Pages that link to "Q34228625"

The following pages link to CagI is an essential component of the Helicobacter pylori Cag type IV secretion system and forms a complex with CagL. (Q34228625):

Displaying 25 items.

• Structure of a three-dimensional domain-swapped dimer of the Helicobacter pylori type IV secretion system pilus protein CagL (Q27690205) ‎ (← links)
• Cag type IV secretion system: CagI independent bacterial surface localization of CagA (Q28533176) ‎ (← links)
• Analysis of surface-exposed outer membrane proteins in Helicobacter pylori. (Q30153404) ‎ (← links)
• Factors that mediate colonization of the human stomach by Helicobacter pylori (Q33626756) ‎ (← links)
• Genes required for assembly of pili associated with the Helicobacter pylori cag type IV secretion system (Q34058916) ‎ (← links)
• Composition, structure and function of the Helicobacter pylori cag pathogenicity island encoded type IV secretion system (Q35823457) ‎ (← links)
• Helicobacter pylori HP0231 Influences Bacterial Virulence and Is Essential for Gastric Colonization (Q36006505) ‎ (← links)
• Inhibitory effects of anthocyanins on secretion of Helicobacter pylori CagA and VacA toxins (Q36400856) ‎ (← links)
• Life in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori (Q37069986) ‎ (← links)
• The functional interplay of Helicobacter pylori factors with gastric epithelial cells induces a multi-step process in pathogenesis (Q38149990) ‎ (← links)
• Molecular Mechanisms of Helicobacter pylori Pathogenesis (Q38339445) ‎ (← links)
• Helicobacter pylori virulence factors in development of gastric carcinoma (Q38582534) ‎ (← links)
• A novel NOD1- and CagA-independent pathway of interleukin-8 induction mediated by the Helicobacter pylori type IV secretion system. (Q39252274) ‎ (← links)
• Functional interaction and structural characteristics of unique components of Helicobacter pylori T4SS. (Q39282756) ‎ (← links)
• Helicobacter pylori exploits human CEACAMs via HopQ for adherence and translocation of CagA. (Q39283179) ‎ (← links)
• The Helicobacter pylori Cag Pathogenicity Island Protein Cag1 is Associated with the Function of T4SS. (Q39925204) ‎ (← links)
• Quantitative analysis of CagA type IV secretion by Helicobacter pylori reveals substrate recognition and translocation requirements (Q40162217) ‎ (← links)
• Genetic variants of Helicobacter pylori type IV secretion system components CagL and CagI and their association with clinical outcomes (Q40239139) ‎ (← links)
• Systematic site-directed mutagenesis of the Helicobacter pylori CagL protein of the Cag type IV secretion system identifies novel functional domains (Q40422135) ‎ (← links)
• A C-Terminal Coiled-Coil Region of CagL is Responsible for Helicobacter Pylori-Induced Il-8 Expression (Q40495058) ‎ (← links)
• Integrin engagement by the helical RGD motif of the Helicobacter pylori CagL protein is regulated by pH-induced displacement of a neighboring helix. (Q40680982) ‎ (← links)
• Molecular dissection of protein-protein interactions between integrin α5β1 and the Helicobacter pylori Cag Type IV secretion system (Q42698144) ‎ (← links)
• Biochemical characterization of the Helicobacter pylori Cag-type IV secretion system unique component CagU. (Q51181211) ‎ (← links)
• In Vivo Structures of the Helicobacter pylori cag Type IV Secretion System. (Q52718557) ‎ (← links)
• CagL polymorphisms D58/K59 are predominant in strains isolated from Mexican patients with chronic gastritis (Q64239306) ‎ (← links)