Pages that link to "Q34055445"

The following pages link to Modifications to the hemagglutinin cleavage site control the virulence of a neurotropic H1N1 influenza virus (Q34055445):

Displaying 46 items.

• Hemagglutinin of Influenza A Virus Antagonizes Type I Interferon (IFN) Responses by Inducing Degradation of Type I IFN Receptor 1 (Q26700091) (← links)
• Aberrant coagulation causes a hyper-inflammatory response in severe influenza pneumonia (Q26753030) (← links)
• Plasminogen controls inflammation and pathogenesis of influenza virus infections via fibrinolysis (Q27335814) (← links)
• Ifitm3 limits the severity of acute influenza in mice (Q27342563) (← links)
• High-throughput profiling of influenza A virus hemagglutinin gene at single-nucleotide resolution (Q30362478) (← links)
• Inhibition of influenza virus infection and hemagglutinin cleavage by the protease inhibitor HAI-2. (Q30364158) (← links)
• Molecular determinants of influenza virus pathogenesis in mice. (Q30364868) (← links)
• Matriptase, HAT, and TMPRSS2 activate the hemagglutinin of H9N2 influenza A viruses (Q30424187) (← links)
• Plasmin-mediated activation of pandemic H1N1 influenza virus hemagglutinin is independent of the viral neuraminidase. (Q30427707) (← links)
• N-linked glycosylation of the hemagglutinin protein influences virulence and antigenicity of the 1918 pandemic and seasonal H1N1 influenza A viruses (Q30431513) (← links)
• ITK signalling via the Ras/IRF4 pathway regulates the development and function of Tr1 cells. (Q33827917) (← links)
• What lies ahead for the proprotein convertases? (Q34169463) (← links)
• Influenza virus-mediated membrane fusion: determinants of hemagglutinin fusogenic activity and experimental approaches for assessing virus fusion (Q34360974) (← links)
• Modeling of influenza-specific CD8+ T cells during the primary response indicates that the spleen is a major source of effectors (Q35403088) (← links)
• Host cell proteases: Critical determinants of coronavirus tropism and pathogenesis (Q35735214) (← links)
• Equine and Canine Influenza H3N8 Viruses Show Minimal Biological Differences Despite Phylogenetic Divergence (Q35745427) (← links)
• Kallistatin ameliorates influenza virus pathogenesis by inhibition of kallikrein-related peptidase 1-mediated cleavage of viral hemagglutinin (Q35960792) (← links)
• A single amino acid at the hemagglutinin cleavage site contributes to the pathogenicity and neurovirulence of H5N1 influenza virus in mice (Q36086623) (← links)
• Cleavage activation of the human-adapted influenza virus subtypes by matriptase reveals both subtype and strain specificities (Q36276614) (← links)
• PAR1 contributes to influenza A virus pathogenicity in mice (Q36497143) (← links)
• Amino acid 316 of hemagglutinin and the neuraminidase stalk length influence virulence of H9N2 influenza virus in chickens and mice (Q36607195) (← links)
• Acquisition of a novel eleven amino acid insertion directly N-terminal to a tetrabasic cleavage site confers intracellular cleavage of an H7N7 influenza virus hemagglutinin (Q36765363) (← links)
• Cleavage activation of human-adapted influenza virus subtypes by kallikrein-related peptidases 5 and 12 (Q36929003) (← links)
• Accurate Measurement of the Effects of All Amino-Acid Mutations on Influenza Hemagglutinin (Q37049959) (← links)
• Coagulation, protease-activated receptors, and viral myocarditis. (Q37619823) (← links)
• TMPRSS2 is a host factor that is essential for pneumotropism and pathogenicity of H7N9 influenza A virus in mice (Q37713993) (← links)
• Multiple roles of the coagulation protease cascade during virus infection (Q37723466) (← links)
• Activation of influenza viruses by proteases from host cells and bacteria in the human airway epithelium (Q38118946) (← links)
• A novel eight amino acid insertion contributes to the hemagglutinin cleavability and the virulence of a highly pathogenic avian influenza A (H7N3) virus in mice (Q38814900) (← links)
• Use of AAScatterPlot tool for monitoring the evolution of the hemagglutinin cleavage site in H9 avian influenza viruses (Q38850386) (← links)
• Modification of the hemagglutinin cleavage site allows indirect activation of avian influenza virus H9N2 by bacterial staphylokinase (Q41357037) (← links)
• A comparison of RSV and influenza in vitro kinetic parameters reveals differences in infecting time (Q48506169) (← links)
• How single mutations affect viral escape from broad and narrow antibodies to H1 influenza hemagglutinin. (Q55318229) (← links)
• Deep mutational scanning of hemagglutinin helps predict evolutionary fates of human H3N2 influenza variants (Q56888351) (← links)
• Glucosylceramidase Maintains Influenza Virus Infection by Regulating Endocytosis (Q64360090) (← links)
• Mapping person-to-person variation in viral mutations that escape polyclonal serum targeting influenza hemagglutinin (Q83230244) (← links)
• Glucosylceramide synthase maintains influenza virus entry and infection (Q89594440) (← links)
• The spike glycoprotein of the new coronavirus 2019-nCoV contains a furin-like cleavage site absent in CoV of the same clade (Q89712904) (← links)
• Elevated Plasmin(ogen) as a Common Risk Factor for COVID-19 Susceptibility (Q90682949) (← links)
• Coronavirus membrane fusion mechanism offers as a potential target for antiviral development (Q91766933) (← links)
• A high throughput screen for TMPRSS2 expression identifies FDA-approved and clinically advanced compounds that can limit SARS-CoV-2 entry (Q98578438) (← links)
• The sequence at Spike S1/S2 site enables cleavage by furin and phospho-regulation in SARS-CoV2 but not in SARS-CoV1 or MERS-CoV (Q100512389) (← links)
• Non-basic amino acids in the hemagglutinin proteolytic cleavage site of a European H9N2 avian influenza virus modulate virulence in turkeys (Q104073718) (← links)
• Spike glycoprotein and host cell determinants of SARS-CoV-2 entry and cytopathic effects (Q104457732) (← links)
• Ser-Leu substitution at P2 position of the hemagglutinin cleavage site attenuates replication and pathogenicity of Eurasian avian-like H1N2 swine influenza viruses (Q104570995) (← links)
• Biochemical evidence of furin specificity and potential for phospho-regulation at Spike protein S1/S2 cleavage site in SARS-CoV2 but not in SARS-CoV1 or MERS-CoV (Q107473208) (← links)