Pages that link to "Q34543633"
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The following pages link to Regional selectivity of rab5 and rab7 protein upregulation in mild cognitive impairment and Alzheimer's disease (Q34543633):
Displaying 50 items.
- Gene expression parallels synaptic excitability and plasticity changes in Alzheimer's disease (Q26785375) (← links)
- The synaptic maintenance problem: membrane recycling, Ca2+ homeostasis and late onset degeneration (Q27000356) (← links)
- Amyloid precursor protein-mediated endocytic pathway disruption induces axonal dysfunction and neurodegeneration (Q27331605) (← links)
- Rabx-5 regulates RAB-5 early endosomal compartments and synaptic vesicles in C. elegans (Q31063785) (← links)
- Acid sphingomyelinase modulates the autophagic process by controlling lysosomal biogenesis in Alzheimer's disease (Q33964297) (← links)
- Pyroglutamate-amyloid-β and glutaminyl cyclase are colocalized with amyloid-β in secretory vesicles and undergo activity-dependent, regulated secretion (Q34076284) (← links)
- Expression profile analysis of vulnerable CA1 pyramidal neurons in young-Middle-Aged Ts65Dn mice (Q34514070) (← links)
- Rab GTPases regulate endothelial cell protein C receptor-mediated endocytosis and trafficking of factor VIIa (Q34648964) (← links)
- Expression profile analysis of hippocampal CA1 pyramidal neurons in aged Ts65Dn mice, a model of Down syndrome (DS) and Alzheimer's disease (AD). (Q34979826) (← links)
- Upregulation of select rab GTPases in cholinergic basal forebrain neurons in mild cognitive impairment and Alzheimer's disease. (Q35186435) (← links)
- Hippocampal endosomal, lysosomal, and autophagic dysregulation in mild cognitive impairment: correlation with aβ and tau pathology (Q35196299) (← links)
- Hook proteins: association with Alzheimer pathology and regulatory role of hook3 in amyloid beta generation (Q35208328) (← links)
- Significance of transcytosis in Alzheimer's disease: BACE1 takes the scenic route to axons (Q35883077) (← links)
- Strain-Specific Altered Regulatory Response of Rab7a and Tau in Creutzfeldt-Jakob Disease and Alzheimer's Disease (Q35894510) (← links)
- Neuronal-Targeted TFEB Accelerates Lysosomal Degradation of APP, Reducing Aβ Generation and Amyloid Plaque Pathogenesis (Q36017239) (← links)
- Hippocampal plasticity during the progression of Alzheimer's disease (Q36054541) (← links)
- Hippocampal proNGF signaling pathways and β-amyloid levels in mild cognitive impairment and Alzheimer disease (Q36347400) (← links)
- Targeting LAMP2 in human cerebrospinal fluid with a combination of immunopurification and high resolution parallel reaction monitoring mass spectrometry (Q36622154) (← links)
- Membrane trafficking in neuronal maintenance and degeneration. (Q37043326) (← links)
- Reducing Endogenous α-Synuclein Mitigates the Degeneration of Selective Neuronal Populations in an Alzheimer's Disease Transgenic Mouse Model (Q37127867) (← links)
- Protein homeostasis gene dysregulation in pretangle-bearing nucleus basalis neurons during the progression of Alzheimer's disease (Q37152711) (← links)
- Meta-analysis of synaptic pathology in Alzheimer's disease reveals selective molecular vesicular machinery vulnerability (Q37326829) (← links)
- Lysosomal network proteins as potential novel CSF biomarkers for Alzheimer's disease (Q37568914) (← links)
- Autophagic/lysosomal dysfunction in Alzheimer's disease (Q37690932) (← links)
- Mechanisms of neural and behavioral dysfunction in Alzheimer's disease (Q37855656) (← links)
- The neuropathological profile of mild cognitive impairment (MCI): a systematic review. (Q37964897) (← links)
- Lysosomal fusion dysfunction as a unifying hypothesis for Alzheimer's disease pathology (Q38042636) (← links)
- G proteins, p60TRP, and neurodegenerative diseases (Q38076364) (← links)
- The role of rab proteins in neuronal cells and in the trafficking of neurotrophin receptors (Q38258099) (← links)
- Rab21, a Novel PS1 Interactor, Regulates γ-Secretase Activity via PS1 Subcellular Distribution. (Q38433126) (← links)
- Enhanced exosome secretion in Down syndrome brain - a protective mechanism to alleviate neuronal endosomal abnormalities (Q38603319) (← links)
- Molecular and cellular pathophysiology of preclinical Alzheimer's disease (Q38836004) (← links)
- Rab7A regulates tau secretion (Q38950095) (← links)
- Critical importance of RAB proteins for synaptic function. (Q39113567) (← links)
- Rab7 may be a novel therapeutic target for neurologic diseases as a key regulator in autophagy. (Q39130182) (← links)
- Probing the druggability of membrane-bound Rab5 by molecular dynamics simulations (Q40370713) (← links)
- The Enigmatic Role of C9ORF72 in Autophagy (Q41222674) (← links)
- Snapin-mediated BACE1 retrograde transport is essential for its degradation in lysosomes and regulation of APP processing in neurons (Q41896833) (← links)
- Withdrawal of BDNF from hippocampal cultures leads to changes in genes involved in synaptic function (Q43148661) (← links)
- Commentary: Rab GTPase: A New Mitotic Delivery Service (Q43172546) (← links)
- Dysregulation of neurotrophin signaling in the pathogenesis of Alzheimer disease and of Alzheimer disease in Down syndrome (Q46073648) (← links)
- Consequences of Rab GTPase dysfunction in genetic or acquired human diseases (Q47294747) (← links)
- The Endosomal-Lysosomal Pathway Is Dysregulated by APOE4 Expression in Vivo (Q47369828) (← links)
- Apolipoprotein E4 Impairs Neuronal Insulin Signaling by Trapping Insulin Receptor in the Endosomes (Q47731042) (← links)
- Par3 and aPKC regulate BACE1 endosome-to-TGN trafficking through PACS1. (Q47747110) (← links)
- Dysregulation of Rab5-Mediated Endocytic Pathways in Alzheimer's Disease (Q47875216) (← links)
- The stress-induced transcription factor NR4A1 adjusts mitochondrial function and synapse number in prefrontal cortex. (Q47885881) (← links)
- Dihydroceramide Desaturase 1 Inhibitors Reduce Amyloid-β Levels in Primary Neurons from an Alzheimer's Disease Transgenic Model. (Q48093215) (← links)
- Neocortical and hippocampal TREM2 protein levels during the progression of Alzheimer's disease. (Q48235737) (← links)
- Dysfunctions in endosomal-lysosomal and autophagy pathways underlie neuropathology in a mouse model for Lafora disease. (Q48866961) (← links)