Pages that link to "Q34204951"
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The following pages link to Little evidence for involvement of MLH3 in colorectal cancer predisposition (Q34204951):
Displaying 19 items.
- Review of the Lynch syndrome: history, molecular genetics, screening, differential diagnosis, and medicolegal ramifications (Q24610838) (← links)
- Impact of DNA mismatch repair system alterations on human fertility and related treatments (Q28074585) (← links)
- A review of the clinical relevance of mismatch-repair deficiency in ovarian cancer (Q28283356) (← links)
- The biochemical basis of microsatellite instability and abnormal immunohistochemistry and clinical behavior in Lynch syndrome: from bench to bedside (Q33762979) (← links)
- Mutation screening of mismatch repair gene Mlh3 in familial esophageal cancer (Q33869354) (← links)
- Evidence that hMLH3 functions primarily in meiosis and in hMSH2-hMSH3 mismatch repair. (Q34441207) (← links)
- Genetics, Cytogenetics, and Epigenetics of Colorectal Cancer (Q34758641) (← links)
- Mismatch repair gene MLH3 Pro844Leu and Thr942Ile polymorphisms and the susceptibility to cervical carcinoma and HPV infection: a case-control study in a Chinese population (Q35156069) (← links)
- Evolution of the nomenclature for the hereditary colorectal cancer syndromes (Q36246478) (← links)
- Lynch syndrome genes (Q36246487) (← links)
- The role of MLH1, MSH2 and MSH6 in the development of multiple colorectal cancers (Q36615234) (← links)
- Genomic instability and carcinogenesis: an update. (Q37514904) (← links)
- Stable expression of MutLγ in human cells reveals no specific response to mismatched DNA, but distinct recruitment to damage sites. (Q39148893) (← links)
- An infant with MLH3 variants, FOXG1-duplication and multiple, benign cranial and spinal tumors: A clinical exome sequencing study (Q46564552) (← links)
- Guidelines on genetic evaluation and management of Lynch syndrome: a consensus statement by the US Multi-society Task Force on colorectal cancer (Q51063696) (← links)
- GAA•TTC repeat expansion in human cells is mediated by mismatch repair complex MutLγ and depends upon the endonuclease domain in MLH3 isoform one. (Q52359109) (← links)
- Expression analysis of MLH3, MLH1, and MSH4 in maturation arrest. (Q52621809) (← links)
- A frameshift mutation in exon 19 of MLH1 in a Chinese Lynch syndrome family: a pedigree study (Q61810176) (← links)
- Hereditary nonpolyposis colorectal cancer and related conditions (Q79098523) (← links)