Pages that link to "Q33279720"
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The following pages link to Inositol-1,4,5-trisphosphate-mediated spontaneous activity in mouse embryonic stem cell-derived cardiomyocytes (Q33279720):
Displaying 46 items.
- Excitation-contraction coupling of human induced pluripotent stem cell-derived cardiomyocytes (Q26781319) (← links)
- A coupled SYSTEM of intracellular Ca2+ clocks and surface membrane voltage clocks controls the timekeeping mechanism of the heart's pacemaker. (Q33722486) (← links)
- Calcium Handling in Human Induced Pluripotent Stem Cell Derived Cardiomyocytes (Q33869568) (← links)
- Developmental aspects of cardiac Ca(2+) signaling: interplay between RyR- and IP(3)R-gated Ca(2+) stores (Q33917202) (← links)
- Excitation-contraction coupling in ventricular myocytes is enhanced by paracrine signaling from mesenchymal stem cells (Q34215785) (← links)
- Rhythmic beating of stem cell-derived cardiac cells requires dynamic coupling of electrophysiology and Ca cycling (Q34479318) (← links)
- Absence of transverse tubules contributes to non-uniform Ca(2+) wavefronts in mouse and human embryonic stem cell-derived cardiomyocytes. (Q35115150) (← links)
- Cardiomyogenesis in the developing heart is regulated by c-kit-positive cardiac stem cells (Q35798731) (← links)
- Inhibition of cardiomyocytes differentiation of mouse embryonic stem cells by CD38/cADPR/Ca2+ signaling pathway (Q36318788) (← links)
- The role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth. (Q36610320) (← links)
- Functional integrity of the T-tubular system in cardiomyocytes depends on p21-activated kinase 1 (Q36921343) (← links)
- Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes (Q37125035) (← links)
- p21-Activated kinase1 (Pak1) is a negative regulator of NADPH-oxidase 2 in ventricular myocytes (Q37594666) (← links)
- Inositol 1,4,5-trisphosphate receptors and pacemaker rhythms (Q38020078) (← links)
- Cardiomyocytes derived from pluripotent stem cells: progress and prospects from China (Q38047654) (← links)
- Calcium signalling in developing cardiomyocytes: implications for model systems and disease (Q38340336) (← links)
- Regulation of calcium clock-mediated pacemaking by inositol-1,4,5-trisphosphate receptors in mouse sinoatrial nodal cells. (Q38547135) (← links)
- Calcium signaling in human stem cell-derived cardiomyocytes: Evidence from normal subjects and CPVT afflicted patients (Q38685507) (← links)
- Requirement of IP3 receptor 3 (IP3R3) in nitric oxide induced cardiomyocyte differentiation of mouse embryonic stem cells (Q38761576) (← links)
- Junctophilin 2 knockdown interfere with mitochondrium status in ESC-CMs and cardiogenesis of ES cells (Q39363091) (← links)
- Non-cardiomyocytes influence the electrophysiological maturation of human embryonic stem cell-derived cardiomyocytes during differentiation (Q39765000) (← links)
- Ca2+ removal mechanisms in mouse embryonic stem cell-derived cardiomyocytes. (Q39824864) (← links)
- The relevance of non-excitable cells for cardiac pacemaker function. (Q40068769) (← links)
- Pacemaking, arrhythmias, inotropy and hypertrophy: the many possible facets of IP3 signalling in cardiac myocytes (Q40143560) (← links)
- Decreased intercellular coupling improves the function of cardiac pacemakers derived from mouse embryonic stem cells. (Q41081640) (← links)
- Spatially defined InsP3-mediated signaling in embryonic stem cell-derived cardiomyocytes (Q41856437) (← links)
- Sources of calcium in agonist-induced contraction of rat distal colon smooth muscle in vitro (Q41861715) (← links)
- Role of Nitric Oxide, Nitric Oxide Synthase, Soluble Guanylyl Cyclase, and cGMP-Dependent Protein Kinase I in Mouse Stem Cell Cardiac Development (Q41862756) (← links)
- The Interaction between Adult Cardiac Fibroblasts and Embryonic Stem Cell-Derived Cardiomyocytes Leads to Proarrhythmic Changes in In Vitro Cocultures. (Q42542823) (← links)
- Spontaneous calcium oscillations regulate human cardiac progenitor cell growth (Q42638868) (← links)
- Local Ca2+ releases enable rapid heart rates in developing cardiomyocytes (Q43138209) (← links)
- Functional expression and pharmaceutical efficacy of cardiac-specific ion channels in human embryonic stem cell-derived cardiomyocytes (Q43500993) (← links)
- Endothelin-1 signalling controls early embryonic heart rate in vitro and in vivo. (Q44513922) (← links)
- Beating to time: calcium clocks, voltage clocks, and cardiac pacemaker activity (Q46158610) (← links)
- 1,4,5-Inositol trisphosphate-operated intracellular Ca(2+) stores and angiotensin-II/endothelin-1 signaling pathway are functional in human embryonic stem cell-derived cardiomyocytes (Q46345959) (← links)
- Why, where, and when do cardiac myocytes express inositol 1,4,5-trisphosphate receptors? (Q46861462) (← links)
- Influence of Electromechanical Activity on Cardiac Differentiation of Mouse Embryonic Stem Cells. (Q46985469) (← links)
- Optogenetic activation of Gq signalling modulates pacemaker activity of cardiomyocytes (Q50684287) (← links)
- Functional Characterization and Comparison of Intercellular Communication in Stem Cell-Derived Cardiomyocytes. (Q50977783) (← links)
- A key role of TRPC channels in the regulation of electromechanical activity of the developing heart. (Q51864131) (← links)
- Mitochondrial Ca2+ Influx Contributes to Arrhythmic Risk in Nonischemic Cardiomyopathy. (Q52600707) (← links)
- Electrophysiological properties and calcium handling of embryonic stem cell-derived cardiomyocytes. (Q53162438) (← links)
- Structural and Mechanistic Bases of Nuclear Calcium Signaling in Human Pluripotent Stem Cell-Derived Ventricular Cardiomyocytes (Q64098044) (← links)
- Na/Ca exchange in the atrium: Role in sinoatrial node pacemaking and excitation-contraction coupling (Q89576527) (← links)
- Mitochondrial Ca2+ flux modulates spontaneous electrical activity in ventricular cardiomyocytes (Q89998674) (← links)
- IP3R-Mediated Compensatory Mechanism for Calcium Handling in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes With Cardiac Ryanodine Receptor Deficiency (Q99237660) (← links)