Pages that link to "Q34360665"
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The following pages link to Prereplicative repair of oxidized bases in the human genome is mediated by NEIL1 DNA glycosylase together with replication proteins (Q34360665):
Displaying 50 items.
- Gut Microbiota Imbalance and Base Excision Repair Dynamics in Colon Cancer (Q26740627) (← links)
- The current state of eukaryotic DNA base damage and repair (Q26777671) (← links)
- New perspectives on oxidized genome damage and repair inhibition by pro-oxidant metals in neurological diseases (Q26996127) (← links)
- Insights into the glycosylase search for damage from single-molecule fluorescence microscopy (Q30585004) (← links)
- Microhomology-mediated end joining is activated in irradiated human cells due to phosphorylation-dependent formation of the XRCC1 repair complex (Q33557991) (← links)
- Destabilization of the PCNA trimer mediated by its interaction with the NEIL1 DNA glycosylase (Q33558133) (← links)
- No cancer predisposition or increased spontaneous mutation frequencies in NEIL DNA glycosylases-deficient mice (Q33854259) (← links)
- Base excision repair: a critical player in many games (Q33908163) (← links)
- New paradigms in the repair of oxidative damage in human genome: mechanisms ensuring repair of mutagenic base lesions during replication and involvement of accessory proteins. (Q35395549) (← links)
- The role of the mammalian DNA end-processing enzyme polynucleotide kinase 3'-phosphatase in spinocerebellar ataxia type 3 pathogenesis (Q35551473) (← links)
- The NEIL glycosylases remove oxidized guanine lesions from telomeric and promoter quadruplex DNA structures (Q35562087) (← links)
- Redox regulation of genome stability by effects on gene expression, epigenetic pathways and DNA damage/repair. (Q35764015) (← links)
- The C-terminal Domain (CTD) of Human DNA Glycosylase NEIL1 Is Required for Forming BERosome Repair Complex with DNA Replication Proteins at the Replicating Genome: DOMINANT NEGATIVE FUNCTION OF THE CTD. (Q35978248) (← links)
- Efficient and Reliable Production of Vectors for the Study of the Repair, Mutagenesis, and Phenotypic Consequences of Defined DNA Damage Lesions in Mammalian Cells (Q36064927) (← links)
- Neil2-null Mice Accumulate Oxidized DNA Bases in the Transcriptionally Active Sequences of the Genome and Are Susceptible to Innate Inflammation (Q36139765) (← links)
- Tautomerization-dependent recognition and excision of oxidation damage in base-excision DNA repair (Q37102321) (← links)
- Cockayne Syndrome group B protein stimulates NEIL2 DNA glycosylase activity. (Q37638150) (← links)
- Regulation of oxidized base damage repair by chromatin assembly factor 1 subunit A. (Q37649433) (← links)
- Scaffold attachment factor A (SAF-A) and Ku temporally regulate repair of radiation-induced clustered genome lesions (Q37688079) (← links)
- Mechanisms of base substitution mutagenesis in cancer genomes (Q37689834) (← links)
- DNA glycosylases search for and remove oxidized DNA bases (Q37719258) (← links)
- Hypersensitivity of mouse NEIL1-knockdown cells to hydrogen peroxide during S phase (Q39007066) (← links)
- Formation and processing of DNA damage substrates for the hNEIL enzymes (Q39018239) (← links)
- A role for the base excision repair enzyme NEIL3 in replication-dependent repair of interstrand DNA cross-links derived from psoralen and abasic sites (Q39163453) (← links)
- Repair of oxidatively induced DNA damage by DNA glycosylases: Mechanisms of action, substrate specificities and excision kinetics (Q39200289) (← links)
- DNA2-An Important Player in DNA Damage Response or Just Another DNA Maintenance Protein? (Q39441548) (← links)
- hMYH and hMTH1 cooperate for survival in mismatch repair defective T-cell acute lymphoblastic leukemia (Q39678540) (← links)
- Pre-Replicative Repair of Oxidized Bases Maintains Fidelity in Mammalian Genomes: The Cowcatcher Role of NEIL1 DNA Glycosylase (Q41225003) (← links)
- Aurora kinase B dependent phosphorylation of 53BP1 is required for resolving merotelic kinetochore-microtubule attachment errors during mitosis (Q41472052) (← links)
- Nei-like 1 (NEIL1) excises 5-carboxylcytosine directly and stimulates TDG-mediated 5-formyl and 5-carboxylcytosine excision (Q41484741) (← links)
- Avoidance of APOBEC3B-induced mutation by error-free lesion bypass (Q41693962) (← links)
- NEIL3 Repairs Telomere Damage during S Phase to Secure Chromosome Segregation at Mitosis (Q41712614) (← links)
- Ubiquitylation-dependent regulation of NEIL1 by Mule and TRIM26 is required for the cellular DNA damage response (Q42116749) (← links)
- Visualizing the Search for Radiation-damaged DNA Bases in Real Time (Q42683844) (← links)
- Chromatin-Bound Oxidized α-Synuclein Causes Strand Breaks in Neuronal Genomes in in vitro Models of Parkinson's Disease (Q46332709) (← links)
- The NEIL1 G83D germline DNA glycosylase variant induces genomic instability and cellular transformation. (Q47100233) (← links)
- The Human DNA glycosylases NEIL1 and NEIL3 Excise Psoralen-Induced DNA-DNA Cross-Links in a Four-Stranded DNA Structure. (Q47139042) (← links)
- Non-bulky Lesions in Human DNA: the Ways of Formation, Repair, and Replication (Q47143560) (← links)
- Loss of NEIL3 DNA glycosylase markedly increases replication associated double strand breaks and enhances sensitivity to ATR inhibitor in glioblastoma cells (Q47652095) (← links)
- Monitoring of the spatial and temporal dynamics of BER/SSBR pathway proteins, including MYH, UNG2, MPG, NTH1 and NEIL1-3, during DNA replication (Q49700613) (← links)
- Acetylation of oxidized base repair-initiating NEIL1 DNA glycosylase required for chromatin-bound repair complex formation in the human genome increases cellular resistance to oxidative stress. (Q52308714) (← links)
- Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis (Q58751835) (← links)
- Stress Marks on the Genome: Use or Lose? (Q61815168) (← links)
- Amyotrophic lateral sclerosis-associated TDP-43 mutation Q331K prevents nuclear translocation of XRCC4-DNA ligase 4 complex and is linked to genome damage-mediated neuronal apoptosis (Q64387024) (← links)
- Motor neuron disease-associated loss of nuclear TDP-43 is linked to DNA double-strand break repair defects (Q64387891) (← links)
- A Perspective on Chromosomal Double Strand Break Markers in Mammalian Cells (Q64390175) (← links)
- NEIL1 and NEIL2 DNA glycosylases protect neural crest development against mitochondrial oxidative stress (Q83228165) (← links)
- Effects of the stimuli-dependent enrichment of 8-oxoguanine DNA glycosylase1 on chromatinized DNA (Q89236313) (← links)
- The Biochemical Role of the Human NEIL1 and NEIL3 DNA Glycosylases on Model DNA Replication Forks (Q90278819) (← links)
- Alkyladenine DNA glycosylase associates with transcription elongation to coordinate DNA repair with gene expression (Q91558544) (← links)