Pages that link to "Q28609771"
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The following pages link to MLL targets SET domain methyltransferase activity to Hox gene promoters (Q28609771):
Displaying 50 items.
- Dual function of histone H3 lysine 36 methyltransferase ASH1 in regulation of Hox gene expression (Q21134949) (← links)
- Cdx4 and menin co-regulate Hoxa9 expression in hematopoietic cells (Q21144471) (← links)
- Proteasome inhibitors evoke latent tumor suppression programs in pro-B MLL leukemias through MLL-AF4 (Q24294703) (← links)
- The BRCT-domain containing protein PTIP links PAX2 to a histone H3, lysine 4 methyltransferase complex (Q24296996) (← links)
- Monoubiquitination of human histone H2B: the factors involved and their roles in HOX gene regulation (Q24297025) (← links)
- Leukemia proto-oncoprotein MLL forms a SET1-like histone methyltransferase complex with menin to regulate Hox gene expression (Q24297027) (← links)
- HoxA10 activates CDX4 transcription and Cdx4 activates HOXA10 transcription in myeloid cells (Q24299118) (← links)
- A higher-order complex containing AF4 and ENL family proteins with P-TEFb facilitates oncogenic and physiologic MLL-dependent transcription (Q24299122) (← links)
- Taspase1: a threonine aspartase required for cleavage of MLL and proper HOX gene expression (Q24300473) (← links)
- Histone methyltransferase MLL1 regulates MDR1 transcription and chemoresistance (Q24300652) (← links)
- PRMT6-mediated methylation of R2 in histone H3 antagonizes H3 K4 trimethylation (Q24303480) (← links)
- Recognition of a mononucleosomal histone modification pattern by BPTF via multivalent interactions (Q24303887) (← links)
- Solution structure of the nonmethyl-CpG-binding CXXC domain of the leukaemia-associated MLL histone methyltransferase (Q24304207) (← links)
- Physical association and coordinate function of the H3 K4 methyltransferase MLL1 and the H4 K16 acetyltransferase MOF (Q24305335) (← links)
- PTIP associates with MLL3- and MLL4-containing histone H3 lysine 4 methyltransferase complex (Q24305512) (← links)
- SET1 and p300 act synergistically, through coupled histone modifications, in transcriptional activation by p53 (Q24310032) (← links)
- Menin associates with a trithorax family histone methyltransferase complex and with the hoxc8 locus (Q24313196) (← links)
- Identification of the MLL2 complex as a coactivator for estrogen receptor alpha (Q24316007) (← links)
- Genome-wide promoter analysis of the SOX4 transcriptional network in prostate cancer cells (Q24317198) (← links)
- Pro isomerization in MLL1 PHD3-bromo cassette connects H3K4me readout to CyP33 and HDAC-mediated repression (Q24319024) (← links)
- The PAF complex synergizes with MLL fusion proteins at HOX loci to promote leukemogenesis (Q24319075) (← links)
- Modulation of 14-3-3 interaction with phosphorylated histone H3 by combinatorial modification patterns (Q24322623) (← links)
- Structural basis for the requirement of additional factors for MLL1 SET domain activity and recognition of epigenetic marks (Q24324784) (← links)
- Cross-talk between histone modifications in response to histone deacetylase inhibitors: MLL4 links histone H3 acetylation and histone H3K4 methylation (Q24336402) (← links)
- Crosstalk between leukemia-associated proteins MOZ and MLL regulates HOX gene expression in human cord blood CD34+ cells (Q24336563) (← links)
- New insight into the molecular mechanisms of MLL-associated leukemia (Q24336625) (← links)
- Knockdown of ALR (MLL2) reveals ALR target genes and leads to alterations in cell adhesion and growth (Q24337427) (← links)
- Histone H2B monoubiquitination functions cooperatively with FACT to regulate elongation by RNA polymerase II (Q24337471) (← links)
- Menin and MLL cooperatively regulate expression of cyclin-dependent kinase inhibitors (Q24337658) (← links)
- Lung cancer cell migration is regulated via repressing growth factor PTN/RPTP β/ζ signaling by menin (Q24339039) (← links)
- On the mechanism of multiple lysine methylation by the human mixed lineage leukemia protein-1 (MLL1) core complex (Q24339403) (← links)
- Global and Hox-specific roles for the MLL1 methyltransferase (Q24529562) (← links)
- The MLL fusion gene, MLL-AF4, regulates cyclin-dependent kinase inhibitor CDKN1B (p27kip1) expression (Q24536056) (← links)
- The tumor suppressor menin regulates hematopoiesis and myeloid transformation by influencing Hox gene expression (Q24536084) (← links)
- The parafibromin tumor suppressor protein is part of a human Paf1 complex (Q24557571) (← links)
- A silencing pathway to induce H3-K9 and H4-K20 trimethylation at constitutive heterochromatin (Q24561636) (← links)
- Hoxa9 and Meis1 are key targets for MLL-ENL-mediated cellular immortalization (Q24603527) (← links)
- Covalent histone modifications--miswritten, misinterpreted and mis-erased in human cancers (Q24605342) (← links)
- Epigenetic control of embryonic stem cell fate (Q24623769) (← links)
- Aberrant overexpression and function of the miR-17-92 cluster in MLL-rearranged acute leukemia (Q24624389) (← links)
- Frequent mutation of histone-modifying genes in non-Hodgkin lymphoma (Q24630610) (← links)
- Histone methylation regulates memory formation. (Q24632499) (← links)
- In vitro and in vivo analyses of a Phe/Tyr switch controlling product specificity of histone lysine methyltransferases (Q24645737) (← links)
- Regulation of mir-196b by MLL and its overexpression by MLL fusions contributes to immortalization (Q24646499) (← links)
- MLL repression domain interacts with histone deacetylases, the polycomb group proteins HPC2 and BMI-1, and the corepressor C-terminal-binding protein (Q24679773) (← links)
- The SET-domain protein superfamily: protein lysine methyltransferases (Q24811144) (← links)
- Targeting histone methylation for cancer therapy: enzymes, inhibitors, biological activity and perspectives (Q26744441) (← links)
- The many faces of histone H3K79 methylation (Q26745514) (← links)
- Epigenetic Mechanisms in Developmental Alcohol-Induced Neurobehavioral Deficits (Q26748876) (← links)
- Epigenetic regulators and their impact on therapy in acute myeloid leukemia (Q26765402) (← links)