Pages that link to "Q34564604"
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The following pages link to Role of endotoxemia in cardiovascular dysfunction and mortality. Escherichia coli and Staphylococcus aureus challenges in a canine model of human septic shock (Q34564604):
Displaying 50 items.
- Depression of constitutive murine cytochromes P450 by Staphylococcal enterotoxin B (Q23921829) (← links)
- Recognition of Staphylococcus aureus by the innate immune system (Q24530569) (← links)
- Does Bacillus anthracis Lethal Toxin Directly Depress Myocardial Function? A Review of Clinical Cases and Preclinical Studies (Q26772000) (← links)
- Towards clinical applications of anti-endotoxin antibodies; a re-appraisal of the disconnect (Q26996658) (← links)
- Changes of platelet surface antigens in patients suffering from abdominal septic shock (Q33330333) (← links)
- Staphylococcus epidermidis induces complement activation, tumor necrosis factor and interleukin-1, a shock-like state and tissue injury in rabbits without endotoxemia. Comparison to Escherichia coli (Q33389219) (← links)
- Interleukin-1 (IL-1) receptor antagonist prevents Staphylococcus epidermidis-induced hypotension and reduces circulating levels of tumor necrosis factor and IL-1 beta in rabbits (Q33493710) (← links)
- Gram-positive sepsis. Mechanisms and differences from gram-negative sepsis. (Q33642547) (← links)
- Lipopolysaccharides induced inflammatory responses and electrophysiological dysfunctions in human-induced pluripotent stem cell derived cardiomyocytes (Q33774097) (← links)
- Gram-negative bacterial sepsis and the sepsis syndrome (Q33786303) (← links)
- The cell wall components peptidoglycan and lipoteichoic acid from Staphylococcus aureus act in synergy to cause shock and multiple organ failure (Q33838051) (← links)
- Myocardial dysfunction in septic shock (Q33893312) (← links)
- Commercial preparations of lipoteichoic acid contain endotoxin that contributes to activation of mouse macrophages in vitro (Q34005884) (← links)
- Experimental models of gram-negative sepsis (Q34117180) (← links)
- TLR2 stimulation induces cardiac inflammation but not cardiac depression in vivo (Q34255776) (← links)
- Endotoxemia: methods of detection and clinical correlates (Q34300796) (← links)
- Thaliporphine preserves cardiac function of endotoxemic rabbits by both directly and indirectly attenuating NFκB signaling pathway. (Q34325901) (← links)
- Cytokine appearance and effects of anti-tumor necrosis factor alpha antibodies in a neonatal rat model of group B streptococcal infection. (Q34521810) (← links)
- Gram-positive cell walls stimulate synthesis of tumor necrosis factor alpha and interleukin-6 by human monocytes. (Q34528810) (← links)
- Multiple organ dysfunction syndrome in humans and animals (Q34661567) (← links)
- Interactions between lipoteichoic acid and peptidoglycan from Staphylococcus aureus: a structural and functional analysis (Q34727384) (← links)
- Effects of antimicrobial peptide L-K6, a temporin-1CEb analog on oral pathogen growth, Streptococcus mutans biofilm formation, and anti-inflammatory activity (Q35212008) (← links)
- Endogenous gamma interferon, tumor necrosis factor, and interleukin-6 in Staphylococcus aureus infection in mice (Q35397535) (← links)
- Role of endotoxemia in cardiovascular dysfunction and lethality: virulent and nonvirulent Escherichia coli challenges in a canine model of septic shock (Q35465958) (← links)
- Streptococcus mitis cell walls and lipopolysaccharide induce lethality in D-galactosamine-sensitized mice by a tumor necrosis factor-dependent pathway. (Q35492787) (← links)
- Correlation of histopathologic and bacteriologic changes with cytokine expression in an experimental murine model of bacteremic Staphylococcus aureus infection (Q35555706) (← links)
- Left ventricular systolic and diastolic dysfunction after infusion of tumor necrosis factor-alpha in conscious dogs (Q35604862) (← links)
- Systemic hemodynamic abnormalities and vasopressor therapy in sepsis and septic shock (Q35804343) (← links)
- Endotoxin and tumor necrosis factor challenges in dogs simulate the cardiovascular profile of human septic shock (Q36356245) (← links)
- The third component of complement protects against Escherichia coli endotoxin-induced shock and multiple organ failure. (Q36362697) (← links)
- Molecules from Staphylococcus aureus that bind CD14 and stimulate innate immune responses (Q36365572) (← links)
- Pathobiology of sepsis: are we still asking the same questions? (Q36371860) (← links)
- Effects of intra-aortic balloon counterpulsation in a model of septic shock (Q37035299) (← links)
- Molecular events in the cardiomyopathy of sepsis (Q37078877) (← links)
- Tyrphostin AG 556 improves survival and reduces multiorgan failure in canine Escherichia coli peritonitis. (Q37366045) (← links)
- Negative inotropic effects of high-mobility group box 1 protein in isolated contracting cardiac myocytes (Q37416128) (← links)
- Human monoclonal antibody against endotoxin (Q37508824) (← links)
- Models of the pathogenesis of necrotizing enterocolitis (Q37817647) (← links)
- Innate immune signaling in cardiac ischemia (Q37859249) (← links)
- Septic shock: the cardiovascular abnormality and therapy (Q38246942) (← links)
- Are platelets a 'forgotten' source of sepsis-induced myocardial depressing factor(s)? (Q38275288) (← links)
- Left ventricular systolic dysfunction during septic shock: the role of loading conditions (Q38958698) (← links)
- A canine model of septic shock: balancing animal welfare and scientific relevance (Q39332693) (← links)
- Interaction of cationic peptides with lipoteichoic acid and gram-positive bacteria (Q39513309) (← links)
- Induction of nitric oxide production by polyosides from the cell walls of Streptococcus mutans OMZ 175, a gram-positive bacterium, in the rat aorta (Q39830001) (← links)
- Gas gangrene (Q40131494) (← links)
- Sepsis in the critically ill patient (Q40381960) (← links)
- Antiendotoxin monoclonal antibodies. What future now? (Q40587497) (← links)
- Response of man to endotoxin (Q40825144) (← links)
- Mechanism of gram-positive shock: identification of peptidoglycan and lipoteichoic acid moieties essential in the induction of nitric oxide synthase, shock, and multiple organ failure (Q41024121) (← links)