Pages that link to "Q27323098"
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The following pages link to Alpha-synuclein induces lysosomal rupture and cathepsin dependent reactive oxygen species following endocytosis (Q27323098):
Displaying 50 items.
- Cathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug Candidate (Q26782938) (← links)
- M1 and M2 immune activation in Parkinson's Disease: Foe and ally? (Q27024667) (← links)
- Role of Different Alpha-Synuclein Strains in Synucleinopathies, Similarities with other Neurodegenerative Diseases (Q28082714) (← links)
- Zn²⁺ dyshomeostasis caused by loss of ATP13A2/PARK9 leads to lysosomal dysfunction and alpha-synuclein accumulation (Q28116171) (← links)
- Activation of Proinflammatory Responses in Cells of the Airway Mucosa by Particulate Matter: Oxidant- and Non-Oxidant-Mediated Triggering Mechanisms (Q28390698) (← links)
- β2-Microglobulin amyloid fibril-induced membrane disruption is enhanced by endosomal lipids and acidic pH (Q28541669) (← links)
- VCP/p97 cooperates with YOD1, UBXD1 and PLAA to drive clearance of ruptured lysosomes by autophagy (Q30274624) (← links)
- Considerations on the role of environmental toxins in idiopathic Parkinson's disease pathophysiology (Q33603777) (← links)
- Brain disposition of α-Synuclein: roles of brain barrier systems and implications for Parkinson's disease (Q33997655) (← links)
- Lysosomes and α-synuclein form a dangerous duet leading to neuronal cell death. (Q34043163) (← links)
- Lys-63-linked ubiquitination by E3 ubiquitin ligase Nedd4-1 facilitates endosomal sequestration of internalized α-synuclein (Q34073890) (← links)
- The role of Galectin-3 in α-synuclein-induced microglial activation (Q34537231) (← links)
- NADH fluorescence lifetime is an endogenous reporter of α-synuclein aggregation in live cells (Q35660114) (← links)
- NLRP3 Inflammasome Is Expressed and Functional in Mouse Brain Microglia but Not in Astrocytes (Q35668969) (← links)
- Iron Toxicity in the Retina Requires Alu RNA and the NLRP3 Inflammasome (Q35782822) (← links)
- The Interplay between Alpha-Synuclein Clearance and Spreading (Q35833984) (← links)
- Direct Observation of α-Synuclein Amyloid Aggregates in Endocytic Vesicles of Neuroblastoma Cells (Q35996987) (← links)
- SOD1 protein aggregates stimulate macropinocytosis in neurons to facilitate their propagation (Q36234963) (← links)
- α-Synuclein interferes with the ESCRT-III complex contributing to the pathogenesis of Lewy body disease (Q36608855) (← links)
- ER Stress and Autophagic Perturbations Lead to Elevated Extracellular α-Synuclein in GBA-N370S Parkinson's iPSC-Derived Dopamine Neurons. (Q36678823) (← links)
- Effects of Serine 129 Phosphorylation on α-Synuclein Aggregation, Membrane Association, and Internalization (Q36744478) (← links)
- The neural chaperone proSAAS blocks α-synuclein fibrillation and neurotoxicity (Q37181875) (← links)
- Perturbation of neuronal cobalamin transport by lysosomal enzyme inhibition (Q37537087) (← links)
- Targeting α-synuclein for treatment of Parkinson's disease: mechanistic and therapeutic considerations (Q37566684) (← links)
- Biophysical groundwork as a hinge to unravel the biology of α-synuclein aggregation and toxicity (Q38180144) (← links)
- Trafficking and degradation pathways in pathogenic conversion of prions and prion-like proteins in neurodegenerative diseases (Q38341877) (← links)
- Impaired endo-lysosomal membrane integrity accelerates the seeding progression of α-synuclein aggregates (Q38631574) (← links)
- Sorting out release, uptake and processing of alpha-synuclein during prion-like spread of pathology (Q38652662) (← links)
- α-Synuclein transfer between neurons and astrocytes indicates that astrocytes play a role in degradation rather than in spreading (Q38671037) (← links)
- Beyond starvation: An update on the autophagic machinery and its functions (Q38673272) (← links)
- Accumulation of autophagosomes confers cytotoxicity. (Q38695406) (← links)
- Targeting α-synuclein: Therapeutic options (Q38754587) (← links)
- Endocytic vesicle rupture is a conserved mechanism of cellular invasion by amyloid proteins (Q38773604) (← links)
- How strong is the evidence that Parkinson's disease is a prion disorder? (Q38852629) (← links)
- Binding, internalization and fate of Huntingtin Exon1 fibrillar assemblies in mitotic and nonmitotic neuroblastoma cells (Q38860098) (← links)
- Inflammasomes link vascular disease with neuroinflammation and brain disorders (Q38916890) (← links)
- Mitochondria and the NLRP3 inflammasome: physiological and pathological relevance. (Q38947693) (← links)
- Inflammasomes, hormesis, and antioxidants in neuroinflammation: Role of NRLP3 in Alzheimer disease. (Q39012955) (← links)
- The prion model for progression and diversity of neurodegenerative diseases (Q39151589) (← links)
- NLRP3 inflammasome: Its regulation and involvement in atherosclerosis (Q39202038) (← links)
- The spread of prion-like proteins by lysosomes and tunneling nanotubes: Implications for neurodegenerative diseases. (Q39445835) (← links)
- Selective imaging of internalized proteopathic α-synuclein seeds in primary neurons reveals mechanistic insight into transmission of synucleinopathies. (Q41378010) (← links)
- Tunneling nanotubes spread fibrillar α-synuclein by intercellular trafficking of lysosomes (Q42388077) (← links)
- Is trehalose an autophagic inducer? Unraveling the roles of non-reducing disaccharides on autophagic flux and alpha-synuclein aggregation (Q45118902) (← links)
- The lysosomal enzyme alpha-Galactosidase A is deficient in Parkinson's disease brain in association with the pathologic accumulation of alpha-synuclein. (Q46249781) (← links)
- Cell Death Pathways: a Novel Therapeutic Approach for Neuroscientists. (Q46279808) (← links)
- Ethanol preconditioning of rat cerebellar cultures targets NMDA receptors to the synapse and enhances peroxiredoxin 2 expression (Q47136365) (← links)
- Galectin-8-mediated selective autophagy protects against seeded tau aggregation (Q47251029) (← links)
- Therapeutic approaches to target alpha-synuclein pathology (Q47567656) (← links)
- Mitochondrial function and autophagy: integrating proteotoxic, redox, and metabolic stress in Parkinson's disease (Q47653685) (← links)