Pages that link to "Q28588847"
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The following pages link to Bax directly induces release of cytochrome c from isolated mitochondria (Q28588847):
Displaying 50 items.
- B cell leukemia/lymphoma 2 (Q14863583) (← links)
- BCL2-associated X protein (Q14905404) (← links)
- Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria (Q22008484) (← links)
- Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner (Q22008727) (← links)
- Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC (Q22009974) (← links)
- Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria (Q22011013) (← links)
- The HIV-1 viral protein R induces apoptosis via a direct effect on the mitochondrial permeability transition pore (Q22011015) (← links)
- BAR: An apoptosis regulator at the intersection of caspases and Bcl-2 family proteins (Q22253337) (← links)
- KIAP, a novel member of the inhibitor of apoptosis protein family (Q24290762) (← links)
- Bcl-B, a novel Bcl-2 family member that differentially binds and regulates Bax and Bak (Q24290999) (← links)
- p53 has a direct apoptogenic role at the mitochondria (Q24298888) (← links)
- Adenovirus-mediated overexpression of p14(ARF) induces p53 and Bax-independent apoptosis (Q24300313) (← links)
- Humanin peptide suppresses apoptosis by interfering with Bax activation (Q24301403) (← links)
- Human alphaA- and alphaB-crystallins bind to Bax and Bcl-X(S) to sequester their translocation during staurosporine-induced apoptosis (Q24306784) (← links)
- Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors (Q24310559) (← links)
- Apoptosis repressor with caspase recruitment domain protects against cell death by interfering with Bax activation (Q24314624) (← links)
- Identification of novel in vivo phosphorylation sites of the human proapoptotic protein BAD: pore-forming activity of BAD is regulated by phosphorylation (Q24323069) (← links)
- Inhibition of mitochondrial neural cell death pathways by protein transduction of Bcl-2 family proteins (Q24324220) (← links)
- MAP-1 is a mitochondrial effector of Bax. (Q24337383) (← links)
- The permeability transition pore triggers Bax translocation to mitochondria during neuronal apoptosis (Q24337594) (← links)
- Therapeutic efficacy of PUMA for malignant glioma cells regardless of p53 status. (Q24542478) (← links)
- Mild trifunctional protein deficiency is associated with progressive neuropathy and myopathy and suggests a novel genotype-phenotype correlation (Q24564364) (← links)
- Mitochondrial release of caspase-2 and -9 during the apoptotic process (Q24676806) (← links)
- BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death (Q24680305) (← links)
- Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activity (Q24680994) (← links)
- Induction of cell death by the BH3-only Bcl-2 homolog Nbk/Bik is mediated by an entirely Bax-dependent mitochondrial pathway (Q24681629) (← links)
- Equine estrogens differentially inhibit DNA fragmentation induced by glutamate in neuronal cells by modulation of regulatory proteins involved in programmed cell death (Q24795759) (← links)
- Glutamate-induced apoptosis in primary cortical neurons is inhibited by equine estrogens via down-regulation of caspase-3 and prevention of mitochondrial cytochrome c release (Q24806246) (← links)
- Molecular and cellular mechanisms of cardiotoxicity (Q24814079) (← links)
- BAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial pore (Q27002044) (← links)
- A review of ceramide analogs as potential anticancer agents (Q27009351) (← links)
- Anticancer Effect of Lycopene in Gastric Carcinogenesis (Q27015650) (← links)
- Solution structure of BID, an intracellular amplifier of apoptotic signaling (Q27617719) (← links)
- The structure of a Bcl-xL/Bim fragment complex: implications for Bim function (Q27642185) (← links)
- BH3-only proteins: a 20-year stock-take (Q28085220) (← links)
- Investigation of bax-induced release of cytochrome c from yeast mitochondria permeability of mitochondrial membranes, role of VDAC and ATP requirement (Q28140258) (← links)
- Oocyte apoptosis is suppressed by disruption of the acid sphingomyelinase gene or by sphingosine-1-phosphate therapy (Q28140319) (← links)
- Cytochrome c release and caspase activation in traumatic axonal injury (Q28140984) (← links)
- Human gelsolin prevents apoptosis by inhibiting apoptotic mitochondrial changes via closing VDAC (Q28141702) (← links)
- Interaction of Alzheimer's presenilin-1 and presenilin-2 with Bcl-X(L). A potential role in modulating the threshold of cell death (Q28142036) (← links)
- Aspirin induces apoptosis through release of cytochrome c from mitochondria (Q28176509) (← links)
- Mitochondrial protease Omi/HtrA2 enhances caspase activation through multiple pathways (Q28182577) (← links)
- The oat mitochondrial permeability transition and its implication in victorin binding and induced cell death (Q28201205) (← links)
- Essential role of voltage-dependent anion channel in various forms of apoptosis in mammalian cells (Q28206500) (← links)
- 14-3-3 Interacts directly with and negatively regulates pro-apoptotic Bax (Q28214507) (← links)
- Activation of mitochondrial voltage-dependent anion channel by apro-apoptotic BH3-only protein Bim (Q28214708) (← links)
- Caspase-2 induces apoptosis by releasing proapoptotic proteins from mitochondria (Q28217828) (← links)
- Survivin splice variants regulate the balance between proliferation and cell death (Q28305497) (← links)
- Induction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactions (Q28343497) (← links)
- Effects and mechanisms of emodin on cell death in human lung squamous cell carcinoma (Q28346695) (← links)