Pages that link to "Q30942254"

The following pages link to Disruption of aldose reductase gene (Akr1b1) causes defect in urinary concentrating ability and divalent cation homeostasis (Q30942254):

Displaying 15 items.

• Loss of NFAT5 results in renal atrophy and lack of tonicity-responsive gene expression (Q24622498) (← links)
• Functional studies of aldo-keto reductases in Saccharomyces cerevisiae (Q27938824) (← links)
• Aldo-Keto Reductases 1B in Adrenal Cortex Physiology (Q28078668) (← links)
• Genetic restoration of aldose reductase to the collecting tubules restores maturation of the urine concentrating mechanism (Q28509075) (← links)
• Aldo keto reductase 1B7 and prostaglandin F2alpha are regulators of adrenal endocrine functions (Q33509010) (← links)
• Genetic deficiency of aldose reductase counteracts the development of diabetic nephropathy in C57BL/6 mice (Q34768479) (← links)
• Polyol pathway and diabetic peripheral neuropathy (Q34800429) (← links)
• Dual effect of lithium on NFAT5 activity in kidney cells (Q36098316) (← links)
• Aldo-Keto Reductases 1B in Endocrinology and Metabolism (Q36135903) (← links)
• Mouse models and the urinary concentrating mechanism in the new millennium (Q36967665) (← links)
• The betaine/GABA transporter and betaine: roles in brain, kidney, and liver (Q37735049) (← links)
• Aldose reductase and cardiovascular diseases, creating human-like diabetic complications in an experimental model. (Q37751655) (← links)
• Disruption of aldehyde reductase increases group size in dictyostelium (Q40627301) (← links)
• The aldo-keto reductase AKR1B7 coexpresses with renin without influencing renin production and secretion (Q44456995) (← links)
• Induction of Osmolyte Pathways in Skeletal Muscle Inflammation: Novel Biomarkers for Myositis (Q58573177) (← links)