Pages that link to "Q78850683"
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The following pages link to Pressure overload selectively up-regulates Ca2+/calmodulin-dependent protein kinase II in vivo (Q78850683):
Displaying 44 items.
- CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy (Q24293269) (← links)
- The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure (Q24299296) (← links)
- Crucial role for Ca2(+)/calmodulin-dependent protein kinase-II in regulating diastolic stress of normal and failing hearts via titin phosphorylation (Q24307553) (← links)
- Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart (Q24323983) (← links)
- Mechanisms of altered Ca²⁺ handling in heart failure (Q27012758) (← links)
- CaMKIIdelta subtypes: localization and function (Q27026987) (← links)
- A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice (Q28587278) (← links)
- Histone deacetylase 5 acquires calcium/calmodulin-dependent kinase II responsiveness by oligomerization with histone deacetylase 4 (Q28592681) (← links)
- Cardiac CD47 drives left ventricular heart failure through Ca2+-CaMKII-regulated induction of HDAC3. (Q30618651) (← links)
- Protein Kinases as Drug Development Targets for Heart Disease Therapy (Q33678033) (← links)
- Cyclic AMP-dependent protein kinase A regulates the alternative splicing of CaMKIIδ (Q34087577) (← links)
- Comparative genome-wide transcriptional analysis of human left and right internal mammary arteries (Q34124895) (← links)
- CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury (Q34880308) (← links)
- Cardiac CaM Kinase II genes δ and γ contribute to adverse remodeling but redundantly inhibit calcineurin-induced myocardial hypertrophy (Q35047672) (← links)
- Targeting the CaMKII/ERK Interaction in the Heart Prevents Cardiac Hypertrophy (Q35674329) (← links)
- Role of RyR2 phosphorylation at S2814 during heart failure progression (Q36021517) (← links)
- CaMK4 Gene Deletion Induces Hypertension (Q36366241) (← links)
- Metabolic syndrome influences cardiac gene expression pattern at the transcript level in male ZDF rats (Q36691127) (← links)
- Ca2+/calmodulin-dependent protein kinase II-dependent remodeling of Ca2+ current in pressure overload heart failure (Q36869428) (← links)
- Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface (Q36887547) (← links)
- Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction (Q36994116) (← links)
- With great power comes great responsibility: using mouse genetics to study cardiac hypertrophy and failure (Q37101602) (← links)
- The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload (Q37114848) (← links)
- Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice. (Q37170829) (← links)
- Role of the 5'-untranslated region in regulating translational efficiency of specific mRNAs in adult cardiocytes (Q37326930) (← links)
- Calcium/calmodulin-dependent kinase II regulation of cardiac ion channels (Q37427343) (← links)
- Cardiac hypertrophy and heart failure development through Gq and CaM kinase II signaling (Q37763489) (← links)
- CaMKII in myocardial hypertrophy and heart failure (Q37833230) (← links)
- CaMKII in the cardiovascular system: sensing redox states. (Q37899670) (← links)
- Oxidative stress-mediated effects of angiotensin II in the cardiovascular system (Q38192646) (← links)
- Integrated mechanisms of CaMKII-dependent ventricular remodeling (Q38198633) (← links)
- GIT1 mediates HDAC5 activation by angiotensin II in vascular smooth muscle cells (Q40010300) (← links)
- Calmodulin/CaMKII inhibition improves intercellular communication and impulse propagation in the heart and is antiarrhythmic under conditions when fibrosis is absent (Q41273205) (← links)
- Role of activated CaMKII in abnormal calcium homeostasis and I(Na) remodeling after myocardial infarction: insights from mathematical modeling (Q41652870) (← links)
- Sisters Acts: Converging Signaling Between CaMKII and CaMKIV, Two Members of the Same Family (Q41952542) (← links)
- CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation. (Q42114357) (← links)
- Repression of the Central Splicing Regulator RBFox2 Is Functionally Linked to Pressure Overload-Induced Heart Failure (Q43084824) (← links)
- Adult cardiac fibroblast proliferation is modulated by calcium/calmodulin-dependent protein kinase II in normal and hypertrophied hearts (Q44706316) (← links)
- Gallic acid attenuates calcium calmodulin-dependent kinase II-induced apoptosis in spontaneously hypertensive rats (Q47265828) (← links)
- Chronic ethanol increases calcium/calmodulin-dependent protein kinaseIIδ gene expression and decreases monoamine oxidase amount in rat heart muscles: Rescue effect of Zingiber officinale (ginger) extract. (Q50019131) (← links)
- Maternal undernutrition in late gestation increases IGF2 signalling molecules and collagen deposition in the right ventricle of the fetal sheep heart (Q62890075) (← links)
- Molecular biomarkers in cardiac hypertrophy (Q64249013) (← links)
- The deubiquitinase ubiquitin-specific protease 20 is a positive modulator of myocardial β1-adrenergic receptor expression and signaling (Q90411083) (← links)
- Compromised cardiovascular function in aged rats corresponds with increased expression and activity of calcium/calmodulin dependent protein kinase IIδ in aortic endothelium (Q91733656) (← links)