Pages that link to "Q27860821"

The following pages link to Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease (Q27860821):

Displaying 50 items.

• Systematic association mapping identifies NELL1 as a novel IBD disease gene (Q21090112) ‎ (← links)
• Intestinal DMBT1 expression is modulated by Crohn's disease-associated IL23R variants and by a DMBT1 variant which influences binding of the transcription factors CREB1 and ATF-2 (Q21090700) ‎ (← links)
• Crohn's disease and early exposure to domestic refrigeration (Q21092151) ‎ (← links)
• Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics (Q21131205) ‎ (← links)
• Genetic Association of Peptidoglycan Recognition Protein Variants with Inflammatory Bowel Disease (Q21133003) ‎ (← links)
• A proposed classification of the immunological diseases (Q21144701) ‎ (← links)
• Variants in a novel epidermal collagen gene (COL29A1) are associated with atopic dermatitis (Q21145893) ‎ (← links)
• Interaction of Crohn's disease susceptibility genes in an Australian paediatric cohort (Q21562133) ‎ (← links)
• Molecular reclassification of Crohn's disease by cluster analysis of genetic variants (Q21562166) ‎ (← links)
• Genetic variation, classification and 'race' (Q22122050) ‎ (← links)
• Use of antibiotics in the treatment of Crohn's disease (Q22241216) ‎ (← links)
• Mycobacteria in Crohn’s disease: how innate immune deficiency may result in chronic inflammation (Q22241484) ‎ (← links)
• Immunology of Crohn's Disease (Q22241677) ‎ (← links)
• New serological markers in inflammatory bowel disease are associated with complicated disease behaviour (Q22242048) ‎ (← links)
• Inflammatory bowel disease (Q22242222) ‎ (← links)
• Defects in mucosal immunity leading to Crohn's disease (Q22242645) ‎ (← links)
• Crohn's disease risk alleles on the NOD2 locus have been maintained by natural selection on standing variation (Q22242851) ‎ (← links)
• Revisiting Crohn's disease as a primary immunodeficiency of macrophages (Q22242983) ‎ (← links)
• Recent Insights Into the Genetics of Inflammatory Bowel Disease (Q22250935) ‎ (← links)
• Microbial Mannan Inhibits Bacterial Killing by Macrophages: A Possible Pathogenic Mechanism for Crohn’s Disease (Q22250936) ‎ (← links)
• A Nonsynonymous SNP in ATG16L1 Predisposes to Ileal Crohn’s Disease and Is Independent of CARD15 and IBD5 (Q22250941) ‎ (← links)
• High prevalence of adherent-invasive Escherichia coli associated with ileal mucosa in Crohn’s disease (Q22250942) ‎ (← links)
• The contribution of NOD2 gene mutations to the risk and site of disease in inflammatory bowel disease (Q22250943) ‎ (← links)
• Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease (Q22251069) ‎ (← links)
• Mechanisms of Disease: pathogenesis of Crohn's disease and ulcerative colitis (Q22251072) ‎ (← links)
• Impaired innate immunity in Crohn's disease (Q22252355) ‎ (← links)
• Crohn's disease: an immune deficiency state (Q22252579) ‎ (← links)
• Crohn’s Disease and SLC11A1 Promoter Polymorphism (Q22252664) ‎ (← links)
• Innate immunity in inflammatory bowel disease: a disease hypothesis (Q22252859) ‎ (← links)
• Probiotics for maintenance of remission in Crohn's disease (Q24244110) ‎ (← links)
• Probiotics for the maintenance of remission in Crohn's disease (Q24246901) ‎ (← links)
• Linkage analysis in the next-generation sequencing era (Q24289003) ‎ (← links)
• Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome (Q24291856) ‎ (← links)
• Binding of Escherichia coli adhesin AfaE to CD55 triggers cell-surface expression of the MHC class I-related molecule MICA (Q24292262) ‎ (← links)
• RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systems (Q24292468) ‎ (← links)
• Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection (Q24292675) ‎ (← links)
• Activation of nucleotide oligomerization domain 2 (NOD2) by human cytomegalovirus initiates innate immune responses and restricts virus replication (Q24293059) ‎ (← links)
• IL-32 synergizes with nucleotide oligomerization domain (NOD) 1 and NOD2 ligands for IL-1beta and IL-6 production through a caspase 1-dependent mechanism (Q24293227) ‎ (← links)
• The nucleotide-binding oligomerization domain-like receptor NLRC5 is involved in IFN-dependent antiviral immune responses (Q24294535) ‎ (← links)
• CATERPILLER 16.2 (CLR16.2), a novel NBD/LRR family member that negatively regulates T cell function (Q24294683) ‎ (← links)
• Stimulation of the intracellular bacterial sensor NOD2 programs dendritic cells to promote interleukin-17 production in human memory T cells (Q24296669) ‎ (← links)
• GRIM-19 interacts with nucleotide oligomerization domain 2 and serves as downstream effector of anti-bacterial function in intestinal epithelial cells (Q24297555) ‎ (← links)
• A novel PAAD-containing protein that modulates NF-kappa B induction by cytokines tumor necrosis factor-alpha and interleukin-1beta (Q24300765) ‎ (← links)
• Human endothelial cells express NOD2/CARD15 and increase IL-6 secretion in response to muramyl dipeptide (Q24301262) ‎ (← links)
• Blau syndrome polymorphisms in NOD2 identify nucleotide hydrolysis and helical domain 1 as signalling regulators (Q24301721) ‎ (← links)
• Galectin 8 targets damaged vesicles for autophagy to defend cells against bacterial invasion (Q24301905) ‎ (← links)
• Cutting edge: Monarch-1: a pyrin/nucleotide-binding domain/leucine-rich repeat protein that controls classical and nonclassical MHC class I genes (Q24302192) ‎ (← links)
• The inflammasomes (Q24304087) ‎ (← links)
• NOD2 controls the nature of the inflammatory response and subsequent fate of Mycobacterium tuberculosis and M. bovis BCG in human macrophages (Q24305119) ‎ (← links)
• Membrane recruitment of NOD2 in intestinal epithelial cells is essential for nuclear factor-{kappa}B activation in muramyl dipeptide recognition (Q24306842) ‎ (← links)