Pages that link to "Q34565321"
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The following pages link to Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts (Q34565321):
Displaying 50 items.
- Ca2+ Sparks and Ca2+ waves are the subcellular events underlying Ca2+ overload during ischemia and reperfusion in perfused intact hearts (Q27302397) (← links)
- Comparison of human induced pluripotent stem-cell derived cardiomyocytes with human mesenchymal stem cells following acute myocardial infarction (Q27318070) (← links)
- Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion (Q30888193) (← links)
- Brief rapid pacing depresses contractile function via Ca(2+)/PKC-dependent signaling in cat ventricular myocytes (Q31722171) (← links)
- Reperfusion injury: experimental evidence and clinical implications (Q33699781) (← links)
- Mechanisms of postischemic contractile dysfunction (Q33786654) (← links)
- Calcium and the anaesthetist (Q34007368) (← links)
- Activity of the Na+/H+ exchanger contributes to cardiac damage following ischaemia and reperfusion (Q34023035) (← links)
- Rapid expression of heat shock protein in the rabbit after brief cardiac ischemia (Q34175854) (← links)
- Calcium oscillations index the extent of calcium loading and predict functional recovery during reperfusion in rat myocardium (Q34245042) (← links)
- Intracellular free calcium concentration measured with 19F NMR spectroscopy in intact ferret hearts (Q34341287) (← links)
- Relationship between ionic perturbations and electrophysiologic changes in a canine Purkinje fiber model of ischemia and reperfusion (Q34557511) (← links)
- Acidosis during early reperfusion prevents myocardial stunning in perfused ferret hearts (Q34562428) (← links)
- Effects of exogenous free radicals on electromechanical function and metabolism in isolated rabbit and guinea pig ventricle. Implications for ischemia and reperfusion injury (Q34574070) (← links)
- Relationship between calcium loading and impaired energy metabolism during Na+, K+ pump inhibition and metabolic inhibition in cultured neonatal rat cardiac myocytes (Q34574527) (← links)
- Effects of brief ischemia and reperfusion on the myocardium and the role of nitric oxide (Q35135762) (← links)
- Novel mechanisms mediating stunned myocardium (Q35135768) (← links)
- Structure of the NH2-terminal variable region of cardiac troponin T determines its sensitivity to restrictive cleavage in pathophysiological adaptation (Q35336277) (← links)
- Triple threat: the Na+/Ca2+ exchanger in the pathophysiology of cardiac arrhythmia, ischemia and heart failure (Q35523734) (← links)
- An inhibitor of poly (ADP-ribose) synthetase activity reduces contractile dysfunction and preserves high energy phosphate levels during reperfusion of the ischaemic rat heart (Q35566493) (← links)
- Sustained left ventricular diastolic dysfunction after exercise in patients with dilated cardiomyopathy. (Q35567313) (← links)
- Extracellular Adenosine Formation by Ecto-5'-Nucleotidase (CD73) Is No Essential Trigger for Early Phase Ischemic Preconditioning (Q35742277) (← links)
- Beneficial effects of N-acetylcysteine and cysteine in stunned myocardium in perfused rat heart (Q35888637) (← links)
- The continuing evolution of the Langendorff and ejecting murine heart: new advances in cardiac phenotyping. (Q36115555) (← links)
- The importance of the Thr17 residue of phospholamban as a phosphorylation site under physiological and pathological conditions (Q36464238) (← links)
- New concepts of endoplasmic reticulum function in the heart: programmed to conserve (Q36570092) (← links)
- Early reperfusion phenomena (Q36588712) (← links)
- Ischemia-reperfusion and cardioprotection: a delicate balance between reactive oxygen species generation and redox homeostasis (Q36844809) (← links)
- Stunning: a radical re-view (Q36846547) (← links)
- Pathogenetic role for calcium in stunning? (Q36846562) (← links)
- Postischemic stunning?The case for calcium as the ultimate culprit (Q36846573) (← links)
- Role of adenosine in the treatment of myocardial stunning (Q36846581) (← links)
- Calcium antagonists and stunned myocardium: Importance for clinicians? (Q36846609) (← links)
- Post-resuscitation hemodynamics and relationship to the duration of ventricular fibrillation (Q36914609) (← links)
- A cardioprotective role for platelet-activating factor through NOS-dependent S-nitrosylation (Q37119219) (← links)
- Role of calcium and other ions in reperfusion injury (Q37188908) (← links)
- Oxygen-derived free radicals and myocardial reperfusion injury: An overview (Q37188913) (← links)
- Abnormal Intracellular Modulation of Calcium as a Major Cause of Cardiac Contractile Dysfunction (Q37212377) (← links)
- Targeting calcium transport in ischaemic heart disease. (Q37422613) (← links)
- Effects of inotropic drugs on mechanical function and oxygen balance in postischemic canine myocardium: comparison of dobutamine, epinephrine, amrinone, and calcium chloride (Q37425657) (← links)
- Pathophysiology of stable angina pectoris. (Q37713607) (← links)
- Pathogenesis of myocardial ischemia-reperfusion injury and rationale for therapy (Q37774078) (← links)
- Calcium and pH in anoxic and toxic injury (Q37787068) (← links)
- Is the protection against ischemia induced by red wine linked to its antioxidant capacity? (Q40169798) (← links)
- Carbon monoxide increases inducible NOS expression that mediates CO-induced myocardial damage during ischemia-reperfusion (Q40250067) (← links)
- The vicious circle of ischemic left ventricular dysfunction (Q40511089) (← links)
- Stunned myocardium and the attenuation of stunning by calcium antagonists (Q40511127) (← links)
- Nuclear imaging techniques for the assessment of myocardial viability. (Q40553594) (← links)
- Myocardial stunning, hibernation, and ischemic preconditioning (Q40925196) (← links)
- Stunned myocardium: a disease of the myofilaments? (Q40930511) (← links)