Pages that link to "Q29617135"
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The following pages link to BH3 domains of BH3-only proteins differentially regulate Bax-mediated mitochondrial membrane permeabilization both directly and indirectly (Q29617135):
Displaying 50 items.
- Genome-wide prediction and validation of peptides that bind human prosurvival Bcl-2 proteins (Q21145296) (← links)
- Use of human cancer cell lines mitochondria to explore the mechanisms of BH3 peptides and ABT-737-induced mitochondrial membrane permeabilization (Q21562635) (← links)
- Noxa/Bcl-2 protein interactions contribute to bortezomib resistance in human lymphoid cells (Q24298561) (← links)
- Bcl-x(L) retrotranslocates Bax from the mitochondria into the cytosol (Q24298758) (← links)
- From death receptor to reactive oxygen species and c-Jun N-terminal protein kinase: the receptor-interacting protein 1 odyssey (Q24299330) (← links)
- Role of bile salt in regulating Mcl-1 phosphorylation and chemoresistance in hepatocellular carcinoma cells (Q24300062) (← links)
- Human Bop is a novel BH3-only member of the Bcl-2 protein family (Q24300623) (← links)
- Human nuclear clusterin mediates apoptosis by interacting with Bcl-XL through C-terminal coiled coil domain (Q24302151) (← links)
- Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis (Q24302248) (← links)
- Heterodimerization of BAK and MCL-1 activated by detergent micelles (Q24305060) (← links)
- BH3 domains other than Bim and Bid can directly activate Bax/Bak (Q24305138) (← links)
- Differential contribution of Puma and Noxa in dual regulation of p53-mediated apoptotic pathways (Q24306130) (← links)
- A stapled BID BH3 helix directly binds and activates BAX (Q24307663) (← links)
- Glycogen synthase kinase-3 regulates mitochondrial outer membrane permeabilization and apoptosis by destabilization of MCL-1 (Q24308005) (← links)
- BimL directly neutralizes Bcl-xL to promote Bax activation during UV-induced apoptosis (Q24309169) (← links)
- The vaccinia virus protein F1L interacts with Bim and inhibits activation of the pro-apoptotic protein Bax (Q24310440) (← links)
- Bax crystal structures reveal how BH3 domains activate Bax and nucleate its oligomerization to induce apoptosis (Q24313049) (← links)
- Mechanisms of apoptosis sensitivity and resistance to the BH3 mimetic ABT-737 in acute myeloid leukemia (Q24313505) (← links)
- Induction of necrotic cell death and mitochondrial permeabilization by heme binding protein 2/SOUL (Q24314304) (← links)
- The hepatitis C virus core protein contains a BH3 domain that regulates apoptosis through specific interaction with human Mcl-1 (Q24314941) (← links)
- PUMA Dissociates Bax and Bcl-X(L) to induce apoptosis in colon cancer cells (Q24316443) (← links)
- A short Nur77-derived peptide converts Bcl-2 from a protector to a killer (Q24319007) (← links)
- Bcl-xL phosphorylation at Ser49 by polo kinase 3 during cell cycle progression and checkpoints (Q24322034) (← links)
- Mutation to Bax beyond the BH3 domain disrupts interactions with pro-survival proteins and promotes apoptosis (Q24322798) (← links)
- Mitochondria primed by death signals determine cellular addiction to antiapoptotic BCL-2 family members (Q24337108) (← links)
- BH3 profiling identifies three distinct classes of apoptotic blocks to predict response to ABT-737 and conventional chemotherapeutic agents (Q24337209) (← links)
- MAP-1 is a mitochondrial effector of Bax. (Q24337383) (← links)
- p53 opens the mitochondrial permeability transition pore to trigger necrosis (Q24338796) (← links)
- Loss of Bif-1 suppresses Bax/Bak conformational change and mitochondrial apoptosis (Q24534937) (← links)
- Pharmacological manipulation of cell death: clinical applications in sight? (Q24536101) (← links)
- The Bcl-2 apoptotic switch in cancer development and therapy (Q24620437) (← links)
- PUMA, a potent killer with or without p53 (Q24631776) (← links)
- Protein phosphatase subunit G5PR is needed for inhibition of B cell receptor-induced apoptosis (Q24646492) (← links)
- The BH3-only protein Puma plays an essential role in cytokine deprivation induced apoptosis of mast cells (Q24650702) (← links)
- BH3 mimetics to improve cancer therapy; mechanisms and examples (Q24655483) (← links)
- An internal EELD domain facilitates mitochondrial targeting of Mcl-1 via a Tom70-dependent pathway (Q24670136) (← links)
- Puma cooperates with Bim, the rate-limiting BH3-only protein in cell death during lymphocyte development, in apoptosis induction (Q24677017) (← links)
- Potential of apoptotic pathway-targeted cancer therapeutic research: Where do we stand? (Q26771601) (← links)
- MicroRNAs Regulate Mitochondrial Function in Cerebral Ischemia-Reperfusion Injury (Q26780451) (← links)
- Regulation of Bim in Health and Disease (Q26781203) (← links)
- BH3 profiling--measuring integrated function of the mitochondrial apoptotic pathway to predict cell fate decisions (Q26828031) (← links)
- Anoikis molecular pathways and its role in cancer progression (Q26852158) (← links)
- The role of BH3-only proteins in apoptosis within the ovary (Q26853329) (← links)
- A new view of the lethal apoptotic pore (Q27000479) (← links)
- BAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial pore (Q27002044) (← links)
- The BCL2 Family: Key Mediators of the Apoptotic Response to Targeted Anticancer Therapeutics (Q27010154) (← links)
- Integration and oligomerization of Bax protein in lipid bilayers characterized by single molecule fluorescence study (Q27314915) (← links)
- Bim and Bmf synergize to induce apoptosis in Neisseria gonorrhoeae infection (Q27317352) (← links)
- Mito-priming as a method to engineer Bcl-2 addiction. (Q27334195) (← links)
- BH3 peptides induce mitochondrial fission and cell death independent of BAX/BAK (Q27349157) (← links)