Pages that link to "Q34378827"

The following pages link to Abasic sites preferentially form at regions undergoing DNA replication (Q34378827):

Displaying 14 items.

• Suicidal cross-linking of PARP-1 to AP site intermediates in cells undergoing base excision repair. (Q33698661) (← links)
• Temporal and functional analysis of DNA replicated in early S phase (Q34810780) (← links)
• Endonuclease IV Is the major apurinic/apyrimidinic endonuclease in Mycobacterium tuberculosis and is important for protection against oxidative damage (Q34926905) (← links)
• APE1-mediated DNA damage repair provides survival advantage for esophageal adenocarcinoma cells in response to acidic bile salts (Q37086100) (← links)
• Correlation of bistranded clustered abasic DNA lesion processing with structural and dynamic DNA helix distortion (Q37336997) (← links)
• Elevated level of acetylation of APE1 in tumor cells modulates DNA damage repair (Q37688921) (← links)
• Role of oxidative stress and DNA damage in human carcinogenesis (Q37826939) (← links)
• DNA damage in cells exhibiting radiation-induced genomic instability (Q38906538) (← links)
• Untwisting of the DNA helix stimulates the endonuclease activity of Bacillus subtilis Nth at AP sites (Q40395945) (← links)
• Oxidative stress at low levels can induce clustered DNA lesions leading to NHEJ mediated mutations (Q41224707) (← links)
• 2,3,7,8-Tetrachlorodibenzo-p-dioxin modulates estradiol-induced aldehydic DNA lesions in human breast cancer cells through alteration of CYP1A1 and CYP1B1 expression (Q45892349) (← links)
• Evolutionary analysis indicates that DNA alkylation damage is a byproduct of cytosine DNA methyltransferase activity. (Q49830775) (← links)
• Sequencing abasic sites in DNA at single-nucleotide resolution (Q92826279) (← links)
• Repair pathway for PARP-1 DNA-protein crosslinks (Q93355907) (← links)