Pages that link to "Q33854529"

The following pages link to Identification of a contractile deficit in adult cardiac myocytes expressing hypertrophic cardiomyopathy-associated mutant troponin T proteins (Q33854529):

Displaying 39 items.

• Cardiac troponin mutations and restrictive cardiomyopathy (Q21328674) (← links)
• Genetic counselling for hypertrophic cardiomyopathy: are we ready for it? (Q24792466) (← links)
• Cell biology of sarcomeric protein engineering: disease modeling and therapeutic potential (Q27010320) (← links)
• Disease-causing mutations in cardiac troponin T: identification of a critical tropomyosin-binding region (Q28199174) (← links)
• The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms (Q28203734) (← links)
• A novel mutant cardiac troponin C disrupts molecular motions critical for calcium binding affinity and cardiomyocyte contractility (Q28265634) (← links)
• Threonine-5 at the N-terminus can modulate sarcolipin function in cardiac myocytes (Q33745811) (← links)
• A transgenic rabbit model for human hypertrophic cardiomyopathy (Q33903354) (← links)
• Molecular genetics of cardiomyopathies (Q33906480) (← links)
• The molecular genetic basis for hypertrophic cardiomyopathy (Q33939892) (← links)
• Hypertrophic cardiomyopathy: from genetics to treatment (Q33992358) (← links)
• Experimental therapies in hypertrophic cardiomyopathy (Q33997765) (← links)
• Tissue Doppler imaging consistently detects myocardial contraction and relaxation abnormalities, irrespective of cardiac hypertrophy, in a transgenic rabbit model of human hypertrophic cardiomyopathy (Q34008230) (← links)
• Molecular genetics and pathogenesis of hypertrophic cardiomyopathy (Q34021080) (← links)
• Ionizing radiation and genetic risks. XII. The concept of "potential recoverability correction factor" (PRCF) and its use for predicting the risk of radiation-inducible genetic disease in human live births (Q34056666) (← links)
• Altered regulation of cardiac muscle contraction by troponin T mutations that cause familial hypertrophic cardiomyopathy. (Q34399560) (← links)
• Molecular mechanisms of inherited cardiomyopathies (Q34882653) (← links)
• Hypertrophic cardiomyopathy: from gene defect to clinical disease (Q35087752) (← links)
• Cardiac troponin T and familial hypertrophic cardiomyopathy: an energetic affair (Q35679277) (← links)
• Molecular genetics in hypertrophic cardiomyopathy: towards individualized management of the disease (Q36344118) (← links)
• Sarcomeric proteins and familial hypertrophic cardiomyopathy: linking mutations in structural proteins to complex cardiovascular phenotypes (Q36369925) (← links)
• Gene transfer into cardiac myocytes. (Q36543945) (← links)
• Analysis of the molecular pathogenesis of cardiomyopathy-causing cTnT mutants I79N, ΔE96, and ΔK210. (Q36824519) (← links)
• Cardiac troponin T mutation in familial cardiomyopathy with variable remodeling and restrictive physiology (Q36954073) (← links)
• Designing heart performance by gene transfer. (Q37299072) (← links)
• Cardiac troponin structure-function and the influence of hypertrophic cardiomyopathy associated mutations on modulation of contractility (Q38724869) (← links)
• Recent advances in genetics and treatment of hypertrophic cardiomyopathy (Q39367648) (← links)
• Reduction of left ventricular longitudinal global and segmental systolic functions in patients with hypertrophic cardiomyopathy: Study of two-dimensional tissue motion annular displacement (Q40239041) (← links)
• A novel method to study contraction characteristics of a single cardiac myocyte using carbon fibers (Q43713948) (← links)
• Myofibrillar responsiveness to cAMP, PKA, and caffeine in an animal model of heart failure (Q44263809) (← links)
• Mutations in MYH7 reduce the force generating capacity of sarcomeres in human familial hypertrophic cardiomyopathy (Q44354566) (← links)
• cTnT1, a cardiac troponin T isoform, decreases myofilament tension and affects the left ventricular pressure waveform (Q45129891) (← links)
• Hypertrophic cardiomyopathy-linked mutation in troponin T causes myofibrillar disarray and pro-arrhythmic action potential changes in human iPSC cardiomyocytes. (Q50022359) (← links)
• Assessment of left ventricular systolic and diastolic abnormalities in patients with hypertrophic cardiomyopathy using real-time three-dimensional echocardiography and two-dimensional speckle tracking imaging (Q57044617) (← links)
• Direct, convergent hypersensitivity of calcium-activated force generation produced by hypertrophic cardiomyopathy mutant alpha-tropomyosins in adult cardiac myocytes (Q64381152) (← links)
• Pathogenesis of diverse clinical and pathological phenotypes in hypertrophic cardiomyopathy (Q73313263) (← links)
• Cardiac troponin T mutations: correlation between the type of mutation and the nature of myofilament dysfunction in transgenic mice (Q74619276) (← links)
• [Molecular genetics of cardiomyopathies] (Q77763042) (← links)
• Left ventricular regional systolic function in patient with hypertrophic cardiomyopathy by quantitative tissue velocity imaging (Q83371149) (← links)