Pages that link to "Q79988855"
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The following pages link to Inhibition of DNA topoisomerase I by natural and synthetic mono- and dimeric protoberberine alkaloids (Q79988855):
Displaying 15 items.
- Anti-cancer natural products isolated from chinese medicinal herbs (Q35143916) (← links)
- The dual topoisomerase inhibitor A35 preferentially and specially targets topoisomerase 2α by enhancing pre-strand and post-strand cleavage and inhibiting DNA religation (Q36545904) (← links)
- Dimeric approaches to anti-cancer chemotherapeutics (Q37294055) (← links)
- Berberine hydrochloride: anticancer activity and nanoparticulate delivery system (Q37935557) (← links)
- Berberine, an epiphany against cancer. (Q38242945) (← links)
- Evaluation of flavonoids binding to DNA duplexes by electrospray ionization mass spectrometry (Q38291155) (← links)
- Effects of resveratrol, curcumin, berberine and other nutraceuticals on aging, cancer development, cancer stem cells and microRNAs (Q39370677) (← links)
- Berberine inhibits the growth of human colorectal adenocarcinoma in vitro and in vivo (Q43646202) (← links)
- DNA topoisomerase I inhibitory alkaloids from Corydalis saxicola (Q46399538) (← links)
- Interaction of herbal compounds with biological targets: a case study with berberine. (Q53130900) (← links)
- Dietary administration of berberine or Phellodendron amurense extract inhibits cell cycle progression and lung tumorigenesis (Q54634175) (← links)
- Structure-based virtual screening of novel natural alkaloid derivatives as potential binders of h-telo and c-myc DNA G-quadruplex conformations. (Q55068987) (← links)
- Glycoside hydrolase family 18 and 20 enzymes are novel targets of the traditional medicine berberine (Q57022022) (← links)
- Dynamic monitoring of the cytotoxic effects of protoberberine alkaloids from Rhizoma Coptidis on HepG2 cells using the xCELLigence system (Q88163971) (← links)
- Synthesis and In Vitro Photocytotoxicity of 9-/13-Lipophilic Substituted Berberine Derivatives as Potential Anticancer Agents (Q89598613) (← links)