase mutation in Russia (Q34206250) (← links)
- Apoptosis in amyotrophic lateral sclerosis: a review of the evidence. (Q34352949) (← links)
- Ageing and apoptosis (Q34463468) (← links)
- Progress in clinical neurosciences: the evidence for ALS as a multisystems disorder of limited phenotypic expression. (Q34477724) (← links)
- Familial amyotrophic lateral sclerosis (Q34547620) (← links)
- Programmed cell death in amyotrophic lateral sclerosis (Q34831541) (← links)
- Therapeutic developments in the treatment of amyotrophic lateral sclerosis (Q34974850) (← links)
- The structural biochemistry of the superoxide dismutases. (Q34993111) (← links)
- Disulfide scrambling in superoxide dismutase 1 reduces its cytotoxic effect in cultured cells and promotes protein aggregation (Q35021849) (← links)
- Recent advances in amyotrophic lateral sclerosis research (Q35036503) (← links)
- Caspase inhibitors as anti-inflammatory and antiapoptotic agents. (Q35049064) (← links)
- Targeting programmed cell death in neurodegenerative diseases (Q35120138) (← links)
- Caspase-1 and -3 are sequentially activated in motor neuron death in Cu,Zn superoxide dismutase-mediated familial amyotrophic lateral sclerosis (Q35580300) (← links)
- Molecular genetics approaches in yeast to study amyloid diseases (Q35764314) (← links)
- Role of DNA mismatch repair in apoptotic responses to therapeutic agents (Q35910747) (← links)
- Oxidative stress mediates impairment of muscle function in transgenic mice with elevated level of wild-type Cu/Zn superoxide dismutase (Q36106817) (← links)
- Lower intracellular hydrogen peroxide levels in cells overexpressing CuZn-superoxide dismutase (Q36163220) (← links)
- Apoptosis in amyotrophic lateral sclerosis--what is the evidence? (Q36201880) (← links)
- Human Bcl-2 reverses survival defects in yeast lacking superoxide dismutase and delays death of wild-type yeast (Q36266887) (← links)
- Reexamination of the mechanism of hydroxyl radical adducts formed from the reaction between familial amyotrophic lateral sclerosis-associated Cu,Zn superoxide dismutase mutants and H2O2. (Q36485372) (← links)
- The role of mitochondria in inherited neurodegenerative diseases (Q36521932) (← links)
- Molecular biology of amyotrophic lateral sclerosis: insights from genetics (Q36573336) (← links)
- RNA interference-mediated silencing of mutant superoxide dismutase rescues cyclosporin A-induced death in cultured neuroblastoma cells (Q36854466) (← links)
- A drosophila model for amyotrophic lateral sclerosis reveals motor neuron damage by human SOD1. (Q36861919) (← links)
- Sensory involvement in the SOD1-G93A mouse model of amyotrophic lateral sclerosis (Q37184758) (← links)
- Reactive oxygen intermediates contribute to necrotic and apoptotic neuronal injury in an infant rat model of bacterial meningitis due to group B streptococci (Q37361937) (← links)