Pages that link to "Q30629661"
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The following pages link to Shank3-mutant mice lacking exon 9 show altered excitation/inhibition balance, enhanced rearing, and spatial memory deficit (Q30629661):
Displaying 50 items.
- Autism-associated SHANK3 haploinsufficiency causes Ih channelopathy in human neurons (Q26269817) (← links)
- Excitation/Inhibition Imbalance in Animal Models of Autism Spectrum Disorders (Q26741939) (← links)
- Prefrontal Cortex and Social Cognition in Mouse and Man (Q26774748) (← links)
- Altered mGluR5-Homer scaffolds and corticostriatal connectivity in a Shank3 complete knockout model of autism. (Q27318286) (← links)
- Pharmacological enhancement of mGlu5 receptors rescues behavioral deficits in SHANK3 knock-out mice (Q27318675) (← links)
- Deletion of JMJD2B in neurons leads to defective spine maturation, hyperactive behavior and memory deficits in mouse (Q27339100) (← links)
- Neurobiology of social behavior abnormalities in autism and Williams syndrome (Q28076343) (← links)
- SHANK proteins: roles at the synapse and in autism spectrum disorder. (Q30008776) (← links)
- Integrative Analysis of Brain Region-specific Shank3 Interactomes for Understanding the Heterogeneity of Neuronal Pathophysiology Related to SHANK3 Mutations (Q33575832) (← links)
- Replicable in vivo physiological and behavioral phenotypes of the Shank3B null mutant mouse model of autism (Q33805524) (← links)
- Brain Injury Impairs Working Memory and Prefrontal Circuit Function (Q36275179) (← links)
- Post-transcriptional regulation of SHANK3 expression by microRNAs related to multiple neuropsychiatric disorders (Q36286987) (← links)
- Monogenic mouse models of autism spectrum disorders: Common mechanisms and missing links. (Q36716971) (← links)
- Proteomic Analysis of Post-synaptic Density Fractions from Shank3 Mutant Mice Reveals Brain Region Specific Changes Relevant to Autism Spectrum Disorder (Q37639891) (← links)
- Emerging Synaptic Molecules as Candidates in the Etiology of Neurological Disorders (Q37694175) (← links)
- Shank synaptic scaffold proteins: keys to understanding the pathogenesis of autism and other synaptic disorders (Q38580572) (← links)
- Behavioural Phenotypes and Neural Circuit Dysfunctions in Mouse Models of Autism Spectrum Disorder (Q38678071) (← links)
- Reelin-Haploinsufficiency Disrupts the Developmental Trajectory of the E/I Balance in the Prefrontal Cortex. (Q38996374) (← links)
- Bridging Autism Spectrum Disorders and Schizophrenia through inflammation and biomarkers - pre-clinical and clinical investigations (Q41611297) (← links)
- Drd3 Signaling in the Lateral Septum Mediates Early Life Stress-Induced Social Dysfunction. (Q47570033) (← links)
- Inhibitory control of the excitatory/inhibitory balance in psychiatric disorders (Q47835820) (← links)
- Shank3-deficient thalamocortical neurons show HCN channelopathy and alterations in intrinsic electrical properties. (Q48255997) (← links)
- Excitatory and inhibitory synaptic dysfunction in mania: an emerging hypothesis from animal model studies. (Q52328697) (← links)
- CRISPR/Cas9-induced shank3b mutant zebrafish display autism-like behaviors. (Q52431309) (← links)
- Brain region-specific disruption of Shank3 in mice reveals a dissociation for cortical and striatal circuits in autism-related behaviors. (Q52560212) (← links)
- Impaired Hippocampal Synaptic Plasticity and Enhanced Excitatory Transmission in a Novel Animal Model of Autism Spectrum Disorders with Telomerase Reverse Transcriptase Overexpression. (Q52562084) (← links)
- Cell-Type-Specific Shank2 Deletion in Mice Leads to Differential Synaptic and Behavioral Phenotypes. (Q53426995) (← links)
- Distinct and Dynamic ON and OFF Neural Ensembles in the Prefrontal Cortex Code Social Exploration (Q57111580) (← links)
- Distinct Phenotypes of Mouse Models Reflect Neuropsychiatric Spectrum Disorders of Human Patients With Variants (Q57175338) (← links)
- Behavioral Phenotyping of an Improved Mouse Model of Phelan-McDermid Syndrome with a Complete Deletion of the Gene (Q57297379) (← links)
- Environmental enrichment has minimal effects on behavior in the Shank3 complete knockout model of autism spectrum disorder (Q57468156) (← links)
- Integrative Brain Transcriptome Analysis Reveals Region-Specific and Broad Molecular Changes in -Overexpressing Mice (Q57752443) (← links)
- GABA Neuronal Deletion of Exons 14-16 in Mice Suppresses Striatal Excitatory Synaptic Input and Induces Social and Locomotor Abnormalities (Q57816596) (← links)
- Toward the Language Oscillogenome (Q58574000) (← links)
- Potentiation of Glutamatergic Synaptic Transmission Onto Dorsal Raphe Serotonergic Neurons in the Valproic Acid Model of Autism (Q59128049) (← links)
- Transcriptome analysis of Shank3-overexpressing mice reveals unique molecular changes in the hypothalamus (Q59326949) (← links)
- Shank and Zinc Mediate an AMPA Receptor Subunit Switch in Developing Neurons (Q59793434) (← links)
- Altered Behaviors and Impaired Synaptic Function in a Novel Rat Model With a Complete Deletion (Q64117957) (← links)
- Brain mGluR5 in Shank3B Mice Studied With [F]FPEB PET Imaging and Immunoblotting (Q64252196) (← links)
- Adult Ube3a Gene Reinstatement Restores the Electrophysiological Deficits of Prefrontal Cortex Layer 5 Neurons in a Mouse Model of Angelman Syndrome. (Q64966648) (← links)
- Rbfox1 Regulates Synaptic Transmission through the Inhibitory Neuron-Specific vSNARE Vamp1 (Q88283327) (← links)
- Shank3 mutation in a mouse model of autism leads to changes in the S-nitroso-proteome and affects key proteins involved in vesicle release and synaptic function (Q89486069) (← links)
- Dysfunction of cortical GABAergic neurons leads to sensory hyper-reactivity in a Shank3 mouse model of ASD (Q89984085) (← links)
- Inhibitory regulation of calcium transients in prefrontal dendritic spines is compromised by a nonsense Shank3 mutation (Q90246327) (← links)
- A DLG2 deficiency in mice leads to reduced sociability and increased repetitive behavior accompanied by aberrant synaptic transmission in the dorsal striatum (Q90275484) (← links)
- Autism-Associated Shank3 Is Essential for Homeostatic Compensation in Rodent V1 (Q90559278) (← links)
- Unexpected Compensatory Increase in Shank3 Transcripts in Shank3 Knock-Out Mice Having Partial Deletions of Exons (Q90679973) (← links)
- Shank3 Mice Carrying the Human Q321R Mutation Display Enhanced Self-Grooming, Abnormal Electroencephalogram Patterns, and Suppressed Neuronal Excitability and Seizure Susceptibility (Q91637313) (← links)
- Home-cage hypoactivity in mouse genetic models of autism spectrum disorder (Q91850695) (← links)
- Enhanced Glutamatergic Currents at Birth in Shank3 KO Mice (Q92240482) (← links)